@article{MTMT:34744814,
	title = {A multicentre evaluation of the NG-test DetecTool OXA-23 for the rapid detection of OXA-23 carbapenemase directly from blood cultures},
	url = {https://m2.mtmt.hu/api/publication/34744814},
	author = {Vasilakopoulou, A. and Naas, T. and Gonzalez, C. and Vila, J. and Szabó, Dóra and Riccobono, E. and Kamotsay, K. and Reissier, S. and Berbel, D. and Aszalós, Zoltán and Rosenmoller, M. and Stankov-Puges, M. and Georgiou, P.-C. and Vourli, S. and Volland, H. and Pournaras, S.},
	doi = {10.1093/jacamr/dlae029},
	journal-iso = {JAC-ANTIMICROB RESIST},
	journal = {JAC-ANTIMICROBIAL RESISTANCE},
	volume = {6},
	unique-id = {34744814},
	year = {2024},
	eissn = {2632-1823},
	orcid-numbers = {Szabó, Dóra/0000-0002-8601-3923; Aszalós, Zoltán/0000-0003-3074-5161}
}

@article{MTMT:34666768,
	title = {Multicenter study to assess the use of BL-DetecTool for the detection of CTX-M-type ESBLs and carbapenemases directly from clinical specimens},
	url = {https://m2.mtmt.hu/api/publication/34666768},
	author = {Fernandez-Pittol, Mariana and Bosch, Jordi and Balleste-Delpierre, Clara and Gonzalez, Camille and Vasilakopoulou, Alexandra and Berbel, Damaris and Riccobono, Eleonora and Gatermann, Soeren and Kamotsay, Katalin and Reissier, Sophie and Szabó, Dóra and Aszalós, Zoltán and Francius, Laura and Volland, Herve and Stankov-Puges, Milovan and Rosenmoller, Magda and Naas, Thierry and Vila, Jordi},
	doi = {10.1128/jcm.01136-23},
	journal-iso = {J CLIN MICROBIOL},
	journal = {JOURNAL OF CLINICAL MICROBIOLOGY},
	unique-id = {34666768},
	issn = {0095-1137},
	keywords = {Antibiotic resistance; Rapid test; ESBL; carbapenemases; lateal flow immunoassay; direct sample},
	year = {2024},
	eissn = {1098-660X},
	orcid-numbers = {Szabó, Dóra/0000-0002-8601-3923; Aszalós, Zoltán/0000-0003-3074-5161}
}

@article{MTMT:34656287,
	title = {Middle ear microbiome in otitis media with effusion, the role of Alloiococcus otitidis},
	url = {https://m2.mtmt.hu/api/publication/34656287},
	author = {Fekete, Szilvia and Juhász, János and Makra, Nóra and Dunai, Zsuzsanna and Kristóf, Katalin and Ostorházi, Eszter and Tamás, László and Szabó, Dóra and Kecskeméti, Nóra and Polony, Gábor},
	doi = {10.2139/ssrn.4693874},
	journal-iso = {HELIYON},
	journal = {HELIYON},
	volume = {in press},
	unique-id = {34656287},
	year = {2024},
	eissn = {2405-8440},
	orcid-numbers = {Kristóf, Katalin/0000-0002-5189-4636; Ostorházi, Eszter/0000-0002-9459-7316; Tamás, László/0000-0003-3723-9149; Szabó, Dóra/0000-0002-8601-3923; Kecskeméti, Nóra/0000-0003-1886-5021; Polony, Gábor/0000-0001-5236-8139}
}

