@article{MTMT:30746292,
	title = {Efficacy and safety of long pulse 1064 and 2940 nm lasers in noninvasive lipolysis and skin tightening},
	url = {https://m2.mtmt.hu/api/publication/30746292},
	author = {Vas, Krisztina and Besenyi, Zsuzsanna and Urbán, Szabolcs and Badawi, Ashraf Mohamed and Pávics, László and Erős, Gábor and Kemény, Lajos},
	doi = {10.1002/jbio.201900083},
	journal-iso = {J BIOPHOTONICS},
	journal = {JOURNAL OF BIOPHOTONICS},
	volume = {12},
	unique-id = {30746292},
	issn = {1864-063X},
	abstract = {Noninvasive body shaping is becoming a growing demand. The aim of this study was to investigate the efficacy and safety of the combined treatments of 1064 nm Nd:YAG and 2940 nm Er:YAG in noninvasive lipolysis and skin tightening. Ten females were enrolled, and all women's side of the waist or the lower part of the abdomen were treated. In the first step, the 1064 nm Nd:YAG was used. As a second step, the 2940 nm Er:YAG laser was applied. Each woman was treated four times, once every 2 weeks. The effects were determined by comparative photo documentation, waist circumference measurement, two-dimensional B-mode ultrasonography and low-dose native computer tomography (CT), whereas body fat was monitored with bioelectric impedance. The tissue firmness was measured by ultrasound shear wave elastography. Combined laser treatment significantly reduced waist circumference and total body fat. Ultrasonography has revealed that the treatment considerably decreased fat thickness and improved skin stiffness in the treated region. Subcutaneous fat volume, measured by low-dose CT, displayed a moderate decrease in the waist region. The combined 1064 nm Nd:YAG and 2940 nm Er:YAG laser treatment results in the reduction of fat tissue and tightens the skin as confirmed by objective measurements.},
	keywords = {Nd:YAG laser; Er:YAG laser; fat reduction; skin stiffness},
	year = {2019},
	eissn = {1864-0648},
	orcid-numbers = {Besenyi, Zsuzsanna/0000-0001-9115-9620; Urbán, Szabolcs/0000-0001-5463-6811; Pávics, László/0000-0002-7319-1667; Kemény, Lajos/0000-0002-2119-9501}
}

@article{MTMT:30729353,
	title = {Új terápiás lehetőségek a plasztikai sebészet és a dermatológia határterületén},
	url = {https://m2.mtmt.hu/api/publication/30729353},
	author = {Varga, János and Bende, Balázs and Altmayer, Anita and Gaál, Magdolna and Kis, Erika and Kocsis, Ádám László and Mohos, Gábor and Varga, Ákos and Vas, Krisztina and Veréb, Zoltán and Kemény, Lajos},
	doi = {10.7188/bvsz.2019.95.2.7},
	journal-iso = {BVSZ},
	journal = {BŐRGYÓGYÁSZATI ÉS VENEROLÓGIAI SZEMLE},
	volume = {95},
	unique-id = {30729353},
	issn = {0006-7768},
	year = {2019},
	eissn = {2064-261X},
	pages = {69-73},
	orcid-numbers = {Veréb, Zoltán/0000-0002-9518-2155; Kemény, Lajos/0000-0002-2119-9501}
}

@article{MTMT:3333145,
	title = {Successful Treatment of Autoimmune Urticaria with Low-Dose Prednisolone Therapy Administered for a Few Months: A Case Series of 42 Patients},
	url = {https://m2.mtmt.hu/api/publication/3333145},
	author = {Vas, Krisztina and Altmayer, Anita and Mihályi, Lilla and Garaczi, Edina and Kinyó, Ágnes and Jakobicz, Eszter and Husz, Sándor and Kemény, Lajos and Csörgő Sándorné Bata, Zsuzsanna},
	doi = {10.1159/000484085},
	journal-iso = {DERMATOLOGY},
	journal = {DERMATOLOGY},
	volume = {233},
	unique-id = {3333145},
	issn = {1018-8665},
	year = {2017},
	eissn = {1421-9832},
	pages = {419-424},
	orcid-numbers = {Mihályi, Lilla/0000-0001-6215-4572; Kinyó, Ágnes/0000-0002-9827-2690; Jakobicz, Eszter/0000-0001-8641-4068; Husz, Sándor/0000-0001-9384-3020; Kemény, Lajos/0000-0002-2119-9501; Csörgő Sándorné Bata, Zsuzsanna/0000-0002-3732-1743}
}

