https://m2.mtmt.hu/ hun 2026-04-22 00:17 true true 1 5 5 10 100 1 5 DESC publishedYear false ASC firstAuthor true ASC title true JournalArticle 36444536 VALIDATED true Funding Agency and Grant Number: CAMS Innovation Fund for Medical Sciences [2019-I2M-5-038] Funding text: This study was supported by the CAMS Innovation Fund for Medical Sciences (2019-I2M-5-038). 0 false 2026-01-24T09:37:50.339+0000 2026-01-01T05:46:48.013+0000 2025-11-19T16:15:27.389+0000 true 10039927 Ella Krisztina /api/author/10039927 Author true 10039927 2026-01-01T09:04:28.488+0000 2026-01-01T05:46:47.125+0000 true 521 Szuper Admin /api/admin/521 Admin true false false true 24 24 /api/publicationtype/24 PublicationType 1 true 24 JournalArticle true 10000059 Article true 24 24 /api/publicationtype/24 PublicationType 1 true 24 JournalArticle /api/subtype/10000059 Szakcikk SubType 101 true 10000059 true 1 /api/category/1 Category true 1 Chi, Yanting Integrated study of DNA methylation and transcriptome: a new perspective for exploring the pathogenesis of Sjögren's syndrome true true /api/journal/10015686 REVIEWED true false 10015686 false 10015686 true 1756-8935 1756-8935 Journal FOREIGN 18 1 70 70 16 2025 Background/PurposeSj & ouml;gren's syndrome (SS) is a chronic systemic autoimmune disease characterized by lymphocytic infiltration and formation of lymphoepithelial lesions (LEL) in exocrine glands, leading to secretory dysfunction. DNA methylation, a dynamically regulated epigenetic mark, has been increasingly recognized as a key regulatory mechanism in the pathogenesis of autoimmune diseases including SS, and holds promise for identifying novel diagnostic and therapeutic strategies.MethodsReduced representation bisulfite sequencing (RRBS) was performed on 4 cases of SS and 3 controls to profile genome-wide DNA methylation patterns. Differentially methylated regions (DMRs) and associated differentially methylated genes (DMGs) were detected, followed by functional enrichment analysis. Integration with transcriptomic data (GSE40611) was performed to identify overlapping epigenetic and transcriptional changes.ResultsA total of 29,462 DMRs were detected, with 24,116 hypermethylated and 5,346 hypomethylated regions, indicating an overall increase in methylation levels of SS, and DMGs located in gene promoter regions were significantly enriched in pathways related to immune response, transcriptional regulation, and inflammation. Nine hub genes (LCP2, BTK, LAPTM5, ARHGAP9, IKZF1, WDFY4, CSF2RB, ARHGAP25, DOCK8) were identified, which displayed promoter hyper-or hypomethylation, indicating the complex epigenetic regulatory mechanisms.ConclusionThis study reveals extensive DNA methylation alterations in SS, providing new insights into the epigenetic mechanisms underlying pathogenesis. Moreover, these findings suggest potential biomarkers or therapeutic targets for further investigation to elucidate detailed molecular mechanisms of SS. 2025 true true true false false GOLD 2026-01-24 false http://www.epigeneticsandchromatin.com/home 0 0 0 0 0 0 0 0 0 0 0 0 4 4 Q1 false false false false 2026-02-17T16:16:01.601+0000 19 53 0 4 0 true Language 10002 /api/language/10002 Angol English true 2 true PersonAuthorship 128549012 /api/authorship/128549012 1 0.25 true false false Chi Yanting true false false AuthorshipType 1 /api/authorshiptype/1 0 true 0 true false true PersonAuthorship 128549013 /api/authorship/128549013 2 0.25 false false false Qin Zhiming true false false AuthorshipType 1 /api/authorshiptype/1 0 true 0 true false true PersonAuthorship 128549014 /api/authorship/128549014 3 0.25 false false false Qiu Jingjing true false false AuthorshipType 1 /api/authorshiptype/1 0 true 0 true false true PersonAuthorship 128549015 /api/authorship/128549015 4 0.25 false true true Li Binbin true false false