@article{MTMT:34824081,
	title = {Mitochondrial dysfunction in long COVID: mechanisms, consequences, and potential therapeutic approaches},
	url = {https://m2.mtmt.hu/api/publication/34824081},
	author = {Molnár, Tihamér and Lehoczki, Andrea Marianna and Fekete, Mónika and Várnai, Réka and Zavori, Laszlo and Erdő-Bonyár, Szabina and Simon, Diána and Berki, Tímea and Csécsei, Péter and Ezer, Erzsébet},
	doi = {10.1007/s11357-024-01165-5},
	journal-iso = {GEROSCIENCE},
	journal = {GEROSCIENCE: OFFICIAL JOURNAL OF THE AMERICAN AGING ASSOCIATION (AGE)},
	volume = {In press},
	unique-id = {34824081},
	issn = {2509-2715},
	abstract = {The COVID-19 pandemic, caused by the SARS-CoV-2 virus, has introduced the medical community to the phenomenon of long COVID, a condition characterized by persistent symptoms following the resolution of the acute phase of infection. Among the myriad of symptoms reported by long COVID sufferers, chronic fatigue, cognitive disturbances, and exercise intolerance are predominant, suggesting systemic alterations beyond the initial viral pathology. Emerging evidence has pointed to mitochondrial dysfunction as a potential underpinning mechanism contributing to the persistence and diversity of long COVID symptoms. This review aims to synthesize current findings related to mitochondrial dysfunction in long COVID, exploring its implications for cellular energy deficits, oxidative stress, immune dysregulation, metabolic disturbances, and endothelial dysfunction. Through a comprehensive analysis of the literature, we highlight the significance of mitochondrial health in the pathophysiology of long COVID, drawing parallels with similar clinical syndromes linked to post-infectious states in other diseases where mitochondrial impairment has been implicated. We discuss potential therapeutic strategies targeting mitochondrial function, including pharmacological interventions, lifestyle modifications, exercise, and dietary approaches, and emphasize the need for further research and collaborative efforts to advance our understanding and management of long COVID. This review underscores the critical role of mitochondrial dysfunction in long COVID and calls for a multidisciplinary approach to address the gaps in our knowledge and treatment options for those affected by this condition.},
	year = {2024},
	eissn = {2509-2723},
	orcid-numbers = {Fekete, Mónika/0000-0001-8632-2120; Berki, Tímea/0000-0002-0134-8127}
}

@{MTMT:34823855,
	title = {Donorgondozás},
	url = {https://m2.mtmt.hu/api/publication/34823855},
	author = {Ezer, Erzsébet},
	booktitle = {Az intenzív terápia gyakorlata},
	unique-id = {34823855},
	year = {2024},
	pages = {268}
}

@{MTMT:34823777,
	title = {Az agyhalál},
	url = {https://m2.mtmt.hu/api/publication/34823777},
	author = {Ezer, Erzsébet},
	booktitle = {Az intenzív terápia gyakorlata},
	unique-id = {34823777},
	year = {2024},
	pages = {28}
}

@{MTMT:34823734,
	title = {Folyadék- és elektrolitzavar koponyasérültekben},
	url = {https://m2.mtmt.hu/api/publication/34823734},
	author = {Ezer, Erzsébet},
	booktitle = {Súlyos baleseti agysérültek ellátása},
	unique-id = {34823734},
	year = {2024},
	pages = {182-185}
}

@{MTMT:34823725,
	title = {Ozmoterápia},
	url = {https://m2.mtmt.hu/api/publication/34823725},
	author = {Ezer, Erzsébet},
	booktitle = {Súlyos baleseti agysérültek ellátása},
	unique-id = {34823725},
	year = {2024},
	pages = {174-174}
}

@{MTMT:34823721,
	title = {Külső kamradrén},
	url = {https://m2.mtmt.hu/api/publication/34823721},
	author = {Ezer, Erzsébet},
	booktitle = {Súlyos baleseti agysérültek ellátása},
	unique-id = {34823721},
	year = {2024},
	pages = {163-169}
}

@article{MTMT:34493390,
	title = {Gravity sedimentation reveals functionally and morphologically different platelets in human blood},
	url = {https://m2.mtmt.hu/api/publication/34493390},
	author = {Ezer, Erzsébet and Schrick, Diana Zsuzsanna and Tőkés-Füzesi, Margit and Papp, István and Réger, Barbara and Molnár, Abigél and Ábrahám, Hajnalka and Koller, Ákos and Hársfalvi, Jolán and Kellermayer, Miklós and Molnár, Tihamér},
	doi = {10.1080/09537104.2023.2298341},
	journal-iso = {PLATELETS},
	journal = {PLATELETS},
	volume = {35},
	unique-id = {34493390},
	issn = {0953-7104},
	year = {2024},
	eissn = {1369-1635},
	orcid-numbers = {Koller, Ákos/0000-0003-3256-8701; Hársfalvi, Jolán/0000-0001-9940-4846; Kellermayer, Miklós/0000-0002-5553-6553}
}