@article{MTMT:3246999,
	title = {Delineating the genetic heterogeneity of OCA in Hungarian patients},
	url = {https://m2.mtmt.hu/api/publication/3246999},
	author = {Fábos, Beáta and Farkas, Katalin and Tóth, Lola and Sulák, Adrienn and Tripolszki, Kornélia and Tihanyi, M and Németh, Réka and Vas, Krisztina and Csoma, Zsanett and Kemény, Lajos and Széll, Márta and Nagy, Nikoletta},
	doi = {10.1186/s40001-017-0262-0},
	journal-iso = {EUR J MED RES},
	journal = {EUROPEAN JOURNAL OF MEDICAL RESEARCH},
	volume = {22},
	unique-id = {3246999},
	issn = {0949-2321},
	abstract = {BACKGROUND: Oculocutaneous albinism (OCA) is a clinically and genetically heterogenic group of pigmentation abnormalities characterized by variable hair, skin, and ocular hypopigmentation. Six known genes and a locus on human chromosome 4q24 have been implicated in the etiology of isolated OCA forms (OCA 1-7). METHODS: The most frequent OCA types among Caucasians are OCA1, OCA2, and OCA4. We aimed to investigate genes responsible for the development of these OCA forms in Hungarian OCA patients (n = 13). Mutation screening and polymorphism analysis were performed by direct sequencing on TYR, OCA2, SLC45A2 genes. RESULTS: Although the clinical features of the investigated Hungarian OCA patients were identical, the molecular genetic data suggested OCA1 subtype in eight cases and OCA4 subtype in two cases. The molecular diagnosis was not clearly identifiable in three cases. In four patients, two different heterozygous known pathogenic or predicted to be pathogenic mutations were present. Seven patients had only one pathogenic mutation, which was associated with non-pathogenic variants in six cases. In two patients no pathogenic mutation was identified. CONCLUSIONS: Our results suggest that the concomitant screening of the non-pathogenic variants-which alone do not cause the development of OCA, but might have clinical significance in association with a pathogenic variant-is important. Our results also show significant variation in the disease spectrum compared to other populations. These data also confirm that the concomitant analysis of OCA genes is critical, providing new insights to the phenotypic diversity of OCA and expanding the mutation spectrum of OCA genes in Hungarian patients.},
	year = {2017},
	eissn = {2047-783X},
	orcid-numbers = {Tripolszki, Kornélia/0000-0002-7734-1992; Csoma, Zsanett/0000-0003-0376-9061; Kemény, Lajos/0000-0002-2119-9501; Széll, Márta/0000-0002-0730-714X; Nagy, Nikoletta/0000-0001-8576-7953}
}

@article{MTMT:3159158,
	title = {Skin diseases and sexually transmitted infection in a Hungarian prison},
	url = {https://m2.mtmt.hu/api/publication/3159158},
	author = {Vanya, Melinda and Szili, Károly and Krisztina, Magori and Vas, Krisztina},
	doi = {10.1097/MRM.0000000000000087},
	journal-iso = {REV MED MICROBIOL},
	journal = {REVIEWS IN MEDICAL MICROBIOLOGY},
	volume = {28},
	unique-id = {3159158},
	issn = {0954-139X},
	year = {2017},
	pages = {95-96},
	orcid-numbers = {Szili, Károly/0000-0001-9803-9103}
}