AuthorshipType 1 /api/authorshiptype/1 0 true 0 true false true PublicationIdentifier 30186811 /api/publicationidentifier/30186811 PlainSource 6 /api/publicationsource/6 PublicationSourceType 10001 /api/publicationsourcetype/10001 true true true DOI DOI https://doi.org/@@@ true true 6 true IDENTICAL 10.1186/s13072-025-00637-y https://doi.org/10.1186/s13072-025-00637-y false true PublicationIdentifier 30186812 /api/publicationidentifier/30186812 PlainSource 1 /api/publicationsource/1 PublicationSourceType 10003 /api/publicationsourcetype/10003 false true true WoS WoS https://www.webofscience.com/wos/woscc/full-record/@@@ true true 1 true IDENTICAL 001600871500001 https://www.webofscience.com/wos/woscc/full-record/001600871500001 false true PublicationIdentifier 30186814 /api/publicationidentifier/30186814 PlainSource 3 /api/publicationsource/3 PublicationSourceType 10003 /api/publicationsourcetype/10003 false true true Scopus Scopus http://www.scopus.com/record/display.url?origin=inward&eid=2-s2.0-@@@ true true 3 true 105019782393 http://www.scopus.com/record/display.url?origin=inward&eid=2-s2.0-105019782393 false true PublicationIdentifier 30186813 /api/publicationidentifier/30186813 PlainSource 17 /api/publicationsource/17 PublicationSourceType 10003 /api/publicationsourcetype/10003 false true true PubMed PubMed http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=@@@&dopt=Abstract true true 17 true IDENTICAL 41146282 http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=41146282&dopt=Abstract false true Keyword 64 /api/keyword/64 true 64 true Keyword 1005240 /api/keyword/1005240 true 1005240 true Keyword 1029050 /api/keyword/1029050 true 1029050 true Keyword 1034819 /api/keyword/1034819 true 1034819 true Keyword 1190720 /api/keyword/1190720 true 1190720 true Keyword 1633642 /api/keyword/1633642 true true Keyword 2380658 /api/keyword/2380658 true true Keyword 3388356 /api/keyword/3388356 true true SjrRating 11584377 /api/sjrrating/11584377 49 0.24 journal RatingType 10002 /api/ratingtype/10002 sjr true true ClassificationExternal 1311 /api/classificationexternal/1311 true 1311 true Q1 FROM_LAST_YEAR true true /api/publication/36444536 <div class="JournalArticle Publication long-list"> <div class="authors"> <img title="Idézőközlemény" style="float: left" src="/frontend/resources/grid/publication-citation-icon.png"> <div class="autype autype0"> <span class="author-name" >Chi Yanting </span> ;&nbsp;&nbsp;&nbsp; <span class="author-name" >Qin Zhiming </span> ;&nbsp;&nbsp;&nbsp; <span class="author-name" >Qiu Jingjing </span> ;&nbsp;&nbsp;&nbsp; <span class="author-name" >Li Binbin ✉ </span> </div> </div> <div class="title"><a href="/gui2/?mode=browse&params=publication;36444536" target="_blank">Integrated study of DNA methylation and transcriptome: a new perspective for exploring the pathogenesis of Sjögren's syndrome</a></div> <div> <span class="journal-title">EPIGENETICS & CHROMATIN</span> <span class="journal-issn">(<a target="_blank" href="https://portal.issn.org/resource/ISSN/1756-8935">1756-8935</a> <a target="_blank" href="https://portal.issn.org/resource/ISSN/1756-8935">1756-8935</a>)</span>: <span class="journal-volume">18</span> <span class="journal-issue">1</span> <span class="page"> Paper 70. 