@article{MTMT:32058440,
	title = {Silencing of Poly(ADP-Ribose) Polymerase-2 Induces Mitochondrial Reactive Species Production and Mitochondrial Fragmentation},
	url = {https://m2.mtmt.hu/api/publication/32058440},
	author = {Jankó, Laura and Kovács, Tünde and Laczik, Miklós and Sári, Zsanett Mercédesz and Ujlaki, Gyula and Kis, Nikoletta Gréta and Horváth, Ibolya and Antal, Miklós and Vigh, László and Bálint, Bálint László and Uray (Davis), Karen L. and Bay, Péter},
	doi = {10.3390/cells10061387},
	journal-iso = {CELLS-BASEL},
	journal = {CELLS},
	volume = {10},
	unique-id = {32058440},
	year = {2021},
	eissn = {2073-4409},
	orcid-numbers = {Bálint, Bálint László/0000-0002-6163-7190; Uray (Davis), Karen L./0000-0001-6997-459X; Bay, Péter/0000-0002-6191-6616}
}

@article{MTMT:31627406,
	title = {Indoxylsulfate, a metabolite of the microbiome, has cytostatic effects in breast cancer via activation of AHR and PXR receptors and induction of oxidative stress},
	url = {https://m2.mtmt.hu/api/publication/31627406},
	author = {Sári, Zsanett Mercédesz and Mikó, Edit and Kovács, Tünde and Boratkó, Anita and Ujlaki, Gyula and Jankó, Laura and Kiss, Borbála Katalin and Uray (Davis), Karen L. and Bay, Péter},
	doi = {10.3390/cancers12102915},
	journal-iso = {CANCERS},
	journal = {CANCERS},
	volume = {12},
	unique-id = {31627406},
	year = {2020},
	eissn = {2072-6694}
}

@article{MTMT:31607797,
	title = {Fecal expression of Escherichia coli lysine decarboxylase (LdcC) is downregulated in E-cadherin negative lobular breast carcinoma},
	url = {https://m2.mtmt.hu/api/publication/31607797},
	author = {Sári, Zsanett Mercédesz and Kovács, Tünde and Csonka, Tamás and Török, M and Sebő, É and Toth, J and Tóth, Dezső and Mikó, Edit and Kiss, Borbála Katalin and Szeőcs, Dóra and Uray (Davis), Karen L. and Karányi, Zsolt and Kovács, Ilona and Méhes, Gábor and Árkosy, Péter and Bay, Péter},
	doi = {10.1556/2060.2020.00016},
	journal-iso = {PHYSIOL INT},
	journal = {PHYSIOLOGY INTERNATIONAL},
	volume = {107},
	unique-id = {31607797},
	issn = {2498-602X},
	year = {2020},
	eissn = {2677-0164},
	pages = {349-358}
}

@article{MTMT:31408725,
	title = {Indolepropionic acid, a metabolite of the microbiome, has cytostatic properties in breast cancer by activating AHR and PXR receptors and inducing oxidative stress},
	url = {https://m2.mtmt.hu/api/publication/31408725},
	author = {Sári, Zsanett Mercédesz and Mikó, Edit and Kovács, Tünde and Jankó, Laura and Csonka, Tamás and Lente, G and Sebő, É and Toth, J and Tóth, Dezső and Árkosy, Péter and Boratkó, Anita and Ujlaki, Gyula and Török, M and Kovács, Ilona and Szabó, Judit and Kiss, Borbála Katalin and Méhes, Gábor and Goedert, JJ and Bay, Péter},
	doi = {10.3390/cancers12092411},
	journal-iso = {CANCERS},
	journal = {CANCERS},
	volume = {12},
	unique-id = {31408725},
	year = {2020},
	eissn = {2072-6694}
}

@{MTMT:31408665,
	title = {Treatment and diagnosis of breast cancer},
	url = {https://m2.mtmt.hu/api/publication/31408665},
	author = {Bay, Péter and Goedert, JJ and Kovács, Tünde and Mikó, Edit and Sári, Zsanett Mercédesz and Sebő, É and Tóth, J},
	unique-id = {31408665},
	year = {2020}
}

@article{MTMT:31156819,
	title = {Silencing of PARP2 blocks autophagic degradation},
	url = {https://m2.mtmt.hu/api/publication/31156819},
	author = {Jankó, Laura and Sári, Zsanett Mercédesz and Kovács, Tünde and Kis, Nikoletta Gréta and Szántó, Magdolna and Antal, Miklós and Juhász, Gábor and Bay, Péter},
	doi = {10.3390/cells9020380},
	journal-iso = {CELLS-BASEL},
	journal = {CELLS},
	volume = {9},
	unique-id = {31156819},
	year = {2020},
	eissn = {2073-4409},
	orcid-numbers = {Juhász, Gábor/0000-0001-8548-8874}
}

