@article{MTMT:34988497,
	title = {ZNF554 Inhibits Endometrial Cancer Progression via Regulating RBM5 and Inactivating WNT/β-Catenin Signaling Pathway},
	url = {https://m2.mtmt.hu/api/publication/34988497},
	author = {Zhu, Cheng-cheng and Sun, Heng-liang and Long, Teng-fei and Lyu, Yuan-yuan and Liu, Jiang-li and Ni, Guan-tai},
	doi = {10.1007/s11596-024-2845-7},
	journal-iso = {CURR MED SCI},
	journal = {CURRENT MEDICAL SCIENCE},
	volume = {44},
	unique-id = {34988497},
	issn = {2096-5230},
	keywords = {endometrial carcinoma; zinc finger protein 554; RNA binding motif 5; Wingless/beta-catenin signaling pathway},
	year = {2024},
	eissn = {2523-899X},
	pages = {406-418}
}

@article{MTMT:34024484,
	title = {ZNF655 promotes the progression of hepatocellular carcinoma through PSMB8},
	url = {https://m2.mtmt.hu/api/publication/34024484},
	author = {Fan, S. and Liu, Y. and Lin, Z. and Zhang, Y. and Zhang, N. and Zhao, Y. and Zhou, J. and Mao, A. and Wang, L. and Feng, Y. and He, X. and Wang, L. and Pan, Q.},
	doi = {10.1002/cbin.12050},
	journal-iso = {CELL BIOL INT},
	journal = {CELL BIOLOGY INTERNATIONAL},
	volume = {47},
	unique-id = {34024484},
	issn = {1065-6995},
	year = {2023},
	eissn = {1095-8355},
	pages = {1535-1546}
}

@article{MTMT:34207144,
	title = {Dysfunction of ZNF554 promotes ROS-induced apoptosis and autophagy in Fetal Growth Restriction via the p62-Keap1-Nrf2 pathway},
	url = {https://m2.mtmt.hu/api/publication/34207144},
	author = {Guo, Y. and Huang, C. and Xu, C. and Qiu, L. and Yang, F.},
	doi = {10.1016/j.placenta.2023.09.009},
	journal-iso = {PLACENTA},
	journal = {PLACENTA},
	volume = {143},
	unique-id = {34207144},
	issn = {0143-4004},
	year = {2023},
	eissn = {1532-3102},
	pages = {34-44}
}

@article{MTMT:33772189,
	title = {Comprehensive analysis of ZNF family genes in prognosis, immunity, and treatment of esophageal cancer},
	url = {https://m2.mtmt.hu/api/publication/33772189},
	author = {Hong, Kunqiao and Yang, Qian and Yin, Haisen and Wei, Na and Wang, Wei and Yu, Baoping},
	doi = {10.1186/s12885-023-10779-5},
	journal-iso = {BMC CANCER},
	journal = {BMC CANCER},
	volume = {23},
	unique-id = {33772189},
	issn = {1471-2407},
	abstract = {BackgroundAs a common malignant tumor, esophageal carcinoma (ESCA) has a low early diagnosis rate and poor prognosis. This study aimed to construct the prognostic features composed of ZNF family genes to effectively predict the prognosis of ESCA patients.MethodsThe mRNA expression matrix and clinical data were downloaded from TCGA and GEO database. Using univariate Cox analysis, lasso regression and multivariate Cox analysis, we screened six prognosis-related ZNF family genes to construct the prognostic model. We then used Kaplan-Meier plot, time-dependent receiver operating characteristic (ROC), multivariable Cox regression analysis of clinical information, and nomogram to evaluate the prognostic value within and across sets, separately and combined. We also validated the prognostic value of the six-gene signature using GSE53624 dataset. The different immune status was observed in the single sample Gene Set Enrichment Analysis (ssGSEA). Finally, real-time quantitative PCR was used to detect the expression of six prognostic ZNF genes in twelve pairs of ESCA and adjacent normal tissues.ResultsA six prognosis-related ZNF family genes model consisted of ZNF91, ZNF586, ZNF502, ZNF865, ZNF106 and ZNF225 was identified. Multivariable Cox regression analysis revealed that six prognosis-related ZNF family genes were independent prognostic factors for overall survival of ESCA patients in TCGA and GSE53624. Further, a prognostic nomogram including the riskScore, age, gender, T, stage was constructed, and TCGA/GSE53624-based calibration plots indicated its excellent predictive performance. Drug Sensitivity and ssGSEA analysis showed that the six genes model was closely related to immune cells infiltration and could be used as a potential predictor of chemotherapy sensitivity.ConclusionWe identified six prognosis-related ZNF family genes model of ESCA, which provide evidence for individualized prevention and treatment.},
	keywords = {IDENTIFICATION; Prognosis; Esophageal cancer; SIGNATURE; PROMOTES; nomogram; RNA-Binding Proteins; Risk model; ZINC-FINGER PROTEINS; ZNF family genes},
	year = {2023},
	eissn = {1471-2407}
}

