@article{MTMT:34502777,
	title = {The Apical Endocytic-Lysosomal Apparatus in CLCN5 Mutations with Phenotypic-Genotypic Correlations in Three Cases},
	url = {https://m2.mtmt.hu/api/publication/34502777},
	author = {Kalmár, Tibor and Jakab, Dániel and Maróti, Zoltán and Lakatos, Orsolya Judit and Vas, Tibor and Bereczki, Csaba and Iványi, Béla},
	doi = {10.3390/ijms25020966},
	journal-iso = {INT J MOL SCI},
	journal = {INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES},
	volume = {25},
	unique-id = {34502777},
	issn = {1661-6596},
	abstract = {Dent disease type 1 is characterized by pathogenic CLCN5 gene variants and impaired receptor-mediated endocytosis in proximal tubules. However, mutation-related abnormalities in proximal tubules have not yet been described. Here, we present three patients with CLCN5 alterations and distinct morphological changes of the apical endocytic-lysosomal apparatus. The proximal tubular ultrastructure was investigated in kidney biopsy samples of three boys genotyped for non-nephrotic proteinuria. Controls: seven patients with nephrotic-range glomerular proteinuria. The genotyping findings revealed an already-known missense mutation in one patient and hitherto undescribed frameshift variants in two patients. Low-molecular-weight proteinuria, focal global glomerulosclerosis, proximal tubular changes, and tubular calcium deposits characterized each case. Three subsets of proximal tubular cells were observed: those without any abnormality, those with aplasia of apical endocytic-lysosomal apparatus and shrinkage of cells, and those with hypoplasia of apical endocytic apparatus, accumulation of proteinaceous substance in dysmorphic lysosomes, and dysmorphic mitochondria. The distribution of subsets varied from patient to patient. In one patient with a frameshift variant, an oxidative stress-like injury of proximal tubular cells and podocytes accompanied the above-mentioned alterations. Focal aplasia/hypoplasia of apical endocytic apparatus and subsequent changes in cytoplasmic organelles characterized proximal tubules in the CLCN5 pathogenic variants.},
	year = {2024},
	eissn = {1422-0067},
	orcid-numbers = {Kalmár, Tibor/0000-0002-0419-2009; Maróti, Zoltán/0000-0002-0515-117X; Bereczki, Csaba/0000-0003-0091-3558}
}

@article{MTMT:33842541,
	title = {Membranous Nephropathy With Extensive Tubular Basement Membrane Deposits Following Allogeneic Hematopoietic Cell Transplant: A Report of 5 Cases},
	url = {https://m2.mtmt.hu/api/publication/33842541},
	author = {Nasr, Samih H. and Leung, Nelson and Said, Samar M. and Alkhateeb, Hassan B. and Madden, Benjamin J. and Charlesworth, M. Cristine and Beck, Laurence H. Jr. and Larsen, Christopher P. and Sethi, Sanjeev},
	doi = {10.1053/j.ajkd.2021.07.021},
	journal-iso = {AM J KIDNEY DIS},
	journal = {AMERICAN JOURNAL OF KIDNEY DISEASES},
	volume = {79},
	unique-id = {33842541},
	issn = {0272-6386},
	abstract = {Tubular basement membrane (TBM) deposits are very uncommon in non-lupus membranous nephropathy. We report 5 patients with membranous nephropathy and extensive TBM deposits following allogeneic hematopoietic cell transplant. Patients presented with nephrotic syndrome (3 also had acute kidney injury) late post-transplant in association with chronic graft-versus-host disease (cGVHD). Kidney biopsies revealed global subepithelial and extensive TBM immune complex deposits, accompanied by acute tubular injury (n = 4) and tubulointerstitial inflammation (n = 4). Proteomic analysis of glomeruli in 4 cases identified PLA(2)R in 1, with no significant protein spectra for PLA(2)R, THSD7A, EX1/2, NELL-1, PCDH7, NCAM1, or SEMA3B detected in the remaining 3. On follow-up (for a mean 42 months), 4 patients had complete and 1 partial remission following prednisone and/or rituximab therapy. We propose that membranous nephropathy with extensive TBM deposits is a distinctive clinicopathologic lesion associated with allogeneic hematopoietic cell transplant. Pathogenesis likely involves cGVHD-driven antibodies against glomerular and TBM components, the identity of which remains to be elucidated.},
	year = {2022},
	eissn = {1523-6838},
	pages = {904-908},
	orcid-numbers = {Leung, Nelson/0000-0002-5651-1411}
}