@article{MTMT:3178937,
	title = {A pikkelysömörös nem léziós bőr elváltozásai},
	url = {https://m2.mtmt.hu/api/publication/3178937},
	author = {Konczné Gubán, Barbara and Vas, Krisztina and Balog, Zs and Manczinger, Máté and Groma, Gergely and Bebes, Attila and Széll, Márta and Kemény, Lajos and Csörgő Sándorné Bata, Zsuzsanna},
	journal-iso = {BVSZ},
	journal = {BŐRGYÓGYÁSZATI ÉS VENEROLÓGIAI SZEMLE},
	volume = {92},
	unique-id = {3178937},
	issn = {0006-7768},
	year = {2016},
	eissn = {2064-261X},
	pages = {278-278},
	orcid-numbers = {Bebes, Attila/0000-0002-8316-5622; Széll, Márta/0000-0002-0730-714X; Kemény, Lajos/0000-0002-2119-9501; Csörgő Sándorné Bata, Zsuzsanna/0000-0002-3732-1743}
}

@misc{MTMT:3165634,
	title = {Fibroblasts contribute to psoriasis pathology due to abnormal regulation of fibronectin production},
	url = {https://m2.mtmt.hu/api/publication/3165634},
	author = {Konczné Gubán, Barbara and Vas, Krisztina and Balog, Zs and Manczinger, Máté and Groma, Gergely and Bebes, Attila and Széll, Márta and Kemény, Lajos and Csörgő Sándorné Bata, Zsuzsanna},
	unique-id = {3165634},
	year = {2016},
	orcid-numbers = {Bebes, Attila/0000-0002-8316-5622; Széll, Márta/0000-0002-0730-714X; Kemény, Lajos/0000-0002-2119-9501; Csörgő Sándorné Bata, Zsuzsanna/0000-0002-3732-1743}
}

@article{MTMT:3150014,
	title = {Az oculocutan albinizmus összetett genetikai hátterének bemutatása},
	url = {https://m2.mtmt.hu/api/publication/3150014},
	author = {Nagy, Nikoletta and Fábos, Beáta and Tóth, L and Farkas, Katalin and Németh, Réka and Vas, Krisztina and Csoma, Zsanett and Kemény, Lajos and Széll, Márta},
	journal-iso = {BVSZ},
	journal = {BŐRGYÓGYÁSZATI ÉS VENEROLÓGIAI SZEMLE},
	volume = {92},
	unique-id = {3150014},
	issn = {0006-7768},
	year = {2016},
	eissn = {2064-261X},
	pages = {277-278},
	orcid-numbers = {Nagy, Nikoletta/0000-0001-8576-7953; Csoma, Zsanett/0000-0003-0376-9061; Kemény, Lajos/0000-0002-2119-9501; Széll, Márta/0000-0002-0730-714X}
}

@article{MTMT:3043239,
	title = {Keratinocytes express functional CARD18, a negative regulator of inflammasome activation, and its altered expression in psoriasis may contribute to disease pathogenesis},
	url = {https://m2.mtmt.hu/api/publication/3043239},
	author = {Göblös, Anikó and Danis, Judit and Vas, Krisztina and Csörgő Sándorné Bata, Zsuzsanna and Kemény, Lajos and Széll, Márta},
	doi = {10.1016/j.molimm.2016.03.009},
	journal-iso = {MOL IMMUNOL},
	journal = {MOLECULAR IMMUNOLOGY},
	volume = {73},
	unique-id = {3043239},
	issn = {0161-5890},
	abstract = {Abstract Caspase recruitment domain family member 18 (CARD18, Iceberg) is known as a negative regulatory molecule that inhibits inflammatory events by terminating inflammasome activation due to a direct interaction with pro-caspase-1. During the investigation of molecular mechanisms in keratinocytes that contribute to the pathogenesis of psoriasis, we found that CARD18 expression differs in healthy and psoriatic skin; moreover, CARD18 demonstrated altered response under inflammatory conditions in healthy and psoriatic skin. In healthy skin, low basal CARD18 expression was detected, which showed significant elevation in response to inflammatory stimuli (lymphokine treatment or mechanical injury). In contrast, higher basal expression was observed in psoriatic non-involved skin, but no further induction could be detected. We demonstrated that keratinocytes express CARD18 both at mRNA and protein levels and the expression increased in parallel with differentiation. The investigation of cellular inflammatory processes revealed that psoriasis-associated danger signals triggered the expression of inflammasome components (AIM2, Caspase-1) and CARD18 as well as IL-1β production of keratinocytes. Furthermore, gene-specific silencing of CARD18 in cells treated with cytosolic DNA (poly(dA:dT)) resulted in increased IL-1β secretion, suggesting a negative regulatory role for CARD18 in keratinocyte inflammatory signaling. The differential regulation of CARD18 in healthy and psoriatic uninvolved epidermis may contribute to the susceptibility of psoriasis. Furthermore, our in vitro results indicate that CARD18 may contribute to the fine tuning of keratinocyte innate immune processes.},
	keywords = {Inflammation; IL-1β; psoriasis; CASPASE-1; CARD18; AIM2},
	year = {2016},
	eissn = {1872-9142},
	pages = {10-18},
	orcid-numbers = {Göblös, Anikó/0000-0001-7229-8940; Danis, Judit/0000-0002-0270-5309; Csörgő Sándorné Bata, Zsuzsanna/0000-0002-3732-1743; Kemény, Lajos/0000-0002-2119-9501; Széll, Márta/0000-0002-0730-714X}
}

