@{MTMT:1886997,
	title = {A személyi sérülésekből eredő kártérítési igények elbírálásnak jogi alapjai},
	url = {https://m2.mtmt.hu/api/publication/1886997},
	author = {Kereszty, Éva Margit},
	booktitle = {A felelősségbiztosítás orvosszakértői irányelvei},
	unique-id = {1886997},
	year = {1995},
	pages = {8-26}
}

@article{MTMT:1617926,
	title = {[Could tuberculosis again be an endemic disease in Hungary? (Model study 1968-1993)].. Népbetegség lehet-e ismét a tuberculosis Magyarországon? (Modellvizsgálat 1968-1993).},
	url = {https://m2.mtmt.hu/api/publication/1617926},
	author = {Abraham, E and Karacsonyi, L and Dinya, Elek},
	journal-iso = {ORV HETIL},
	journal = {ORVOSI HETILAP},
	volume = {136},
	unique-id = {1617926},
	issn = {0030-6002},
	abstract = {The aim of this examination started in an industrial district of Budapest with 100,000 inhabitants in 1968, was the determination of risk groups of pulmonary tuberculosis and the organisation of X-ray screening. The initial decrease in the incidence of pulmonary tuberculosis stopped (between 1981 and 1993) and, a minimal increase could be even demonstrated in the last years. The cause of the increase is explained by sociological factors (the influx of refugees, growth in the number of homeless people and of chronic alcoholics). The authors propose the modernization of the preventive and exploratory medical work: the use of computers in the lung-screening center and the institutionalisation of screening aimed at risk groups of pulmonary tuberculosis. In the population over forty years of age 92% of 408 new pulmonary tuberculosis patients, emerged from risk groups between 1981 and 1993, and only 8% from people who did not belong to the risk groups. The effectiveness of screening in the risk groups was 3 to 4 times higher than that of indiscriminate screening.},
	keywords = {Female; Male; Humans; Hungary/epidemiology; Risk Factors; History, 20th Century; Epidemiologic Methods; World Health Organization; Longitudinal Studies; Tuberculosis, Pulmonary/*epidemiology/history/mortality/radiography; *Mass Chest X-Ray; AIDS-Related Opportunistic Infections/epidemiology},
	year = {1995},
	eissn = {1788-6120},
	pages = {1329-1332},
	orcid-numbers = {Dinya, Elek/0000-0002-2620-8649}
}