16 p. </span> <span class="year">(2025)</span> </div> <div class="pub-footer"> <span class="language" xmlns="http://www.w3.org/1999/html">Nyelv: Angol | </span> <span class="identifiers"> <span class="id identifier oa_none" title="none"> <a style="color:blue" title="10.1186/s13072-025-00637-y" target="_blank" href="https://doi.org/10.1186/s13072-025-00637-y"> DOI </a> </span> <span class="id identifier oa_none" title="none"> <a style="color:blue" title="001600871500001" target="_blank" href="https://www.webofscience.com/wos/woscc/full-record/001600871500001"> WoS </a> </span> <span class="id identifier oa_none" title="none"> <a style="color:black" title="105019782393" target="_blank" href="http://www.scopus.com/record/display.url?origin=inward&eid=2-s2.0-105019782393"> Scopus </a> </span> <span class="id identifier oa_none" title="none"> <a style="color:blue" title="41146282" target="_blank" href="http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=41146282&dopt=Abstract"> PubMed </a> </span> </span> <div class="publication-citation"> <a target="_blank" href="/api/publication?cond=citations.related;eq;36444536&sort=publishedYear,desc&sort=title"> Idézett közlemények száma: 4 </a> </div> <div class="mtid"><span class="long-pub-mtid">Közlemény: 36444536</span> | <span class="status-data status-VALIDATED"> Egyeztetett </span> Idéző | <span class="type-subtype">Folyóiratcikk ( Szakcikk ) </span> | <span class="pub-category">Tudományos</span> | <span class="publication-sourceOfData">WOS TAGGED</span> </div> <div class="lastModified">Utolsó módosítás: 2026.01.01. 06:46 Szuper Admin (admin) </div> <pre class="comment" style="margin-top: 0; margin-bottom: 0;"><u>Megjegyzés</u>: Funding Agency and Grant Number: CAMS Innovation Fund for Medical Sciences [2019-I2M-5-038] Funding text: This study was supported by the CAMS Innovation Fund for Medical Sciences (2019-I2M-5-038).</pre> </div></div> JournalArticle 35792375 VALIDATED true Funding Agency and Grant Number: National Research Development and Innovation Office, Hungary [FK_18 128376, TKP2021-EGA-24, TKP2021-EGA-25]; Janos Bolyai Research Scholarship of the Hungarian Academy of Sciences [EFOP-3.6.3-VEKOP-16-2017-00009] Funding text: The author(s) declare that financial support was received for the research, authorship, and/or publication of this article. This research was funded by research grant No. FK_18 128376 to R. Cs-K., TKP2021-EGA-24 and TKP2021-EGA-25 from the National Research Development and Innovation Office, Hungary. The project also received funding from the Janos Bolyai Research Scholarship of the Hungarian Academy of Sciences to R. Cs-K. and K. E. The SE250+ Excellence PhD Scholarship supported D. Cz. and P. S. 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<span class="author-name" mtid="10085543"><a href="/gui2/?type=authors&mode=browse&sel=10085543" target="_blank">Sasvári Péter (<span class="authorship-author-name">Sasvári Péter</span> <span class="authorAux-mtmt"> Élettan</span>) </a> </span> <span class="author-affil"><span title="Semmelweis Egyetem">SE</span>/<span title="Általános Orvostudományi Kar">AOK</span>/<span title="Intézet">I</span>/Élettani Intézet</span> ;&nbsp;&nbsp;&nbsp; <span class="author-name" mtid="10038409"><a href="/gui2/?type=authors&mode=browse&sel=10038409" target="_blank">Lőrincz Kende (<span class="authorship-author-name">Lőrincz Kende Kálmán</span> <span class="authorAux-mtmt"> Bőrgyógyászat</span>) </a> </span> <span class="author-affil"><span title="Semmelweis Egyetem">SE</span>/<span title="Általános Orvostudományi Kar">AOK</span>/<span title="Klinikum">K</span>/Bőr-, Nemikórtani és Bőronkológiai Klinika</span> ;&nbsp;&nbsp;&nbsp; <span class="author-name" mtid="10039927"><a href="/gui2/?type=authors&mode=browse&sel=10039927" target="_blank">Ella Krisztina (<span class="authorship-author-name">Ella Krisztina</span> <span class="authorAux-mtmt"> Élettan</span>) </a> </span> <span class="author-affil"><span title="Semmelweis Egyetem">SE</span>/<span title="Általános Orvostudományi Kar">AOK</span>/<span title="Intézet">I</span>/Élettani Intézet</span> ;&nbsp;&nbsp;&nbsp; <span class="author-name" >Gellén Virág </span> ;&nbsp;&nbsp;&nbsp; <span class="author-name" mtid="10023793"><a href="/gui2/?type=authors&mode=browse&sel=10023793" target="_blank">Csépányi-Kömi Roland ✉ (<span class="authorship-author-name">Csépányi-Kömi