@article{MTMT:33286181, title = {Oxyhemoglobin and cerebral blood flow transients detect infarction in rat focal brain ischemia}, url = {https://m2.mtmt.hu/api/publication/33286181}, author = {Lückl, János and Baker, Wesley and Boda, Krisztina and Emri, Miklós and YodhArjun G Yodh, Arjun G and Greenberg, Joel H}, doi = {10.1016/j.neuroscience.2022.11.028}, journal-iso = {NEUROSCIENCE}, journal = {NEUROSCIENCE}, volume = {509}, unique-id = {33286181}, issn = {0306-4522}, abstract = {depolarizations (SD) refer to the near-complete depolarization of neurons that is associated with brain injuries such as ischemic stroke. The present gold standard for SD monitoring in humans is invasive electrocorticography (ECoG). A promising non-invasive alternative to ECoG is diffuse optical monitoring of SD -related flow and hemoglobin transients. To investigate the clinical utility of flow and hemoglobin transients, we analyzed their association with infarction in rat focal brain ischemia. Optical images of flow, oxy-hemoglobin, and deoxy-hemoglobin were continuously acquired with Laser Speckle and Optical Intrinsic Signal imaging for 2 h after photochemically induced distal middle cerebral artery occlusion in Sprague-Dawley rats (n = 10). Imag-ing was performed through a 6 x 6 mm window centered 3 mm posterior and 4 mm lateral to Bregma. Rats were sacrificed after 24 h, and the brain slices were stained for assessment of infarction. We mapped the infarcted area onto the imaging data and used nine circular regions of interest (ROI) to distinguish infarcted from non-infarcted tissue. Transients propagating through each ROI were characterized with six parameters (negative, positive, and total amplitude; negative and positive slope; duration). Transients were also classified into three morphology types (positive monophasic, biphasic, negative monophasic). Flow transient morphology, positive amplitude, positive slope, and total amplitude were all strongly associated with infarction (p < 0.001). Associations with infarction were also observed for oxy-hemoglobin morphology, oxy-hemoglobin positive amplitude and slope, and deoxy-hemoglobin positive slope and duration (all p < 0.01). These results suggest that flow and hemoglobin transients accompanying SD have value for detecting infarction.(c) 2022 IBRO. Published by Elsevier Ltd. All rights reserved.}, year = {2023}, eissn = {1873-7544}, pages = {132-144}, orcid-numbers = {Lückl, János/0000-0001-8094-771X; Boda, Krisztina/0000-0002-9937-0460} } @article{MTMT:32788547, title = {PET/MRI in the Presurgical Evaluation of Patients with Epilepsy: A Concordance Analysis}, url = {https://m2.mtmt.hu/api/publication/32788547}, author = {Borbély, Katalin and Emri, Miklós and Kenessey, István and Tóth, Márton and Singer, Júlia and Barsi, Péter and Vajda, Zsolt and Pál, Endre and Tóth, Zoltán and Beyer, Thomas and Dóczi, Tamás Péter and Bajzik, Gábor and Fabó, Dániel and Janszky, József Vladimír and Jordán, Zsófia and Fajtai, Dániel and Kelemen, Anna and Juhos, Vera and Wintermark, Max and Nagy, Ferenc and Moizs, Mariann and Nagy , Dávid Gergő and Lückl, János and Repa, Imre}, doi = {10.3390/biomedicines10050949}, journal-iso = {BIOMEDICINES}, journal = {BIOMEDICINES}, volume = {10}, unique-id = {32788547}, keywords = {epilepsy surgery; presurgical evaluation; Concordance analysis; metabolic PET; Preoperative workflow; medically refractory focal epilepsy; MRI-negative patients; discordant electroclinical and MRI data; hybrid [18F]-FDG PET/MRI; epilepsy team}, year = {2022}, eissn = {2227-9059}, orcid-numbers = {Borbély, Katalin/0000-0002-1675-4128; Kenessey, István/0000-0002-6963-8489; Barsi, Péter/0000-0002-3574-9973; Tóth, Zoltán/0000-0002-8096-3813; Bajzik, Gábor/0000-0002-0996-9294; Fabó, Dániel/0000-0001-5141-5351; Janszky, József Vladimír/0000-0001-6100-832X; Fajtai, Dániel/0000-0001-7591-3101; Kelemen, Anna/0000-0003-3942-3409; Moizs, Mariann/0000-0003-1055-5918; Lückl, János/0000-0001-8094-771X} } @article{MTMT:32781700, title = {Spreading depolarizations in ischaemia after subarachnoid haemorrhage, a diagnostic phase III study}, url = {https://m2.mtmt.hu/api/publication/32781700}, author = {Dreier, Jens P. and Winkler, Maren K. L. and Major, Sebastian and Horst, Viktor and Lublinsky, Svetlana and Kola, Vasilis and Lemale, Coline L. and Kang, Eun-Jeung and Maslarova, Anna and Salur, Irmak and Lückl, János and Platz, Johannes and Jorks, Devi and Oliveira-Ferreira, Ana I. and Schoknecht, Karl and Reiffurth, Clemens and Milakara, Denny and Wiesenthal, Dirk and Hecht, Nils and Dengler, Nora F. and Liotta, Agustin and Wolf, Stefan and Kowoll, Christina M. and Schulte, André P. and Santos, Edgar and Güresir, Erdem and Unterberg, Andreas W. and Sarrafzadeh, Asita and Sakowitz, Oliver W. and Vatter, Hartmut and Reiner, Michael and Brinker, Gerrit and Dohmen, Christian and Shelef, Ilan and Bohner, Georg and Scheel, Michael and Vajkoczy, Peter and Hartings, Jed A. and Friedman, Alon and Martus, Peter and Woitzik, Johannes}, doi = {10.1093/brain/awab457}, journal-iso = {BRAIN}, journal = {BRAIN}, volume = {145}, unique-id = {32781700}, issn = {0006-8950}, abstract = {Focal brain damage after aneurysmal subarachnoid haemorrhage predominantly results from intracerebral haemorrhage, and early and delayed cerebral ischaemia. The prospective, observational, multicentre, cohort, diagnostic phase III trial, DISCHARGE-1, primarily investigated whether the peak total spreading depolarization-induced depression duration of a recording day during delayed neuromonitoring (delayed depression duration) indicates delayed ipsilateral infarction. Consecutive patients (n = 205) who required neurosurgery were enrolled in six university hospitals from September 2009 to April 2018. Subdural electrodes for electrocorticography were implanted. Participants were excluded on the basis of exclusion criteria, technical problems in data quality, missing neuroimages or patient withdrawal (n = 25). Evaluators were blinded to other measures. Longitudinal MRI, and CT studies if clinically indicated, revealed that 162/180 patients developed focal brain damage during the first 2 weeks. During 4.5 years of cumulative recording, 6777 spreading depolarizations occurred in 161/180 patients and 238 electrographic seizures in 14/180. Ten patients died early; 90/170 developed delayed infarction ipsilateral to the electrodes. Primary objective was to investigate whether a 60-min delayed depression duration cut-off in a 24-h window predicts delayed infarction with >0.60 sensitivity and >0.80 specificity, and to estimate a new cut-off. The 60-min cut-off was too short. Sensitivity was sufficient [= 0.76 (95% confidence interval: 0.65-0.84), P = 0.0014] but specificity was 0.59 (0.47-0.70), i.e. <0.80 (P < 0.0001). Nevertheless, the area under the receiver operating characteristic (AUROC) curve of delayed depression duration was 0.76 (0.69-0.83, P < 0.0001) for delayed infarction and 0.88 (0.81-0.94, P < 0.0001) for delayed ischaemia (reversible delayed neurological deficit or infarction). In secondary analysis, a new 180-min cut-off indicated delayed