@article{MTMT:1447277,
	title = {Apolipoprotein E and complement C3 polymorphism and their role in the response to gemfibrozil and low fat low cholesterol therapy},
	url = {https://m2.mtmt.hu/api/publication/1447277},
	author = {Németh, András and Szakrnary, K and Kramer, Judit and Dinya, Elek and Pados, G and Füst, György and Huettinger, M},
	doi = {10.1515/cclm.1995.33.11.799},
	journal-iso = {EUR J CLIN CHEM CLIN},
	journal = {EUROPEAN JOURNAL OF CLINICAL CHEMISTRY AND CLINICAL BIOCHEMISTRY},
	volume = {33},
	unique-id = {1447277},
	issn = {0939-4974},
	abstract = {Three different allelic variants of apolipoprotein E determine, in concert with other gene products, the levels of plasma lipoproteins. Recently, cleavage products of the complement C3 molecule have also been implicated in determining plasma triacylglycerol concentrations. This study presents data of an ongoing study to dissect the role of the apolipoprotein E gene locus in the response to low fat/low cholesterol diet combined with gemfibrozil treatment. In addition, for the first time, the significance of C3 allelic variants to such hypolipidaemic therapy response was analysed. To this end data from 81 obese hyperlipoproteinaemic patients (Fredrickson type II/A and B and type TV and V) confirmed the usefulness of the combined gemfibrozil/diet treatment and unveiled apolipoprotein E allele group specific therapy responses. The mean changes of lipid properties due to combined treatment was 15% for total cholesterol, 48% for triacylglycerols and 28% for atherogenic index. Division into hyperlipidaemia types according to Fredrickson and subgrouping into E2, E3 and E4 groups (apolipoprotein E2/2 and 2/3, apolipoprotein E3/3 and apolipoprotein E4/2 and 4/3 phenotype groups respectively) exposed pronounced differences from these mean changes, suggesting substantial influence of apolipoprotein E variants on this therapy. We observed triacylglycerol reductions of from 17% in type IIA-apolipoprotein E3 group patients up to 78% in the type IV and V-apolipoprotein E2 group. Thus it might be concluded the apolipoprotein E genotyping aides therapy success prediction. Although, low sample numbers in some subgroups obscures significance in this pilot study, significant therapy success emerges for the E3 and E4 group in type IV and V hyperlipidaemia and type IIB-apolipoprotein E3 homozygous patients can be predicted to respond better than apolipoprotein E2 carriers. Finally, we present evidence that positive changes of lipid properties are also determined by the 'fast' complement C3 allel (C3-F). Patients with complement factor C3-FS pattern respond better to treatment than patients with C3-SS configuration. In summary these data endorse the genotyping of apolipoprotein E alleles to predict maximal success of 'fibrate' treatment. In addition they argue strongly for further assessment of the involvement of complement C3 allelic variations in lipid homeostasis.},
	keywords = {Aged; Adult; Female; Male; PHENOTYPE; ARTICLE; ALLELE; human; ALLELES; Genotype; gene locus; Variation (Genetics); polymerase chain reaction; comparative study; priority journal; major clinical study; controlled study; OBESITY; Support, Non-U.S. Gov't; Sex Characteristics; Homeostasis; Combined Modality Therapy; Homozygote; Middle Age; cholesterol; triacylglycerol blood level; triacylglycerol; lipoprotein blood level; cholesterol blood level; Lipoproteins; complement component C3; fat intake; hyperlipidemia; apolipoprotein E; Antilipemic Agents; gemfibrozil; Apolipoproteins E; atherogenesis; dietary intake; Lipoproteins, HDL Cholesterol; Diet, Fat-Restricted; Complement 3; Cholesterol, Dietary; apolipoprotein e4; apolipoprotein e3; apolipoprotein e2; Polymorphism (Genetics); Apolipoproteins},
	year = {1995},
	pages = {799-804},
	orcid-numbers = {Dinya, Elek/0000-0002-2620-8649}
}

@{MTMT:2021449,
	title = {Sexual offences},
	url = {https://m2.mtmt.hu/api/publication/2021449},
	author = {Kereszty, Éva Margit and Törő, Klára Andrea},
	booktitle = {Lecture notes of forensic medicine},
	unique-id = {2021449},
	year = {1994},
	pages = {73-79},
	orcid-numbers = {Törő, Klára Andrea/0000-0002-0254-1439}
}

@{MTMT:2021447,
	title = {Forensic Points of reproduction},
	url = {https://m2.mtmt.hu/api/publication/2021447},
	author = {Kereszty, Éva Margit and Törő, Klára Andrea},
	booktitle = {Lecture notes of forensic medicine},
	unique-id = {2021447},
	year = {1994},
	pages = {58-61},
	orcid-numbers = {Törő, Klára Andrea/0000-0002-0254-1439}
}

@{MTMT:1898903,
	title = {Medico legal aspects of mental disease. Chapter X1V},
	url = {https://m2.mtmt.hu/api/publication/1898903},
	author = {Kereszty, Éva Margit},
	booktitle = {Lecture notes of forensic medicine},
	unique-id = {1898903},
	year = {1994},
	pages = {105-110}
}

@{MTMT:1898899,
	title = {Law and legal points of medicine. (Chapter 1)},
	url = {https://m2.mtmt.hu/api/publication/1898899},
	author = {Kereszty, Éva Margit},
	booktitle = {Lecture notes of forensic medicine},
	unique-id = {1898899},
	year = {1994},
	pages = {8-12}
}

