@mastersthesis{MTMT:30773719,
	title = {The isolation of new tissue polarity genes in Drosophila and the genetic analysis of Rab23 involved in the regulation of the number and planar organization of the adult cuticular hairs},
	url = {https://m2.mtmt.hu/api/publication/30773719},
	author = {Pataki, Andrea Csilla},
	publisher = {SZTE},
	unique-id = {30773719},
	year = {2010}
}

@article{MTMT:1921070,
	title = {Drosophila Rab23 Is Involved in the Regulation of the Number and Planar Polarization of the Adult Cuticular Hairs},
	url = {https://m2.mtmt.hu/api/publication/1921070},
	author = {Pataki, Andrea Csilla and Matusek, Tamás and Kurucz, Judit Éva and Andó, István and Jenny, A and Mihály, József},
	doi = {10.1534/genetics.109.112060},
	journal-iso = {GENETICS},
	journal = {GENETICS},
	volume = {184},
	unique-id = {1921070},
	issn = {0016-6731},
	year = {2010},
	eissn = {1943-2631},
	pages = {1051-1065},
	orcid-numbers = {Andó, István/0000-0002-4648-9396}
}

@article{MTMT:1916859,
	title = {Combinatorial signaling by the Frizzled/PCP and Egfr pathways during planar cell polarity establishment in the Drosophila eye},
	url = {https://m2.mtmt.hu/api/publication/1916859},
	author = {Weber, U and Pataki, Andrea Csilla and Mihály, József and Mlodzik, M},
	doi = {10.1016/j.ydbio.2008.01.016},
	journal-iso = {DEV BIOL},
	journal = {DEVELOPMENTAL BIOLOGY},
	volume = {316},
	unique-id = {1916859},
	issn = {0012-1606},
	abstract = {Frizzled (Fz)/PCP signaling regulates planar, vectorial orientation of cells or groups of cells within whole tissues. Although Fz/PCP signaling has been analyzed in several contexts, little is known about nuclear events acting downstream of Fz/PCP signaling in the R3/R4 cell fate decision in the Drosophila eye or in other contexts. Here we demonstrate a specific requirement for Egfir-signaling and the transcription factors Fos (AP-1), Yan and Put in PCP dependent R3/R4 specification. Loss and gain-of-function assays suggest that the transcription factors integrate input from Fz/ PCP and Egfr-signaling and that the ETS factors Pnt and Yan cooperate with Fos (and Jun) in the PCP-specific R3/R4 determination. Our data indicate that Fos (either downstream of Fz/PCP signaling or parallel to it) and Yan are required in R3 to specify its fate (Fos) or inhibit R4 fate (Yan) and that Egfr-signaling is required in R4 via Pnt for its fate specification. Taken together with previous work establishing a Notch-dependent Su(H) function in R4. we conclude that Fos, Yan, Put, and Su(H) integrate Egfr, Fz, and Notch signaling input in R3 or R4 to establish cell fate and ommatidial polarity. (C) 2008 Elsevier Inc. All rights reserved.},
	year = {2008},
	eissn = {1095-564X},
	pages = {110-123}
}

@article{MTMT:1343161,
	title = {Formin Proteins of the DAAM Subfamily Play a Role during Axon Growth},
	url = {https://m2.mtmt.hu/api/publication/1343161},
	author = {Matusek, Tamás and Gombos, Rita I and Szécsényi, Anita and Sanchez-Soriano, N and Czibula, Ágnes and Pataki, Andrea Csilla and Gedai, Anita and Prokop, A and Raskó, István and Mihály, József},
	doi = {10.1523/JNEUROSCI.2727-08.2008},
	journal-iso = {J NEUROSCI},
	journal = {JOURNAL OF NEUROSCIENCE},
	volume = {28},
	unique-id = {1343161},
	issn = {0270-6474},
	abstract = {The regulation of growth cone actin dynamics is a critical aspect of axonal growth control. Among the proteins that are directly involved in the regulation of actin dynamics, actin nucleation factors play a pivotal role by promoting the formation of novel actin filaments. However, the essential nucleation factors in developing neurons have so far not been clearly identified. Here, we show expression data, and use true loss-of-function analysis and targeted expression of activated constructs to demonstrate that the Drosophila formin DAAM plays a critical role in axonal morphogenesis. In agreement with this finding, we show that dDAAM is required for filopodia formation at axonal growth cones. Our genetic interaction, immunoprecipitation and protein localization studies argue that dDAAM acts in concert with Rac GTPases, Profilin and Enabled during axonal growth regulation. We also show that mouse Daam1 rescues the CNS defects observed in dDAAM mutant flies to a high degree, and vice versa, that Drosophila DAAM induces the formation of neurite-like protrusions when expressed in mouse P19 cells, strongly suggesting that the function of DAAM in developing neurons has been conserved during evolution.},
	year = {2008},
	eissn = {1529-2401},
	pages = {13310-13319},
	orcid-numbers = {Czibula, Ágnes/0000-0003-4461-2773}
}

@article{MTMT:1786592,
	title = {Expression pattern of Filamin-240 in Drosophila blood cells},
	url = {https://m2.mtmt.hu/api/publication/1786592},
	author = {Rus, Florentina and Kurucz, Judit Éva and Márkus, Róbert and Sinenko, SA and Laurinyecz, Barbara and Pataki, Andrea Csilla and Gausz, János and Hegedűs, Zoltán and Udvardy, Andor and Hultmark, D and Andó, István},
	doi = {10.1016/j.modgep.2006.03.005},
	journal-iso = {GENE EXPR PATTERNS},
	journal = {GENE EXPRESSION PATTERNS},
	volume = {6},
	unique-id = {1786592},
	issn = {1567-133X},
	keywords = {Animals; Female; TISSUE DISTRIBUTION; PHENOTYPE; PROTEIN ISOFORMS; ARTICLE; DROSOPHILA; HEMOCYTES; Cell Differentiation; priority journal; controlled study; nonhuman; animal tissue; animal cell; larva; Protein Binding; immune system; Western blotting; Animals, Genetically Modified; Gene Expression Profiling; PROTEIN FUNCTION; unclassified drug; protein expression; DNA, Complementary; Blood cells; encapsulation; in vivo study; blood cell; protein domain; TRANSGENE; immunofluorescence test; HOMOZYGOSITY; FILAMIN; protein motif; MUTANT; Contractile proteins; Microfilament Proteins; Vespoidea; Leptopilina boulardi; Wasps; larval development; insect development; filamin 240; Lamellocyte; Immune induction; Hemocyte; Blood cell development; Actin network},
	year = {2006},
	eissn = {1872-7298},
	pages = {928-934},
	orcid-numbers = {Laurinyecz, Barbara/0000-0003-0620-2239; Andó, István/0000-0002-4648-9396}
}

@article{MTMT:1913929,
	title = {Diego and friends play again - Old planar cell polarity players in new positions},
	url = {https://m2.mtmt.hu/api/publication/1913929},
	author = {Mihály, József and Matusek, Tamás and Pataki, Andrea Csilla},
	doi = {10.1111/j.1742-4658.2005.04758.x},
	journal-iso = {FEBS J},
	journal = {FEBS JOURNAL},
	volume = {272},
	unique-id = {1913929},
	issn = {1742-464X},
	year = {2005},
	eissn = {1742-4658},
	pages = {3241-3252}
}