@{MTMT:30941569,
	title = {The microbiome as a component of the tumor microenvironment},
	url = {https://m2.mtmt.hu/api/publication/30941569},
	isbn = {9783030357269},
	author = {Kovács, Tünde and Mikó, Edit and Ujlaki, Gyula and Sári, Zsanett Mercédesz and Bay, Péter},
	booktitle = {Tumor Microenvironment: Recent advances},
	doi = {10.1007/978-3-030-35727-6_10},
	unique-id = {30941569},
	year = {2020},
	pages = {137-153}
}

@article{MTMT:30774202,
	title = {Lithocholic acid, a metabolite of the microbiome, increases oxidative stress in breast cancer},
	url = {https://m2.mtmt.hu/api/publication/30774202},
	author = {Kovács, Patrik Bence and Csonka, Tamás and Kovács, Tünde and Sári, Zsanett Mercédesz and Ujlaki, Gyula and Sipos, Adrienn and Karányi, Zsolt and Szeőcs, Dóra and Hegedűs, Csaba and Uray (Davis), Karen L. and Jankó, Laura and Kiss, M and Kiss, Borbála Katalin and Laoui, D and Virág, László and Méhes, Gábor and Bay, Péter and Mikó, Edit},
	doi = {10.3390/cancers11091255},
	journal-iso = {CANCERS},
	journal = {CANCERS},
	volume = {11},
	unique-id = {30774202},
	year = {2019},
	eissn = {2072-6694}
}

@article{MTMT:3357955,
	title = {Lithocholic acid, a bacterial metabolite reduces breast cancer cell proliferation and aggressiveness},
	url = {https://m2.mtmt.hu/api/publication/3357955},
	author = {Mikó, Edit and Vida, András and Kovács, Tünde and Ujlaki, Gyula and Trencsényi, György and Márton, Judit and Sári, Zsanett Mercédesz and Kovács, Patrik Bence and Boratkó, Anita and Hujber, Zoltán and Csonka, Tamás and Antal-Szalmás, Péter and Watanabe, M and Gombos, Imre and Csoka, B and Kiss, Borbála Katalin and Vigh, László and Szabó, Judit and Méhes, Gábor and Sebestyén, Anna and Goedert, JJ and Bay, Péter},
	doi = {10.1016/j.bbabio.2018.04.002},
	journal-iso = {BBA-BIOENERGETICS},
	journal = {BIOCHIMICA ET BIOPHYSICA ACTA-BIOENERGETICS},
	volume = {1859},
	unique-id = {3357955},
	issn = {0005-2728},
	abstract = {Our study aimed at finding a mechanistic relationship between the gut microbiome and breast cancer. Breast cancer cells are not in direct contact with these microbes, but disease could be influenced by bacterial metabolites including secondary bile acids that are exclusively synthesized by the microbiome and known to enter the human circulation. In murine and bench experiments, a secondary bile acid, lithocholic acid (LCA) in concentrations corresponding to its tissue reference concentrations (< 1 mu M), reduced cancer cell proliferation (by 10-20%) and VEGF production (by 37%), aggressiveness and metastatic potential of primary tumors through inducing mesenchymal-to-epithelial transition, increased antitumor immune response, OXPHOS and the TCA cycle. Part of these effects was due to activation of TGR5 by LCA. Early stage breast cancer patients, versus control women, had reduced serum LCA levels, reduced chenodeoxycholic acid to LCA ratio, and reduced abundance of the baiH (7 alpha/beta-hydroxysteroid dehydroxylase, the key enzyme in LCA generation) gene in fecal DNA, all suggesting reduced microbial generation of LCA in early breast cancer.},
	keywords = {RISK; PATHWAYS; LIVER; MALIGNANCY; GUT; breast cancer; Antibiotic use; lithocholic acid; PROSTATE-CANCER; microbiome; Intestinal microbiota; Biochemistry & Molecular Biology; Endothelial-mesenchymal transition; OXPHOS; BILE-ACIDS},
	year = {2018},
	eissn = {1879-2650},
	pages = {958-974},
	orcid-numbers = {Trencsényi, György/0000-0001-6456-6212; Sebestyén, Anna/0000-0001-8814-4794}
}