infarction with a targeted 0.62 sensitivity and 0.83 specificity. In awake patients, the AUROC curve of delayed depression duration was 0.84 (0.70-0.97, P = 0.001) and the prespecified 60-min cut-off showed 0.71 sensitivity and 0.82 specificity for reversible neurological deficits. In multivariate analysis, delayed depression duration (beta = 0.474, P < 0.001), delayed median Glasgow Coma Score (beta = -0.201, P = 0.005) and peak transcranial Doppler (beta = 0.169, P = 0.016) explained 35% of variance in delayed infarction. Another key finding was that spreading depolarization-variables were included in every multiple regression model of early, delayed and total brain damage, patient outcome and death, strongly suggesting that they are an independent biomarker of progressive brain injury. While the 60-min cut-off of cumulative depression in a 24-h window indicated reversible delayed neurological deficit, only a 180-min cut-off indicated new infarction with >0.60 sensitivity and >0.80 specificity. Although spontaneous resolution of the neurological deficit is still possible, we recommend initiating rescue treatment at the 60-min rather than the 180-min cut-off if progression of injury to infarction is to be prevented.Focal damage after subarachnoid haemorrhage results from intracerebral haemorrhage and cerebral ischaemia. In a prospective, observational, multicentre, diagnostic phase III trial, DISCHARGE-1, Dreier et al. examine whether monitoring cortical spreading depolarizations can predict delayed infarction-and thus poor outcomes.}, year = {2022}, eissn = {1460-2156}, pages = {1264-1284}, orcid-numbers = {Dreier, Jens P./0000-0001-7459-2828; Major, Sebastian/0000-0003-0970-1308; Lückl, János/0000-0001-8094-771X; Milakara, Denny/0000-0003-3148-6122; Hecht, Nils/0000-0001-9989-649X; Brinker, Gerrit/0000-0002-0184-0680; Shelef, Ilan/0000-0002-8398-6455; Friedman, Alon/0000-0001-6907-1040} } @article{MTMT:32663200, title = {Migraine Aura, Transient Ischemic Attacks, Stroke, and Dying of the Brain Share the Same Key Pathophysiological Process in Neurons Driven by Gibbs–Donnan Forces, Namely Spreading Depolarization}, url = {https://m2.mtmt.hu/api/publication/32663200}, author = {Lemale, Coline L. and Lückl, János and Horst, Viktor and Reiffurth, Clemens and Major, Sebastian and Hecht, Nils and Woitzik, Johannes and Dreier, Jens P.}, doi = {10.3389/fncel.2022.837650}, journal-iso = {FRONT CELL NEUROSCI}, journal = {FRONTIERS IN CELLULAR NEUROSCIENCE}, volume = {16}, unique-id = {32663200}, issn = {1662-5102}, year = {2022}, eissn = {1662-5102}, orcid-numbers = {Lückl, János/0000-0001-8094-771X} } @article{MTMT:32219141, title = {The role of hybrid FDG-PET/MRI on decision-making in presurgical evaluation of drug-resistant epilepsy}, url = {https://m2.mtmt.hu/api/publication/32219141}, author = {Tóth, Márton and Barsi, Péter and Tóth, Zoltán and Borbély, Katalin and Lückl, János and Emri, Miklós and Repa, Imre and Janszky, József Vladimír and Dóczi, Tamás Péter and Horváth, Zsolt and Halász, Péter and Juhos, Vera and Trischlerné Gyimesi, Csilla and Bóné, Beáta and Kuperczkó, Diána and Horváth, Réka and Nagy, Ferenc and Kelemen, Anna and Jordán, Zsófia and Újvári, Ákos and Hagiwara, Koichi and Isnard, Jean and Pál, Endre and Fekésházy, Attila and Fabó, Dániel and Vajda, Zsolt}, doi = {10.1186/s12883-021-02352-z}, journal-iso = {BMC NEUROL}, journal = {BMC NEUROLOGY}, volume = {21}, unique-id = {32219141}, issn = {1471-2377}, abstract = {When MRI fails to detect a potentially epileptogenic lesion, the chance of a