@book{MTMT:34118867,
	title = {ANAESTHESIOLOGY AND INTENSIVE CARE THERAPY UNIVERSITY COURSE BOOK},
	url = {https://m2.mtmt.hu/api/publication/34118867},
	author = {Bátai, István and Bogár, Lajos and Csontos, Csaba and Ezer, Erzsébet and Molnár, Tihamér and Mühl, Diána and Almási, Róbert Gyula and Kövesi, Tamás and Márton, Sándor},
	publisher = {Pécsi Tudományegyetem Általános Orvostudományi Kar},
	unique-id = {34118867},
	year = {2022},
	orcid-numbers = {Almási, Róbert Gyula/0000-0002-1134-9932}
}

@article{MTMT:33195955,
	title = {Measurement invariance of six language versions of the post-traumatic stress disorder checklist for DSM-5 in civilians after traumatic brain injury},
	url = {https://m2.mtmt.hu/api/publication/33195955},
	author = {Bockhop, Fabian and Zeldovich, Marina and Cunitz, Katrin and Van Praag, Dominique and van der Vlegel, Marjolein and Beissbarth, Tim and Hagmayer, York and von Steinbuechel, Nicole},
	doi = {10.1038/s41598-022-20170-2},
	journal-iso = {SCI REP},
	journal = {SCIENTIFIC REPORTS},
	volume = {12},
	unique-id = {33195955},
	issn = {2045-2322},
	abstract = {Traumatic brain injury (TBI) is frequently associated with neuropsychiatric impairments such as symptoms of post-traumatic stress disorder (PTSD), which can be screened using self-report instruments such as the Post-Traumatic Stress Disorder Checklist for DSM-5 (PCL-5). The current study aims to inspect the factorial validity and cross-linguistic equivalence of the PCL-5 in individuals after TBI with differential severity. Data for six language groups (n ≥ 200; Dutch, English, Finnish, Italian, Norwegian, Spanish) were extracted from the CENTER-TBI study database. Factorial validity of PTSD was evaluated using confirmatory factor analyses (CFA), and compared between four concurrent structural models. A multi-group CFA approach was utilized to investigate the measurement invariance (MI) of the PCL-5 across languages. All structural models showed satisfactory goodness-of-fit with small between-model variation. The original DSM-5 model for PTSD provided solid evidence of MI across the language groups. The current study underlines the validity of the clinical DSM-5 conceptualization of PTSD and demonstrates the comparability of PCL-5 symptom scores between language versions in individuals after TBI. Future studies should apply MI methods to other sociodemographic (e.g., age, gender) and injury-related (e.g., TBI severity) characteristics to improve the monitoring and clinical care of individuals suffering from PTSD symptoms after TBI.},
	year = {2022},
	eissn = {2045-2322},
	orcid-numbers = {Barzó, Pál/0000-0001-8717-748X; Czeiter, Endre/0000-0002-9578-6944}
}

@article{MTMT:33163294,
	title = {Neurocognitive correlates of probable posttraumatic stress disorder following traumatic brain injury},
	url = {https://m2.mtmt.hu/api/publication/33163294},
	author = {Van Praag, Dominique L G and Wouters, Kristien and Van Den Eede, Filip and Wilson, Lindsay and Maas, Andrew I R},
	doi = {10.1016/j.bas.2021.100854},
	journal-iso = {BRAIN SPINE},
	journal = {BRAIN AND SPINE},
	volume = {2},
	unique-id = {33163294},
	issn = {2772-5294},
	abstract = {Neurocognitive problems associated with posttraumatic stress disorder (PTSD) can interact with impairment resulting from traumatic brain injury (TBI).We aimed to identify neurocognitive problems associated with probable PTSD following TBI in a civilian sample.The study is part of the CENTER-TBI project (Collaborative European Neurotrauma Effectiveness Research) that aims to better characterize TBI. For this cross-sectional study, we included patients of all severities aged over 15, and a Glasgow Outcome Score Extended (GOSE) above 3. Participants were assessed at six months post-injury on the PTSD Checklist-5 (PCL-5), the Trail Making Test (TMT), the Rey Auditory Verbal Learning Test (RAVLT) and the Cambridge Neuropsychological Test Automated Battery (CANTAB). Primary analysis was a complete case analysis. Regression analyses were performed to investigate the association between the PCL-5 and cognition.Of the 1134 participants included in the complete case analysis, 13.5% screened positive for PTSD. Probable PTSD was significantly associated with higher TMT-(B-A) (OR ​= ​1.35, 95% CI: 1.14-1.60, p ​< ​.001) and lower RAVLT-delayed recall scores (OR ​= ​0.74, 95% CI: 0.61-0.91, p ​= ​.004) after controlling for age, sex, psychiatric history, baseline Glasgow Coma Scale and education.Poorer performance on cognitive tests assessing task switching and, to a lesser extent, delayed verbal recall is associated with probable PTSD in civilians who have suffered TBI.},
	keywords = {STRESS; cognition; head injury; neuropsychology; posttraumatic stress disorder},
	year = {2022},
	orcid-numbers = {Barzó, Pál/0000-0001-8717-748X; Czeiter, Endre/0000-0002-9578-6944}
}