@article{MTMT:32934658,
	title = {Fanconi syndrome, nephrotic-range proteinuria, and hypoalbuminemia in a newborn-Occam's razor or Hickam's dictum? Answers},
	url = {https://m2.mtmt.hu/api/publication/32934658},
	author = {Nugent, James T. and Reardon, Juliann and Crana, Christine and Greenberg, Jason H. and Warejko, Jillian K. and Goodwin, Julie E.},
	doi = {10.1007/s00467-021-05255-w},
	journal-iso = {PEDIATR NEPHROL},
	journal = {PEDIATRIC NEPHROLOGY},
	volume = {37},
	unique-id = {32934658},
	issn = {0931-041X},
	keywords = {proteinuria; metabolic acidosis; nephrin; Fanconi Syndrome; congenital nephrotic syndrome},
	year = {2022},
	eissn = {1432-198X},
	pages = {129-132}
}

@article{MTMT:30902374,
	title = {Anti-LRP2 NephroPathy With Abundant IgG4-Positive Plasma Cells: A Case Report},
	url = {https://m2.mtmt.hu/api/publication/30902374},
	author = {Dinesh, Kumar P. and Raniele, Dean and Michels, Kendall and Avasare, Rupali S. and Larsen, Christopher P. and Kayton, Robert and Khalighi, Mazdak A. and Andeen, Nicole K.},
	doi = {10.1053/j.ajkd.2018.12.039},
	journal-iso = {AM J KIDNEY DIS},
	journal = {AMERICAN JOURNAL OF KIDNEY DISEASES},
	volume = {74},
	unique-id = {30902374},
	issn = {0272-6386},
	abstract = {In older adults, the most common kidney biopsy diagnoses include pauci-immune crescentic glomerulonephritis, membranous nephropathy, and focal segmental glomerulosclerosis. Recently, investigators described a small series of older patients (aged 66-80 years) with acute kidney injury and a kidney biopsy demonstrating tubular basement membrane (TBM) immune deposits of polytypic immunoglobulin G (IgG) and C3, acute tubular injury, and tubulointerstitial inflammation. They identified a circulating antibody against kidney tubular low-density lipoprotein (LDL) receptor-related protein 2 (LRP2; also known as megalin) in patients' sera and colocalization of LRP2 with IgG in TBM deposits. We present a rare case of anti-LRP2 nephropathy/anti-brush border antibody disease and describe the novel feature of abundant IgG4-positive interstitial plasma cells. Along with the combination of TBM deposits, tubulointerstitial nephritis (TIN), and segmental glomerular subepithelial immune deposits seen in both entities, this newly described feature adds to the morphologic overlap with IgG4-related TIN. Identification of large TBM deposits using light microscopy and IgG staining of apical aspects of proximal tubules using immunofluorescence microscopy can point to the correct diagnosis of anti-LRP2 nephropathy and prompt confirmatory studies. Particularly in older patients with immune complex-mediated TIN who lack clinical, laboratory, radiographic, and/or characteristic histologic features of IgG4-TIN or other autoimmune, infectious, or drug-related injury, a diagnosis of anti-LRP2 nephropathy should be considered.},
	year = {2019},
	eissn = {1523-6838},
	pages = {132-137}
}

@article{MTMT:24799680,
	title = {Urinary proteins induce lysosomal membrane permeabilization and lysosomal dysfunction in renal tubular epithelial cells},
	url = {https://m2.mtmt.hu/api/publication/24799680},
	author = {Liu, Wei Jing and Xu, Bi-Hua and Ye, Lin and Liang, Dong and Wu, Hong-Luan and Zheng, Yuan-Yuan and Deng, Jian Kun and Li, Benyi and Liu, Hua-feng},
	doi = {10.1152/ajprenal.00383.2014},
	journal-iso = {AM J PHYSIOL RENAL},
	journal = {AMERICAN JOURNAL OF PHYSIOLOGY: RENAL PHYSIOLOGY},
	volume = {308},
	unique-id = {24799680},
	issn = {1931-857X},
	year = {2015},
	eissn = {1522-1466},
	pages = {F639-F649}
}