favorable outcome after epilepsy surgery becomes significantly lower (from 60 to 90% to 20-65%). Hybrid FDG-PET/MRI may provide additional information for identifying the epileptogenic zone. We aimed to investigate the possible effect of the introduction of hybrid FDG-PET/MRI into the algorithm of the decision-making in both lesional and non-lesional drug-resistant epileptic patients.In a prospective study of patients suffering from drug-resistant focal epilepsy, 30 nonlesional and 30 lesional cases with discordant presurgical results were evaluated using hybrid FDG-PET/MRI.The hybrid imaging revealed morphological lesion in 18 patients and glucose hypometabolism in 29 patients within the nonlesional group. In the MRI positive group, 4 patients were found to be nonlesional, and in 9 patients at least one more epileptogenic lesion was discovered, while in another 17 cases the original lesion was confirmed by means of hybrid FDG-PET/MRI. As to the therapeutic decision-making, these results helped to indicate resective surgery instead of intracranial EEG (iEEG) monitoring in 2 cases, to avoid any further invasive diagnostic procedures in 7 patients, and to refer 21 patients for iEEG in the nonlesional group. Hybrid FDG-PET/MRI has also significantly changed the original therapeutic plans in the lesional group. Prior to the hybrid imaging, a resective surgery was considered in 3 patients, and iEEG was planned in 27 patients. However, 3 patients became eligible for resective surgery, 6 patients proved to be inoperable instead of iEEG, and 18 cases remained candidates for iEEG due to the hybrid FDG-PET/MRI. Two patients remained candidates for resective surgery and one patient became not eligible for any further invasive intervention.The results of hybrid FDG-PET/MRI significantly altered the original plans in 19 of 60 cases. The introduction of hybrid FDG-PET/MRI into the presurgical evaluation process had a potential modifying effect on clinical decision-making.Trial registry: Scientific Research Ethics Committee of the Medical Research Council of Hungary.008899/2016/OTIG . Date of registration: 08 February 2016.}, keywords = {clinical decision-making; epilepsy surgery; Drug-resistant epilepsy; Hybrid FDG-PET/MRI; Preoperative workflow}, year = {2021}, eissn = {1471-2377}, orcid-numbers = {Barsi, Péter/0000-0002-3574-9973; Tóth, Zoltán/0000-0002-8096-3813; Borbély, Katalin/0000-0002-1675-4128; Lückl, János/0000-0001-8094-771X; Janszky, József Vladimír/0000-0001-6100-832X; Kelemen, Anna/0000-0003-3942-3409; Fabó, Dániel/0000-0001-5141-5351} } @article{MTMT:32007928, title = {Subclinical epileptiform activity accelerates the progression of Alzheimer’s disease: a long-term EEG study}, url = {https://m2.mtmt.hu/api/publication/32007928}, author = {Horváth, András Attila and Papp, Anikó and Zsuffa, János and Szűcs, Anna and Lückl, János and Radai, Ferenc and Nagy, Ferenc and Hidasi, Zoltán and Csukly, Gábor and Barcs, Gábor and Kamondi, Anita}, doi = {10.1016/j.clinph.2021.03.050}, journal-iso = {CLIN NEUROPHYSIOL}, journal = {CLINICAL NEUROPHYSIOLOGY}, volume = {132}, unique-id = {32007928}, issn = {1388-2457}, year = {2021}, eissn = {1872-8952}, pages = {1982-1989}, orcid-numbers = {Papp, Anikó/0000-0003-2052-9107; Szűcs, Anna/0000-0002-9990-5787; Lückl, János/0000-0001-8094-771X; Hidasi, Zoltán/0000-0002-7798-0145; Csukly, Gábor/0000-0002-5006-9407; Kamondi, Anita/0000-0001-9860-730X} } @misc{MTMT:31686790, title = {The Role of Hybrid FDG-PET/MRI on Decision-Making in Presurgical Evaluation of Drug-Resistant Epilepsy}, url = {https://m2.mtmt.hu/api/publication/31686790}, author = {Tóth, Márton and Barsi, Péter and Tóth, Zoltán and Borbély, Katalin and Lückl, János and Emri, Miklós and Repa, Imre and Janszky, Jozsef and Doczi, Tamas and Horvath, Zsolt and Halasz, Peter and Juhos, Vera and Gyimesi, Csilla and Bone, Beata and Kuperczkó, Diána and Horvath, Reka and Nagy, Ferenc and Kelemen, Anna and Jordan, Zsoa and Ujvari, Akos and Hagiwara, Koichi and Isnard, Jean and Pal, Endre and Fekésházy, Attila and Fabo, Daniel and Vajda, Zsolt}, unique-id = {31686790}, year = {2020}, orcid-numbers = {Barsi, Péter/0000-0002-3574-9973; Tóth, Zoltán/0000-0002-8096-3813; Borbély, Katalin/0000-0002-1675-4128; Lückl, János/0000-0001-8094-771X} } @article{MTMT:31138704, title = {Quantitative Signal Intensity in Fluid-Attenuated Inversion Recovery and Treatment Effect in the WAKE-UP Trial}, url = {https://m2.mtmt.hu/api/publication/31138704}, author = {Cheng, Bastian and Boutitie, Florent and Nickel, Alina and Wouters, Anke and Cho, Tae-Hee and Ebinger, Martin and Endres, Matthias and Fiebach, Jochen B. and Fiehler, Jens and Galinovic, Ivana and Puig, Josep and Thijs, Vincent and Lemmens, Robin and Muir, Keith W. and Nighoghossian, Norbert and Pedraza, Salvador and Simonsen, Claus Z. and Gerloff, Christian and Thomalla, Götz and Golsari, Amir and Alegiani, Anna and Beck, Christoph and Choe, Chi-un and Voget, Dieke and Hoppe, Julia and Schröder, Julian and Rozanski, Michal and Nave, Alexander H. and Wollboldt, Christian and van Sloten, Isabelle and Göhler, Jos and Herm, Juliane and Jungehülsing, Jan and Kröber, Jan M. and Schurig, Johannes and Koehler, Luzie and Schlemm, Ludwig and Knops, Michael and Roennefarth, Maria and Ipsen, Nils and Harmel, Peter and Bathe-Peters, Rouven and Fleischmann, Robert and Ganeshan, Ramanan and Geran, Rohat and Hellwig, Simon and Schmidt, Sein and Tütüncü, Serdar and Krause, Thomas and Gramse, Verena and Röther, Joachim and Michels, Peter and Michalski, Dominik and Pelz, Johann and Schulz, Andreas and Hobohm, Carsten and Weise, Christopher and Weise, Gesa and Orthgieß, Johannes and Pomrehn, Katrin and Wegscheider, Mirko and Mueller, Anne K. and Hennerici, Michael and Griebe, Martin and Alonso, Angelika and Filipov, Alexandra and Marzina, Annika and Anders, Bettina and Bähr, Christian and Hoyer, Carolin and Schwarzbach, Christopher and Weber, Claudia and Hornberger, Eva and Pledl, Hans-Werner and Klockziem, Matti and Stuermlinger, Markus and Wittayer, Matthias and Wolf, Marc and Meyer, Nadja and Eisele, Philipp and Steinert, Susanne and Sauer, Tamara and Held, Valentin and Ringleb, Peter and Nagel, Simon and Veltkamp, Roland and Schwarting, Sönke and Schwarz, Alexander and Gumbinger, Christoph and Hametner, Christian and Amiri, Hemasse and Purrucker, Jan and Ciatipis, Mareva and Menn, Oliver and Mundiyanapurath, Sibu and Schieber, Simon and Kessler, Tobias and Reiff, Tilman and Panitz, Verena and Singer, Oliver and Foerch, Christian and Lauer, Arne and Männer, Anika and Seiler, Alexander and Guerzoglu, Damla and Schäfer, Jan H. and Filipski, Katharina and Lorenz, Matthias and Kurka, Natalia and Zeiner, Pia and Pfeilschifter, Waltraud and Dziewas, Rainer and Minnerup, Jens and Albiker, Christian and Ritter, Martin and Seidel, Matthias and Dittrich, Ralf and Kallmünzer, Bernd and Bobinger, Tobias and Madzar, Dominik and Stark, David and Sembill, Jochen and Macha, Kosmas and Winder, Klemens and Breuer, Lorenz and Koehrmann, Martin and Spruegel, Maximilian and Gerner, Stefan and Kraft, Peter and Mackenrodt, Daniel and