@article{MTMT:2958783,
	title = {Abnormal regulation of fibronectin production by fibroblasts in psoriasis},
	url = {https://m2.mtmt.hu/api/publication/2958783},
	author = {Konczné Gubán, Barbara and Vas, Krisztina and Balog, Z and Manczinger, Máté and Bebes, Attila and Groma, Gergely and Széll, Márta and Kemény, Lajos and Csörgő Sándorné Bata, Zsuzsanna},
	doi = {10.1111/bjd.14219},
	journal-iso = {BRIT J DERMATOL},
	journal = {BRITISH JOURNAL OF DERMATOLOGY},
	volume = {174},
	unique-id = {2958783},
	issn = {0007-0963},
	abstract = {BACKGROUND: Data indicate that in psoriasis abnormalities are already present in the non-lesional skin. TGFbeta, KGF together with fibronectin and alpha5beta1 integrin was suggested to play a crucial role in the pathogenesis of psoriasis by influencing inflammation and keratinocyte hyperproliferation. OBJECTIVES: To investigate the expression of KGF, FGFR2, fibronectin (FN) and EDA+ FN in healthy and non-lesional psoriatic skin and to study the effect of KGF on the regulation of FN and EDA+ FN production by fibroblasts. METHODS: Healthy, non-lesional psoriatic skin and lesional psoriatic skin were immmunostained for alpha5 integrin, KGF, FGFR2, EDA+ FN and STAT1. KGF treated cell cultures were analyzed for FN and EDA+ FN mRNA and protein by real-time RT-PCR and flow cytometry respectively. The four major downstream signaling of KGF was investigated by blocking experiments using MEK1, AKT1/2, STAT1 and STAT3 inhibitors. RESULTS: The expression of alpha5 integrin, EDA+ FN, KGF and its receptor FGFR2 is elevated in psoriatic non-lesional skin compared to healthy skin. KGF mildly induced EDA+ FN, but not FN expression in healthy fibroblasts through MAPK signaling. Fibroblasts express the FGFR2-IIIc. STAT1 negatively regulates both FN and EDA+ FN expressions in healthy fibroblasts and this regulation is compromised in fibroblasts derived from non-lesional psoriatic dermis. We detected active STAT1 in healthy and lesional skin, similar to a previous report, however, in the non-lesional skin STAT1 activation was absent in tissues far away from lesions. CONCLUSIONS: The production of FN, EDA+ FN by fibroblasts and the signaling of STAT1 is abnormally regulated in the psoriatic non-lesional skin. This article is protected by copyright. All rights reserved.},
	year = {2016},
	eissn = {1365-2133},
	pages = {533-541},
	orcid-numbers = {Manczinger, Máté/0000-0003-0831-9617; Bebes, Attila/0000-0002-8316-5622; Groma, Gergely/0000-0001-8487-0465; Széll, Márta/0000-0002-0730-714X; Kemény, Lajos/0000-0002-2119-9501; Csörgő Sándorné Bata, Zsuzsanna/0000-0002-3732-1743}
}