@{MTMT:26107060,
	title = {Membranous nephropathy in children},
	url = {https://m2.mtmt.hu/api/publication/26107060},
	author = {Valentini, RP},
	booktitle = {Pediatric Nephrology, Seventh Edition},
	doi = {10.1007/978-3-662-43596-0_30},
	publisher = {Springer Netherlands},
	unique-id = {26107060},
	year = {2015},
	pages = {1055-1075}
}

@{MTMT:26107062,
	title = {Acute and chronic tubulointerstitial nephritis},
	url = {https://m2.mtmt.hu/api/publication/26107062},
	author = {Brodsky, SV and Nadasdy, T},
	booktitle = {Heptinstall's Pathology of the Kidney},
	publisher = {Wolters Kluwer Health},
	unique-id = {26107062},
	year = {2014}
}

@{MTMT:26107061,
	title = {Membranous glomerulonephritis},
	url = {https://m2.mtmt.hu/api/publication/26107061},
	author = {Markowitz, GS and D'Agati, VD},
	booktitle = {Heptinstall's Pathology of the Kidney},
	publisher = {Wolters Kluwer Health},
	unique-id = {26107061},
	year = {2014}
}

@article{MTMT:23226594,
	title = {Isaac Albéniz (1860-1909): Spanish musician who died of chronic renal disease},
	url = {https://m2.mtmt.hu/api/publication/23226594},
	author = {García-Nieto, VM and Peralta-Aros, C},
	doi = {10.1258/jmb.2011.011023},
	journal-iso = {J MED BIOGRAP},
	journal = {JOURNAL OF MEDICAL BIOGRAPHY},
	volume = {21},
	unique-id = {23226594},
	issn = {0967-7720},
	year = {2013},
	eissn = {1758-1087},
	pages = {26-31}
}

@article{MTMT:23543010,
	title = {Membranous nephropathy},
	url = {https://m2.mtmt.hu/api/publication/23543010},
	author = {Mercadal, L},
	doi = {10.1016/j.nephro.2013.10.002},
	journal-iso = {NEPHROL THER},
	journal = {NEPHROLOGIE & THERAPEUTIQUE},
	volume = {9},
	unique-id = {23543010},
	issn = {1769-7255},
	year = {2013},
	eissn = {1872-9177},
	pages = {507-517}
}

@article{MTMT:22345435,
	title = {Autoimmune bullous diseases in childhood},
	url = {https://m2.mtmt.hu/api/publication/22345435},
	author = {Sansaricq, F and Stein, S L and Petronic-Rosic, V},
	doi = {10.1016/j.clindermatol.2011.03.018},
	journal-iso = {CLIN DERMATOL},
	journal = {CLINICS IN DERMATOLOGY},
	volume = {30},
	unique-id = {22345435},
	issn = {0738-081X},
	year = {2012},
	eissn = {1879-1131},
	pages = {114-127}
}

@article{MTMT:22345446,
	title = {Membranous nephropathy in children: clinical presentation and therapeutic approach},
	url = {https://m2.mtmt.hu/api/publication/22345446},
	author = {Menon, S and Valentini, RP},
	doi = {10.1007/s00467-009-1324-5},
	journal-iso = {PEDIATR NEPHROL},
	journal = {PEDIATRIC NEPHROLOGY},
	volume = {25},
	unique-id = {22345446},
	issn = {0931-041X},
	year = {2010},
	eissn = {1432-198X},
	pages = {1419-1428}
}

@article{MTMT:20692579,
	title = {A 59-kd renal antigen as a new target for rapidly progressive glomerulonephritis},
	url = {https://m2.mtmt.hu/api/publication/20692579},
	author = {Audard, V and Hellmark, T and El, Karoui K and Noel, LH and Pardon, A and Desvaux, D and Touchard, G and Remy, P and Lang, P and Sahali, D},
	doi = {10.1053/j.ajkd.2007.02.002},
	journal-iso = {AM J KIDNEY DIS},
	journal = {AMERICAN JOURNAL OF KIDNEY DISEASES},
	volume = {49},
	unique-id = {20692579},
	issn = {0272-6386},
	year = {2007},
	eissn = {1523-6838},
	pages = {710-716}
}