Roland</span> <span class="authorAux-mtmt"> élettan</span>) </a> </span> <span class="author-affil"><span title="Semmelweis Egyetem">SE</span>/<span title="Általános Orvostudományi Kar">AOK</span>/<span title="Intézet">I</span>/Élettani Intézet</span> </div> </div> <div class="title"><a href="/gui2/?mode=browse&params=publication;35792375" target="_blank">ARHGAP25: a novel player in the Pathomechanism of allergic contact hypersensitivity</a></div> <div> <span class="journal-title">FRONTIERS IN IMMUNOLOGY</span> <span class="journal-issn">(<a target="_blank" href="https://portal.issn.org/resource/ISSN/1664-3224">1664-3224</a> <a target="_blank" href="https://portal.issn.org/resource/ISSN/1664-3224">1664-3224</a>)</span>: <span class="journal-volume">16</span> <span class="page"> Paper 1509713. 17 p. </span> <span class="year">(2025)</span> </div> <div class="pub-footer"> <span class="language" xmlns="http://www.w3.org/1999/html">Nyelv: Angol | </span> <span class="identifiers"> <span class="id identifier oa_none" title="none"> <a style="color:blue" title="10.3389/fimmu.2025.1509713" target="_blank" href="https://doi.org/10.3389/fimmu.2025.1509713"> DOI </a> </span> <span class="id identifier oa_none" title="none"> <a style="color:blue" title="001441825000001" target="_blank" href="https://www.webofscience.com/wos/woscc/full-record/001441825000001"> WoS </a> </span> <span class="id identifier oa_none" title="none"> <a style="color:blue" title="86000535624" target="_blank" href="http://www.scopus.com/record/display.url?origin=inward&eid=2-s2.0-86000535624"> Scopus </a> </span> <span class="id identifier oa_none" title="none"> <a style="color:blue" title="40078992" target="_blank" href="http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=40078992&dopt=Abstract"> PubMed </a> </span> </span> <OnlyViewableByAuthor><div class="ratings"> <div class="journal-subject">Folyóirat szakterülete: Scopus - Immunology&nbsp;&nbsp;&nbsp;SJR indikátor:&nbsp;Q1</div> <div class="journal-subject">Folyóirat szakterülete: Scopus - Immunology and Allergy&nbsp;&nbsp;&nbsp;SJR indikátor:&nbsp;Q1</div> </div></OnlyViewableByAuthor> <div class="publication-citation" style="margin-left: 0.5cm;"> <span title="Nyilvános idézőközlemények összesen, említések nélkül" class="citingPub-count">Nyilvános idéző összesen: 1</span> | Független: 1 | Függő: 0 | Nem jelölt: 0 | WoS jelölt: 1 | Scopus jelölt:&nbsp;1 | WoS/Scopus jelölt:&nbsp;1 | DOI jelölt:&nbsp;1 </div> <div class="publication-citation"> <a target="_blank" href="/api/publication?cond=citations.related;eq;35792375&sort=publishedYear,desc&sort=title"> Idézett közlemények száma: 11 </a> </div> <div class="mtid"><span class="long-pub-mtid">Közlemény: 35792375</span> | <span class="status-data status-VALIDATED"> Egyeztetett </span> Forrás Idéző | <span class="type-subtype">Folyóiratcikk ( Szakcikk ) </span> | <span class="pub-category">Tudományos</span> | <span class="publication-sourceOfData">kézi felvitel</span> </div> <div class="funder"> (FK_18 128376), (TKP2021-EGA-24), (TKP2021-EGA-25), Az orvos-, egészségtudományi- és gyógyszerészképzés tudományos műhelyeinek fejlesztése(EFOP-3.6.3-VEKOP-16-2017-00009) Támogató: EFOP-VEKOP </div> <div class="lastModified">Utolsó módosítás: 2026.01.05. 11:37 Kondor Zóra (SE_KK_Admin5_KZ, admin) </div> <pre class="comment" style="margin-top: 0; margin-bottom: 0;"><u>Megjegyzés</u>: Funding Agency and Grant Number: National Research Development and Innovation Office, Hungary [FK_18 128376, TKP2021-EGA-24, TKP2021-EGA-25]; Janos Bolyai Research Scholarship of the Hungarian Academy of Sciences [EFOP-3.6.3-VEKOP-16-2017-00009] Funding text: The author(s) declare that financial support was received for the research, authorship, and/or publication of this article. T...