@article{MTMT:1617927,
	title = {A Gevilon-terápia hatása az apolipoprotein E polimorfizmus függvényében},
	url = {https://m2.mtmt.hu/api/publication/1617927},
	author = {Németh, András and Dinya, Elek and Audikovszky, M and Pados, G and Mannfred, H},
	journal-iso = {ORV HETIL},
	journal = {ORVOSI HETILAP},
	volume = {135},
	unique-id = {1617927},
	issn = {0030-6002},
	abstract = {Our clinical trial was set out to estimate the apoE allele frequency by way of polymerase chain reaction and restriction enzyme isoform genotyping amongst 81 obese, hyperlipoproteinemic patients (treated with gemfibrozil + diet). The relative frequencies of the E2, E3, E4 alleles were 0.28, 0.61, 0.11 respectively (according to the Hardy-Weinberg equilibrium). Phenotypes with increased triglycerides levels were dominant in both sexes: IV + V > II/B > II/A (44.4%, 39.5%, 16.0%). Therapy significantly decreased the atherogen lipid values with differing efficacy among each genotype group: E3 > E4 > E2. Our findings demonstrate that in the II/A group there was no great change using the gemfibrozil + diet combination: whereas in IV + V group this combination was effective in each apoE isoform. In the case of II/B it is highly important to determine the apoE genotypes, because the therapy is only efficacious in the E33 group taking all lipid parameters into consideration.},
	keywords = {Adult; Female; Middle Aged; Male; Humans; PHENOTYPE; ALLELES; Genotype; polymerase chain reaction; Diet, Reducing; *Polymorphism, Genetic/genetics; Obesity, Morbid/metabolism/*therapy; Hyperlipoproteinemias/*therapy; Cyclohexanecarboxylic Acids/pharmacology/*therapeutic use; Apolipoproteins E/*metabolism},
	year = {1994},
	eissn = {1788-6120},
	pages = {735-741},
	orcid-numbers = {Dinya, Elek/0000-0002-2620-8649}
}

@article{MTMT:1447282,
	title = {C4B(ASTERISK)Q0 ALLOTYPE AS RISK FACTOR FOR MYOCARDIAL-INFARCTION},
	url = {https://m2.mtmt.hu/api/publication/1447282},
	author = {Kramer, Judit and Rajczy, K and Hegyi, L and Fulop, T and Mohácsi, Attila and Mezei, Z and Keltai, Mátyás and Blaskó, György and Ferenczy, E and Anh-Tuan, N and Füst, György},
	journal-iso = {BRIT MED J},
	journal = {BRITISH MEDICAL JOURNAL (BMJ)},
	volume = {309},
	unique-id = {1447282},
	issn = {0959-535X},
	keywords = {Aged; Adult; Female; Male; PREVALENCE; GENETICS; ARTICLE; MORTALITY; human; Hungary; Risk Factors; genetic analysis; risk factor; priority journal; major clinical study; heterozygote; controlled study; Support, Non-U.S. Gov't; Middle Age; myocardial infarction; heart infarction; complement component C4b; allotype},
	year = {1994},
	eissn = {1756-1833},
	pages = {313-314}
}

@article{MTMT:1617930,
	title = {Dohányzás és halálozás: longitudinális epidemiológiai vizsgálat (1975-1991)},
	url = {https://m2.mtmt.hu/api/publication/1617930},
	author = {Abraham, E and Karacsonyi, L and Dinya, Elek},
	journal-iso = {ORV HETIL},
	journal = {ORVOSI HETILAP},
	volume = {134},
	unique-id = {1617930},
	issn = {0030-6002},
	abstract = {The authors have started a basic screening examination in one of the industrial districts of Budapest in 1975. The district has nearly 100,000 inhabitants. The aim of the examination was to determine the risk factors of lung cancer. 30,566 persons have participated. The health of all patients has been followed up between 1975-1991. During this period 3,573 patients have moved out of the district. On 26,993 patients they have examined the death ratio, the prospective life-span, in the various age groups divided into subgroups according to their smoking habits. They have studied the formation of death rate in the four risk groups determined by them on the basis of the simultaneous examination of more lung cancer risk-factors (risk-free, moderate, high and superhigh risk groups). During these 16 years the death rate both among the heavy smokers and among patients in the high and superhigh risk groups has been two, three, even almost five times higher than that in the non-smoking and in the risk-free group of the relatively younger age groups of the population (born between 1921-1930 and 1931-1938). The knowledge of these connections makes economic calculations possible in respect of life and health insurance.},
	keywords = {Aged; Age Factors; Adult; Female; Middle Aged; Male; Humans; Hungary/epidemiology; Aged, 80 and over; Risk Factors; Smoking Cessation; Longitudinal Studies; Smoking/*mortality; Lung Neoplasms/etiology/mortality},
	year = {1993},
	eissn = {1788-6120},
	pages = {1137-1141},
	orcid-numbers = {Dinya, Elek/0000-0002-2620-8649}
}