@article{MTMT:20692578,
	title = {Target antigens and nephritogenic antibodies in membranous nephropathy: of rats and men},
	url = {https://m2.mtmt.hu/api/publication/20692578},
	author = {Ronco, P and Debiec, H},
	doi = {10.1007/s00281-007-0091-2},
	journal-iso = {SEMIN IMMUNOPATHOL},
	journal = {SEMINARS IN IMMUNOPATHOLOGY},
	volume = {29},
	unique-id = {20692578},
	issn = {1863-2297},
	year = {2007},
	eissn = {1863-2300},
	pages = {445-458}
}

@article{MTMT:22345449,
	title = {Mechanisms of Disease: alloimmunization in renal diseases},
	url = {https://m2.mtmt.hu/api/publication/22345449},
	author = {Ronco, P and Debiec, H and Guigonis, V},
	doi = {10.1038/ncpneph0198},
	journal-iso = {NAT CLIN PRACT NEPHR},
	journal = {NATURE CLINICAL PRACTICE NEPHROLOGY},
	volume = {2},
	unique-id = {22345449},
	issn = {1745-8323},
	year = {2006},
	eissn = {1745-8331},
	pages = {388-397}
}

@article{MTMT:22345436,
	title = {Childhood membranous nephropathy in association with interstitial nephritis and Fanconi syndrome [1]},
	url = {https://m2.mtmt.hu/api/publication/22345436},
	author = {Shenoy, M and Krishnan, R and Moghal, N},
	doi = {10.1007/s00467-005-2104-5},
	journal-iso = {PEDIATR NEPHROL},
	journal = {PEDIATRIC NEPHROLOGY},
	volume = {21},
	unique-id = {22345436},
	issn = {0931-041X},
	year = {2006},
	eissn = {1432-198X},
	pages = {441}
}

@{MTMT:20692393,
	title = {Molecular Pathomechanisms of Membranous Nephropathy : from Bench to Bedside},
	url = {https://m2.mtmt.hu/api/publication/20692393},
	author = {Ronco, P and Debiec, H},
	booktitle = {Nephrology, hypertension, dialysis, transplantation : Budapest Nephrology School},
	publisher = {Magyar Vese-Alapítvány},
	unique-id = {20692393},
	year = {2005},
	pages = {183-198}
}

@article{MTMT:22345450,
	title = {Molecular pathomechanisms of membranous nephropathy: From Heymann nephritis to alloimmunization},
	url = {https://m2.mtmt.hu/api/publication/22345450},
	author = {Ronco, P and Debiec, H},
	doi = {10.1681/ASN.2004121080},
	journal-iso = {J AM SOC NEPHROL},
	journal = {JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY},
	volume = {16},
	unique-id = {22345450},
	issn = {1046-6673},
	year = {2005},
	eissn = {1533-3450},
	pages = {1205-1213}
}

@article{MTMT:22345437,
	title = {Extra-membranous nephropathy. Glomérulopathie extramembraneuse},
	url = {https://m2.mtmt.hu/api/publication/22345437},
	author = {Mercadal, L},
	doi = {10.1016/j.emcnep.2004.04.001},
	journal-iso = {EMC - Nephrologie},
	journal = {EMC - Nephrologie},
	volume = {1},
	unique-id = {22345437},
	issn = {1638-6248},
	year = {2004},
	pages = {55-72}
}

@inproceedings{MTMT:23141904,
	title = {Tubulointerstitial immune deposits in renal diseases},
	url = {https://m2.mtmt.hu/api/publication/23141904},
	author = {Vizjak, A and Jurcic, V and Hvala, A and Ferluga, M},
	booktitle = {UPDATE IN PATHOLOGY, PROCEEDINGS},
	publisher = {Univerza v Ljubljani},
	unique-id = {23141904},
	year = {2003},
	pages = {240-242}
}