Kleinschnitz, Christoph and Elhfnawy, Ahmed and Heinen, Florian and Gunreben, Ignaz and Poli, Sven and Ziemann, Ulf and Gaenslen, Alexandra and Schlak, Dennis and Haertig, Florian and Russo, Francesca and Richter, Hardy and Ebner, Matthias and Ribitsch, Martin and Wolf, Martin and Weimar, Christian and Zegarac, Vesna and Chen, Hsin-Chieh and Althaus, Katharina and Neugebauer, Hermann and Jüttler, Eric and Meier, Julia and Stösser, Sebastian and Puetz, Volker and Bodechtel, Ulf and Ostergaard, Leif and Møller, Anette and Damgaard, Dorte and Dupont, Kristina Hougaard and Poulsen, Marika and Hjort, Niels and de Morales, Noelia Ruiz and von Weitzel, Paul and Harbo, Thomas and Marstrand, Jacob and Hansen, Andreas and Christensen, Hanne and Aegidius, Karen and Jeppesen, Lise and Meden, Per and Rosenbaum, Sverre and Iversen, Helle and Hansen, Jakob and Michelsen, Lene and Truelsen, Thomas and Modrau, Boris and Vestergaard, Karsten and Oppel, Lorenz and Sygehus, Aalborg and Aalborg, Syd and Swinnen, Bart and Smets, Ide and Demeestere, Jelle and Dobbels, Laurens and Brouns, Raf and De Smedt, Ann and DeKeyser, Jacques and Yperzeele, Laetita and Van Hooff, Robbert- Jan and Peeters, Andre and Dusart, Anne and Etexberria, Ana and Hanseeuw, Bernard and London, Frederic and Leempoel, Jonathan and Hohenbichler, Katharina and Younan, Nadia and Maqueda, Vicky and Laloux, Patrice and De Coene, Beatrijs and De Maeseneire, Charlotte and Turine, Gaetane and Vandermeeren, Yves and De Klippel, Nina and Willems, Chris and de Hollander, Isabel and Soors, Peter and Hermans, Sylvia and Hemelsoet, Dimitri and Desfontaines, Philippe and Vanacker, Peter and Rutgers, Matthieu and Druart, Charlotte and Peeters, Dirk and Bruneel, Bart and Vancaester, Evelien and Vanhee, Frederik and Meersman, Guy and Bourgeois, Paul and Vanderdonckt, Patrick and Benoit, Amandine and Derex, Laurent and Mechthouff, Laura and Berhoune, Nadia and Ritzenthaler, Thomas and Amarenco, Pierre and Hobeanu, Cristina and Gancedo, Elena Meseguer and Calvet, David and Ladoux, Alexandre and Machet, Alexandre and Lamy, Catherine and Mellerio, Charles and Oppenheim, Catherine and Rodriguez-Regent, Christine and Bodiguel, Eric and Turc, Guillaume and Birchenall, Julia and Legrand, Laurence and Morin, Ludovic and Edjali-Goujon, Myriam and Naggara, Oliver and Raphaelle, Soulliard and Godon-Hardy, Sylvie and Domigo, Valérie and Guiraud, Vincent and Samson, Yves and Leger, Anne and Rosso, Charlotte and Baronnet-Chauvet, Flore and Crozier, Sophie and Deltour, Sandrine and Yger, Marion and Sibon, Igor and Renou, Pauline and Sagnier, Sharmilla and Zuber, Mathieu and Tamazyan, Ruben and Rodier, Gilles and Morel, Nathalie and Felix, Sandra and Vadot, Wilfried and Wolff, Valerie and Aniculaesei, Adriana and Yalo, Bertrand and Bindila, Daniela and Quenardelle, Veronique and Blanc-Lasserre, Karine and Landrault, Emmanuelle and Breynaert, Ludovic and Cakmak, Serkan and Peysson, Stéphane and Viguier, Alain and Lebely, Claire and Raposo, Nicolas and Vallet, Anne-Evelyne and Vallet, Pauline and Brugirard, Sébastian and Cheripelli, Bharath and Kalladka, Dheeraj and Moreton, Fiona and Dani, Krishna and Tawil, Salwa El and Ramachandran, Sankaranarayanan and Huang, Xuya and Warburton, Elisabeth and Evans, Nicholas and Perry, Richard and Patel, Bhavni and Cloud, Geoffrey and Pereira, Anthony and Moynihan, Barry and Lovelock, Caroline and Choy, Lillian and Khan, Usman and Roffe, Christine and Tyrell, Pippa and Smith, Craig and Dixit, Anand and Louw, Stephen and Broughton, David and Shetty, Ashit and