{ "labelLang" : "hun", "responseDate" : "2024-03-29 05:37", "paging" : { "last" : true, "first" : true, "totalPages" : 1, "totalElements" : 16, "totalUncutElements" : 16, "totalEstimatedElements" : 9, "size" : 100, "number" : 1, "numberOfElements" : 16, "sort" : [ { "direction" : "DESC", "property" : "publishedYear", "ascending" : false }, { "direction" : "ASC", "property" : "firstAuthor", "ascending" : true }, { "direction" : "ASC", "property" : "title", "ascending" : true } ] }, "content" : [ { "otype" : "JournalArticle", "mtid" : 33779935, "status" : "VALIDATED", "published" : true, "comment" : "Cited By :1 \n Export Date: 27 April 2023 \n Correspondence Address: Yin, Z.-S.; Department of Orthopedics, Anhui Province, China; email: ahmu_zsyin@163.com \n Chemicals/CAS: carprofen, 52263-47-5, 53716-49-7; chondroitin sulfate iron, 54391-57-0; erythropoietin, 11096-26-7; glutathione, 70-18-8; iron, 14093-02-8, 53858-86-9, 7439-89-6; malonaldehyde, 542-78-9; pentobarbital, 57-33-0, 76-74-4; phospholipid hydroperoxide glutathione peroxidase, 97089-70-8 \n Tradenames: hy 100218a, MedChemexpress, China; hy 100579, MedChemexpress, ChinaA006-2-1, Nanjing Jiancheng, China; A039-2-1, Nanjing Jiancheng, China; ASP300S, Leica, Germany; Axio Observer 3; C0037, Beyotime; C2003s, Beyotime; Discovery MR750w, General Electric, United States; DM6B, Leica, Germany; FACSCelesta, Becton Dickinson, United States; ImageJ, National Institute of Health, United States; LightCycler 96, Hoffmann La Roche, United States; P0015L, Beyotime; RM2255, Leica, Germany; S0131S, Beyotime; Talos L120C G2, Thermo, United States; WLA131, WanleiBio, China \n Manufacturers: Beyotime, China; Four Rings, China; MedChemexpress, ChinaBeyotime; Nanjing Jiancheng, China; WanleiBio, China; Leica, Germany; Olympus, Japan; Becton Dickinson, United States; General Electric, United States; Hoffmann La Roche, United States; National Institute of Health, United States; Thermo, United States \n Funding details: National Natural Science Foundation of China, NSFC, 81672161, 81871785 \n Funding text 1: This study was supported by the National Natural Science Foundation of China, Nos. 81871785 and 81672161 (both to ZSY). \n Funding text 2: Funding: This study was supported by the National Natural Science Foundation of China 唀 Nos 堀 and 縁༁紁騁娀 to ZSY Z ? 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inhibits ferroptosis and ameliorates neurological function after spinal cord injury", "identifiers" : [ { "otype" : "PublicationIdentifier", "mtid" : 23451006, "link" : "/api/publicationidentifier/23451006", "label" : "DOI: 10.4103/1673-5374.353496", "source" : { "otype" : "PlainSource", "mtid" : 6, "link" : "/api/publicationsource/6", "label" : "DOI", "type" : { "otype" : "PublicationSourceType", "mtid" : 10001, "link" : "/api/publicationsourcetype/10001", "label" : "DOI", "mayHaveOa" : true, "published" : true, "snippet" : true }, "name" : "DOI", "nameEng" : "DOI", "linkPattern" : "https://doi.org/@@@", "publiclyVisible" : true, "published" : true, "oldId" : 6, "snippet" : true }, "validState" : "IDENTICAL", "idValue" : "10.4103/1673-5374.353496", "realUrl" : "https://doi.org/10.4103/1673-5374.353496", "published" : false, "snippet" : true }, { "otype" : "PublicationIdentifier", "mtid" : 23451007, "link" : "/api/publicationidentifier/23451007", "label" : "WoS: 000903723200035", 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"881", "pageLength" : 8, "publishedYear" : 2023, "abstractText" : "Ferroptosis is one of the critical pathological events in spinal cord injury. Erythropoietin has been reported to improve the recovery of spinal cord injury. However, whether ferroptosis is involved in the neuroprotective effects of erythropoietin on spinal cord injury has not been examined. In this study, we established rat models of spinal cord injury by modified Allen's method and intraperitoneally administered 1000 and 5000 IU/kg erythropoietin once a week for 2 successive weeks. Both low and high doses of erythropoietin promoted recovery of hindlimb function, and the high dose of erythropoietin led to better outcome. High dose of erythropoietin exhibited a stronger suppressive effect on ferroptosis relative to the low dose of erythropoietin. The effects of erythropoietin on inhibiting ferroptosis-related protein expression and restoring mitochondrial morphology were similar to those of Fer-1 (a ferroptosis suppressor), and the effects of erythropoietin were largely diminished by RSL3 (ferroptosis activator). In vitro experiments showed that erythropoietin inhibited RSL3-induced ferroptosis in PC12 cells and increased the expression of xCT and Gpx4. This suggests that xCT and Gpx4 are involved in the neuroprotective effects of erythropoietin on spinal cord injury. Our findings reveal the underlying anti-ferroptosis role of erythropoietin and provide a potential therapeutic strategy for treating spinal cord injury. © 2023 Wolters Kluwer Medknow Publications. 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