</pre> </div></div> JournalArticle 36762386 VALIDATED true 0 false 2026-01-01T19:47:53.045+0000 2025-12-31T19:46:27.070+0000 2025-12-31T19:46:25.923+0000 true 521 Szuper Admin /api/admin/521 Admin true 2026-01-01T19:45:44.050+0000 2025-12-31T19:46:26.128+0000 true 521 Szuper Admin /api/admin/521 Admin true false false true 24 24 /api/publicationtype/24 PublicationType 1 true 24 JournalArticle true 10000059 Article true 24 24 /api/publicationtype/24 PublicationType 1 true 24 JournalArticle /api/subtype/10000059 Szakcikk SubType 101 true 10000059 true 1 /api/category/1 Category true 1 Wu, Yan Integrating Genomic, eQTL, and Mendelian Randomization Analyses to Identify Microglial Drug Targets in Multiple Sclerosis true true /api/journal/10001233 REVIEWED true false 10001233 false 10001233 true 1582-1838 1582-4934 Journal FOREIGN 29 22 e70754 e70754 11 2025 2025 true true true false false GOLD 2026-01-01 false http://onlinelibrary.wiley.com/journal/10.1111/%28ISSN%291582-4934 0 0 0 0 0 0 0 0 0 0 0 0 2 2 Q2 false false false false 18 54 0 3 0 true Language 10002 /api/language/10002 Angol English true 2 true PersonAuthorship 132023820 /api/authorship/132023820 1 0.33333334 true false true Wu Yan true false false AuthorshipType 1 /api/authorshiptype/1 0 true 0 true false true PersonAuthorship 132023821 /api/authorship/132023821 2 0.33333334 false false false Jiang Wen true false false AuthorshipType 1 /api/authorshiptype/1 0 true 0 true false true PersonAuthorship 132023822 /api/authorship/132023822 3 0.33333334 false true true Wang Jianhong true false false AuthorshipType 1 /api/authorshiptype/1 0 true 0 true false true PublicationIdentifier 30937229 /api/publicationidentifier/30937229 PlainSource 6 /api/publicationsource/6 PublicationSourceType 10001 /api/publicationsourcetype/10001 true true true DOI DOI https://doi.org/@@@ true true 6 true IDENTICAL 10.1111/jcmm.70754 https://doi.org/10.1111/jcmm.70754 false true PublicationIdentifier 30937228 /api/publicationidentifier/30937228 PlainSource 1 /api/publicationsource/1 PublicationSourceType 10003 /api/publicationsourcetype/10003 false true true WoS WoS https://www.webofscience.com/wos/woscc/full-record/@@@ true true 1 true IDENTICAL 001614659200001 https://www.webofscience.com/wos/woscc/full-record/001614659200001 false true Classification 12125 /api/classification/12125 true true Classification 10545 /api/classification/10545 true true Keyword 17902 /api/keyword/17902 true 17902 true Keyword 1002285 /api/keyword/1002285 true 1002285 true Keyword 1579398 /api/keyword/1579398 true true Keyword 2105674 /api/keyword/2105674 true true Keyword 3395458 /api/keyword/3395458 true true Keyword 4112570 /api/keyword/4112570 true true SjrRating 11563840 /api/sjrrating/11563840 101 0.49 journal RatingType 10002 /api/ratingtype/10002 sjr true true ClassificationExternal 1307 /api/classificationexternal/1307 true 1307 true Q2 FROM_LAST_YEAR true true /api/publication/36762386 <div class="JournalArticle Publication long-list"> <div class="authors"> <img title="Idézőközlemény" style="float: left" src="/frontend/resources/grid/publication-citation-icon.png"> <div class="autype autype0"> <span class="author-name" >Wu Yan ✉ </span> ;&nbsp;&nbsp;&nbsp; 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margin-bottom: 0;"><u>Megjegyzés</u>: Export Date: 28 August 2024</pre> </div></div> JournalArticle 35217359 VALIDATED true 0 false 2026-01-02T03:58:19.156+0000 2024-09-30T09:06:07.435+0000 2024-09-06T14:35:00.070+0000 true 10052421 Wisniewski Éva /api/author/10052421 Author true 10052421 2024-12-18T09:49:43.030+0000 2024-09-12T09:52:14.897+0000 true 10080205 Szalóky-Siki Ágnes /api/admin/10080205 Admin true Admin 10014568 /api/admin/10014568 Vándor Andrea true 10014568 true 2025-04-30T11:24:07.640+0000 true false true 24 24 /api/publicationtype/24 PublicationType 1 true 24 JournalArticle