Appleton, Jason and Sprigg, Nikola and Acosta, Boris Raul and van Eendenburg, Cecile and Leal, Joaquin Serena and Mar Castellanos Rodrigo, Maria del and Izaga, Mikel Terceno and Guillamon, Olga Belchí and Arenillas, Juan and Calleja, Ana and Cortijo, Elisa and Mulero, Patricia and de la Ossa, Natalia Pérez and Garrido, Alicia and Martinez, Alicia and Esperón, Carlos García and Guerrero, Cristina and Carrera, David and Vilas, Dolores and Lopez-cancio, Elena and Palomeras, Ernest and Lucente, Giuseppe and Gomis, Meritxell and Isern, Irina and Becerra, Juan L. and Vicente, Jose Hervas and Sánchez, Josep and Dorado, Laura and Grau, Laia and Ispierto, Lourdes and Prats, Luis and Almendrote, Miriam and Hernández, María and Jimenez, Marta and Sánchez, Manuel Lozano and Torne, Monica Millán and Presas, Silvia and Ustrell, Xavier and Pellisé, Anna and Navalpotro, Irene and Luna, Alain and Schonewille, Wouter and Nederkoorn, Paul and Majoie, Charles and van den Berg, Lucie and van den Berg, Sophie and Zonneveld, Thomas and Remmers, Michel and Fazekas, Franz and Pichler, Alexander and Fandler, Simon and Gattringer, Thomas and Mutzenbach, Johannes and Weber, Jörg and Höfner, Elmar and Kohlfürst, Heinz and Weinstich, Karin and Kellert, Lars and Bayer-Karpinska, Anna and Opherk, Christian and Wollenweber, Frank and Klein, Matthias and Neumann- Haefelin, Tobias and Pierskalla, Alexandra and Harloff, Andreas and Bardutzky, Jürgen and Buggle, Florian and von Schrader, Jörg and Kollmar, Rainer and Schill, Josef and Löbbe, Anna-Mareike and Moulin, Thierry and Bouamra, Benjamin and Bonnet, Louise and Touzé, Emmanuel and Bonnet, Anne-Laure and Touze, Emmanuel and Cogez, Julien and Li, Lin and Guettier, Sophie and Kar, Arindam and Sivagnanaratham, Aravinth and Geraghty, Olivia and Bojaryn, Urszula and Nallasivan, Arumugam and Gonzales, Miguel Blanco and Rodríguez-Yáñez, Manuel and Tembl, Jose and Gorriz, David and Oberndorfer, Stefan and Prohaska, Elisabeth}, doi = {10.1161/STROKEAHA.119.027390}, journal-iso = {STROKE}, journal = {STROKE}, volume = {51}, unique-id = {31138704}, issn = {0039-2499}, year = {2020}, eissn = {1524-4628}, pages = {209-215}, orcid-numbers = {Lückl, János/0000-0001-8094-771X} } @CONFERENCE{MTMT:30747579, title = {MULTIMODÁLIS VIZUALIZÁCIÓS ÉS STATISZTIKAI MÓDSZEREK FEJLESZTÉSE AZ EPILEPSZIA DIAGNOSZTIKA SZÁMÁRA}, url = {https://m2.mtmt.hu/api/publication/30747579}, author = {Emri, Miklós and Aranyi, Sándor Csaba and Fajtai, Dániel and Tóth, Márton and Lückl, János and Vajda, Zsolt and Repa, Imre and Tóth, Zoltán}, booktitle = {MONT XXI. kongresszus 2019}, unique-id = {30747579}, year = {2019}, pages = {72-72}, orcid-numbers = {Aranyi, Sándor Csaba/0000-0001-9569-5404; Fajtai, Dániel/0000-0001-7591-3101; Lückl, János/0000-0001-8094-771X; Tóth, Zoltán/0000-0002-8096-3813} } @CONFERENCE{MTMT:30757620, title = {Neurológiai indikációjú DOPA PET/MR vizsgálatokkal szerzett kezdeti tapasztalatok}, url = {https://m2.mtmt.hu/api/publication/30757620}, author = {Tóth, Zoltán and Fekésházy, Attila and Mendly, József and Emri, Miklós and Fajtai, Dániel and Lukács, Gábor and Lückl, János and Bajzik, Gábor and Hadjiev, Janaki and Repa, Imre and Kovács, Árpád}, booktitle = {Magyar Neurológiai Társaság XXVI. Kongresszusa és Továbbképző Kurzusa}, unique-id = {30757620}, year = {2018}, pages = {44-45}, orcid-numbers = {Tóth, Zoltán/0000-0002-8096-3813; Fajtai, Dániel/0000-0001-7591-3101; Lückl, János/0000-0001-8094-771X; Bajzik, Gábor/0000-0002-0996-9294; Hadjiev, Janaki/0000-0003-4419-353X; Kovács, Árpád/0000-0002-8469-5764} }