@article{MTMT:1388983,
	title = {The value of electron microscopy in the diagnosis of chronic renal allograft rejection},
	url = {https://m2.mtmt.hu/api/publication/1388983},
	author = {Iványi, Béla and Nagyné Kemény, Éva Ilona and Szederkényi, Edit and Marofka, Ferenc and Szenohradszky, Pál},
	doi = {10.1038/modpathol.3880461},
	journal-iso = {MODERN PATHOL},
	journal = {MODERN PATHOLOGY},
	volume = {14},
	unique-id = {1388983},
	issn = {0893-3952},
	abstract = {The main causes of the late dysfunction of renal allografts are chronic rejection and chronic transplant nephropathy. Both are clinicopathologic entities, with a similar clinical presentation, but different histologic appearances. Chronic rejection is characterized by the presence of alloantigen-induced lesions (transplant arteriopathy and transplant glomerulopathy), and chronic transplant nephropathy by nonspecific sclerosing changes. The incidence of transplant arteriopathy and transplant glomerulopathy is relatively low. Electron microscopy (EM) may overcome the limitations In the histologic diagnosis of chronic rejection, because It verifies alloantigen-Induced chronic microvasculopathy in the peritubular capillaries (transplant capillaropathy), and identifies transplant glomerulopathy more precisely than does fight microscopy. To assess the value of EM in chronic rejection diagnosis, a retrospective search for transplant capillaropathy and transplant glomerulopathy was performed In a consecutive series of 91 biopsies performed; greater than or equal to6 months after Implantation (median: 26 months, range 6-186) and the diagnoses were reclassified on the basis of the ultrastructural findings. The definitions used were: transplant capillaropathy: a peritubular capillary profile with seven or more circumferential basement membrane layers, or at least three profiles with five or six circumferential layers; ultrastructurally verified transplant glomerulopathy: thickening of the capillary wall in at least three loops in consequence of the widening of the subendothelial space by abnormal basement membrane material, and the formation of a new layer(s) of basal lamina; and chronic rejection: the presence of transplant capillaropathy and/or transplant glomerulopathy and/or transplant arteriopathy. Histologically, chronic transplant nephropathy, chronic rejection, chronic cyclosporine nephrotoxicity, glomerulonephritis, acute rejection, "suspicious" for acute rejection, and "others" were diagnosed In 37%,34%,21%,19%,57%,30%, and 5% of the specimens, respectively. The results of EM increased the diagnosis of chronic rejection to 69% of the cases, and decreased chronic transplant nephropathy to 15%. The individual incidence of transplant capillaropathy and transplant glomerulopathy was 79% and 57%, respectively, and their cumulative incidence was 92%. Five biopsies exhibited merely transplant arteriopathy. A late dysfunction typically had more than one cause; the most frequent combination was chronic rejection and acute rejection. In conclusion, the EM search for transplant capillaropathy and transplant glomerulopathy doubled the frequency of the diagnosis of chronic rejection. Currently, the evaluation of renal allograft biopsies from recipients with a late dysfunction relies on standard light microscopy. Because fight microscopy per se proved to be insensitive in the diagnosis of chronic rejection, incorporation of EM into the evaluation of late dysfunction biopsies is strongly recommended.},
	year = {2001},
	eissn = {1530-0285},
	pages = {1200-1208},
	orcid-numbers = {Szederkényi, Edit/0000-0002-3583-6874}
}

@{MTMT:20098156,
	title = {Az acut tubulointerstitialis nephritis pathologiája},
	url = {https://m2.mtmt.hu/api/publication/20098156},
	author = {Iványi, B},
	booktitle = {Nephrologia 2000},
	publisher = {DEOEC},
	unique-id = {20098156},
	year = {2000},
	pages = {197-200}
}

@article{MTMT:22345453,
	title = {Membranous glomerulopathy with Bowman's capsular and tubular basement membrane deposits},
	url = {https://m2.mtmt.hu/api/publication/22345453},
	author = {Markowitz, GS and Kambham, N and Maruyama, S and Appel, GB and Cohen, DJ and Kim, RC and Andres, GA and D'Agati, VD},
	journal-iso = {CLIN NEPHROL},
	journal = {CLINICAL NEPHROLOGY},
	volume = {54},
	unique-id = {22345453},
	issn = {0301-0430},
	year = {2000},
	eissn = {0301-0430},
	pages = {478-486}
}

@article{MTMT:22345452,
	title = {Membranous nephropathy with anti-tubular basement membrane antibody may be X-linked},
	url = {https://m2.mtmt.hu/api/publication/22345452},
	author = {Tay, AHN and Ren, EC and Murugasu, B and Sim, SK and Tan, PH and Cohen, AH and Yap, HK},
	journal-iso = {PEDIATR NEPHROL},
	journal = {PEDIATRIC NEPHROLOGY},
	volume = {14},
	unique-id = {22345452},
	issn = {0931-041X},
	year = {2000},
	eissn = {1432-198X},
	pages = {747-753}
}