true 10000059 Article true 24 24 /api/publicationtype/24 PublicationType 1 true 24 JournalArticle /api/subtype/10000059 Szakcikk SubType 101 true 10000059 true 1 /api/category/1 Category true 1 Sasvári, Péter Neutrophil-specific interactome of ARHGAP25 reveals novel partners and regulatory insights true true /api/journal/10019966 REVIEWED true false 10019966 false 10019966 true 2045-2322 Journal FOREIGN 14 1 20106 20106 13 2024 ARHGAP25, a crucial molecule in immunological processes, serves as a Rac-specific GTPase-activating protein. Its role in cell migration and phagocyte functions, affecting the outcome of complex immunological diseases such as rheumatoid arthritis, renders it a promising target for drug research. Despite its importance, our knowledge of its intracellular interactions is still limited. This study employed proteomic analysis of glutathione S-transferase (GST)-tag pulldowns and co-immunoprecipitation from neutrophilic granulocyte cell lysate, revealing 76 candidates for potential physical interactions that complement ARHGAP25’s known profile. Notably, four small GTPases (RAC2, RHOG, ARF4, and RAB27A) exhibited high affinity for ARHGAP25. The ARHGAP25–RAC2 and ARHGAP25–RHOG interactions appeared to be affected by the activation state of the small GTPases, suggesting a GTP–GDP cycle-dependent interaction. In silico dimer prediction pinpointed ARHGAP25’s GAP domain as a credible binding interface, suggesting its suitability for GTP hydrolysis. Additionally, a list of Fc receptor-related kinases, phosphatases, and three of the 14-3-3 members were identified as potential partners, with in silico predictions highlighting eight binding sites, presenting novel insight on a potential regulatory mechanism for ARHGAP25. 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Intézet</span> ;&nbsp;&nbsp;&nbsp; <span class="author-name" mtid="10023793"><a href="/gui2/?type=authors&mode=browse&sel=10023793" target="_blank">Csépányi-Kömi Roland ✉ (<span class="authorship-author-name">Csépányi-Kömi Roland</span> <span class="authorAux-mtmt"> élettan</span>) </a> </span> <span class="author-affil"><span title="Semmelweis Egyetem">SE</span>/<span title="Általános Orvostudományi Kar">AOK</span>/<span title="Intézet">I</span>/Élettani Intézet</span> </div> </div> <div class="title"><a href="/gui2/?mode=browse&params=publication;35217359" target="_blank">Neutrophil-specific interactome of ARHGAP25 reveals novel partners and regulatory insights</a></div> <div> <span class="journal-title">SCIENTIFIC REPORTS</span> <span class="journal-issn">( <a target="_blank" href="https://portal.issn.org/resource/ISSN/2045-2322">2045-2322</a>)</span>: <span class="journal-volume">14</span> <span class="journal-issue">1</span> <span class="page"> Paper 20106. 13 p. </span> <span 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target="_blank" href="/api/publication?cond=citations.related;eq;35217359&sort=publishedYear,desc&sort=title"> Idézett közlemények száma: 10 </a> </div> <div class="mtid"><span class="long-pub-mtid">Közlemény: 35217359</span> | <span class="status-data status-VALIDATED"> Egyeztetett </span> Forrás Idéző | <span class="type-subtype">Folyóiratcikk ( Szakcikk ) </span> | <span class="pub-category">Tudományos</span> | <span class="publication-sourceOfData">kézi felvitel</span> </div> <div class="funder"> (FK_18 128376), (TKP2021-EGA-24), HCEMM(2022-2.1.1-NL-2022-00005) Támogató: NKFIH, (GINOP-2.3.2-15-2016-00001), European Regional Development Fund(GINOP-2.3.2-15-2016-00020) Támogató: GINOP, (Open access funding provided by Semmelweis University) </div> <div class="lastModified">Utolsó módosítás: 2024.09.30. 11:06 Patkósné Hanesz Andrea (SZBK-4-es admin) </div> <div class="lockedBy">Központi kezelésű 2025.04.30. 13:24 Vándor Andrea (VA, admin) </div> </div></div>