@article{MTMT:33060460,
	title = {ZNF655 accelerates progression of pancreatic cancer by promoting the binding of E2F1 and CDK1},
	url = {https://m2.mtmt.hu/api/publication/33060460},
	author = {Shao, Z. and Li, C. and Wu, Q. and Zhang, X. and Dai, Y. and Li, S. and Liu, X. and Zheng, X. and Zhang, J. and Fan, H.},
	doi = {10.1038/s41389-022-00418-2},
	journal-iso = {ONCOGENESIS},
	journal = {ONCOGENESIS},
	volume = {11},
	unique-id = {33060460},
	issn = {2157-9024},
	year = {2022},
	eissn = {2157-9024}
}

@article{MTMT:32627659,
	title = {Zinc Finger Proteins in Neuro-Related Diseases Progression},
	url = {https://m2.mtmt.hu/api/publication/32627659},
	author = {Bu, S. and Lv, Y. and Liu, Y. and Qiao, S. and Wang, H.},
	doi = {10.3389/fnins.2021.760567},
	journal-iso = {FRONT NEUROSCI-SWITZ},
	journal = {FRONTIERS IN NEUROSCIENCE},
	volume = {15},
	unique-id = {32627659},
	issn = {1662-4548},
	abstract = {Zinc finger proteins (ZNF) are among the most abundant proteins in eukaryotic genomes. It contains several zinc finger domains that can selectively bind to certain DNA or RNA and associate with proteins, therefore, ZNF can regulate gene expression at the transcriptional and translational levels. In terms of neurological diseases, numerous studies have shown that many ZNF are associated with neurological diseases. The purpose of this review is to summarize the types and roles of ZNF in neuropsychiatric disorders. We will describe the structure and classification of ZNF, then focus on the pathophysiological role of ZNF in neuro-related diseases and summarize the mechanism of action of ZNF in neuro-related diseases. Copyright © 2021 Bu, Lv, Liu, Qiao and Wang.},
	keywords = {SCHIZOPHRENIA; SCHIZOPHRENIA; DNA; Dopamine; EPILEPSY; EPILEPSY; PATHOPHYSIOLOGY; RNA; review; neurologic disease; human; gene expression regulation; disease association; METALLOTHIONEIN; nonhuman; disease course; autism; protein phosphorylation; PROTEIN FUNCTION; Alzheimer's disease; ischemic stroke; ischemic stroke; protein domain; RNA binding; mental disease; DNA binding; Alzheimer disease; protein structure; eukaryote; genetic transcription; neurofibrillary tangle; amyloid beta protein; tau protein; zinc finger protein; Autism Spectrum Disorder; Zinc finger proteins; kruppel like factor 2; neuro-related diseases; transcriptional activator GLI3},
	year = {2021},
	eissn = {1662-453X}
}