@article{MTMT:22345454,
	title = {Three-year-old boy with partial Fanconi syndrome},
	url = {https://m2.mtmt.hu/api/publication/22345454},
	author = {Markowitz, GS and Seigle, RL and D'Agati, VD},
	doi = {10.1016/S0272-6386(99)70130-7},
	journal-iso = {AM J KIDNEY DIS},
	journal = {AMERICAN JOURNAL OF KIDNEY DISEASES},
	volume = {34},
	unique-id = {22345454},
	issn = {0272-6386},
	year = {1999},
	eissn = {1523-6838},
	pages = {184-188}
}

@article{MTMT:22345438,
	title = {Membranous glomerulonephritis induced in the pig by antibody to angiotensin-converting enzyme: Considerations on its relevance to the pathogenesis of human idiopathic: Membranous glomerulonephritis},
	url = {https://m2.mtmt.hu/api/publication/22345438},
	author = {Maruyama, S and Cantu, III E and Demartino, C and Vladutiu, A and Caldwell, P R B and Wang, C Y and D'Agati, V and Godman, G and Stern, D M and Andres, G},
	journal-iso = {J AM SOC NEPHROL},
	journal = {JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY},
	volume = {10},
	unique-id = {22345438},
	issn = {1046-6673},
	year = {1999},
	eissn = {1533-3450},
	pages = {2102-2108}
}

@article{MTMT:20098149,
	title = {Probable diltiazem-induced acute interstitial nephritis},
	url = {https://m2.mtmt.hu/api/publication/20098149},
	author = {Abadin, JA and Duran, JA and Perez, de Leon JA},
	journal-iso = {ANN PHARMACOTHER},
	journal = {ANNALS OF PHARMACOTHERAPY},
	volume = {32},
	unique-id = {20098149},
	issn = {1060-0280},
	year = {1998},
	eissn = {1542-6270},
	pages = {656-658}
}

@article{MTMT:20098151,
	title = {Monoclonal antibody analysis of crescentic membranous glomerulonephropathy},
	url = {https://m2.mtmt.hu/api/publication/20098151},
	author = {Arrizabalaga, P and Sans, Boix A and Torras, Rabassa A and Darnell, Tey A and Revert, Torrellas L},
	journal-iso = {AM J NEPHROL},
	journal = {AMERICAN JOURNAL OF NEPHROLOGY},
	volume = {18},
	unique-id = {20098151},
	issn = {0250-8095},
	year = {1998},
	eissn = {1421-9670},
	pages = {77-82}
}

@article{MTMT:20098140,
	title = {Lupus-like nephrotropic autoantibodies in non-autoimmune mice harboring an anti-basement membraneanti-DNA Ig heavy chain transgene},
	url = {https://m2.mtmt.hu/api/publication/20098140},
	author = {Foster, MH and Fitzsimons, MM},
	journal-iso = {MOL IMMUNOL},
	journal = {MOLECULAR IMMUNOLOGY},
	volume = {35},
	unique-id = {20098140},
	issn = {0161-5890},
	year = {1998},
	eissn = {1872-9142},
	pages = {83-94}
}

@article{MTMT:20098142,
	title = {Recurrent crescentic membranous nephropathy in two successive renal transplants: association with choroidal effusions and retinal detachment},
	url = {https://m2.mtmt.hu/api/publication/20098142},
	author = {Lazowski, P and Sablay, LB and Glicklich, D},
	journal-iso = {AM J NEPHROL},
	journal = {AMERICAN JOURNAL OF NEPHROLOGY},
	volume = {18},
	unique-id = {20098142},
	issn = {0250-8095},
	year = {1998},
	eissn = {1421-9670},
	pages = {146-150}
}

@article{MTMT:20098135,
	title = {Anti-glomerular basement membrane disease and dual positivity for antineutrophil cytoplasmic antibody in a patient with membranous nephropathy},
	url = {https://m2.mtmt.hu/api/publication/20098135},
	author = {Meisels, IS and Stillman, IE and Kuhlik, AB},
	journal-iso = {AM J KIDNEY DIS},
	journal = {AMERICAN JOURNAL OF KIDNEY DISEASES},
	volume = {32},
	unique-id = {20098135},
	issn = {0272-6386},
	year = {1998},
	eissn = {1523-6838},
	pages = {646-648}
}