@article{MTMT:34481244,
	title = {Pseudomonas aeruginosa antimicrobial susceptibility profiles, resistance mechanisms and international clonal lineages. update from ESGARS-ESCMID/ISARPAE Group.},
	url = {https://m2.mtmt.hu/api/publication/34481244},
	author = {Oliver, Antonio and Rojo-Molinero, Estrella and Arca-Suarez, Jorge and Beşli, Yeşim and Bogaerts, Pierre and Cantón, Rafael and Cimen, Cansu and Croughs, Peter D and Denis, Olivier and Giske, Christian G and Graells, Tíscar and Daniel Huang, Te-Din and Iorga, Bogdan I and Karatuna, Onur and Kocsis, Béla and Kronenberg, Andreas and López-Causapé, Carla and Malhotra-Kumar, Surbhi and Martínez, Luis Martínez and Mazzariol, Annarita and Meyer, Sylvain and Naas, Thierry and Notermans, Daan W and Oteo-Iglesias, Jesús and Pedersen, Torunn and Pirš, Mateja and Poeta, Patricia and Poirel, Laurent and Pournaras, Spyros and Sundsfjord, Arnfinn and Szabó, Dóra and Tambić-Andrašević, Arjana and Vatcheva-Dobrevska, Rossitza and Vitkauskienė, Astra and Jeannot, Katy},
	doi = {10.1016/j.cmi.2023.12.026},
	journal-iso = {CLIN MICROBIOL INFEC},
	journal = {CLINICAL MICROBIOLOGY AND INFECTION},
	volume = {In press},
	unique-id = {34481244},
	issn = {1198-743X},
	abstract = {Pseudomonas aeruginosa, a ubiquitous opportunistic pathogen considered one of the paradigms of antimicrobial resistance, is among the main causes of hospital-acquired and chronic infections associated with significant morbidity and mortality. This growing threat results from the extraordinary capacity of P. aeruginosa to develop antimicrobial resistance through chromosomal mutations, the increasing prevalence of transferable resistance determinants (such as the carbapenemases and the extended spectrum β-lactamases), and the global expansion of epidemic lineages. The general objective of this initiative is to provide a comprehensive update of P. aeruginosa resistance mechanisms, especially for the extensively drug-resistant (XDR)/difficult to treat resistance (DTR) international high-risk epidemic lineages, and how the recently approved β-lactams and β-lactam/β-lactamase inhibitor combinations may affect resistance mechanisms and the definition of susceptibility profiles.To address this challenge, the European Study Group for Antimicrobial Resistance Surveillance (ESGARS) from the European Society of Clinical Microbiology and Infectious Diseases (ESCMID) launched the "Improving Surveillance of Antibiotic-Resistant Pseudomonas aeruginosa in Europe" (ISARPAE) initiative in 2022, supported by the Joint programming initiative on antimicrobial resistance (JPIAMR) network call and included a panel of over 40 researchers from 18 European Countries. Thus, an ESGARS-ISARPAE position paper was designed and the final version agreed after four rounds of revision and discussion by all panel members.To provide an update on (i) the emerging resistance mechanisms to classical and novel antipseudomonal agents, with a particular focus on β-lactams, (ii) the susceptibility profiles associated with the most relevant β-lactam resistance mechanisms, (iii) the impact of the novel agents and resistance mechanisms on the definitions of resistance profiles and (iv) the globally expanding XDR/DTR high-risk lineages and their association with transferable resistance mechanisms.The evidence presented herein can be used for coordinated epidemiological surveillance and decision-making at the European and global level.},
	year = {2024},
	eissn = {1469-0691},
	orcid-numbers = {Kocsis, Béla/0000-0002-7067-7055; Szabó, Dóra/0000-0002-8601-3923}
}

@{MTMT:34493971,
	title = {A bélrendszer baktériumflórája},
	url = {https://m2.mtmt.hu/api/publication/34493971},
	author = {Szabó, Dóra},
	booktitle = {Gasztroenterológia},
	unique-id = {34493971},
	year = {2023},
	pages = {120-130},
	orcid-numbers = {Szabó, Dóra/0000-0002-8601-3923}
}

@article{MTMT:34271363,
	title = {Analysis of Genetic and MRI Changes, Blood Markers, and Risk Factors in a Twin Pair Discordant of Progressive Supranuclear Palsy},
	url = {https://m2.mtmt.hu/api/publication/34271363},
	author = {Persely, Alíz and Beszedics, B. and Pálóczi, Krisztina and Piroska, Márton and Alijanpourotaghsara, Amirreza and Strelnikov, David and Vessal, A. and Szabó, Helga and Hernyes, Anita and Zöldi, Luca and Jokkel, Zsófia and Fekete, A. and Juhász, János and Makra, Nóra and Szabó, Dóra and Buzás, Edit Irén and Tárnoki, Ádám Domonkos and Tárnoki, Dávid László},
	doi = {10.3390/medicina59101696},
	journal-iso = {MED LITH},
	journal = {MEDICINA-LITHUANIA},
	volume = {59},
	unique-id = {34271363},
	issn = {1010-660X},
	year = {2023},
	eissn = {1648-9144},
	orcid-numbers = {Pálóczi, Krisztina/0000-0001-7065-3582; Piroska, Márton/0000-0002-6000-5044; Strelnikov, David/0000-0002-5911-9335; Hernyes, Anita/0000-0001-5875-4006; Zöldi, Luca/0000-0003-4187-1673; Jokkel, Zsófia/0000-0002-5407-6540; Szabó, Dóra/0000-0002-8601-3923; Buzás, Edit Irén/0000-0002-3744-206X; Tárnoki, Ádám Domonkos/0000-0001-5909-3780; Tárnoki, Dávid László/0000-0002-7001-7647}
}

@article{MTMT:34043587,
	title = {Investigation of Delafloxacin Resistance in Multidrug-Resistant Escherichia coli Strains and the Detection of E. coli ST43 International High-Risk Clone},
	url = {https://m2.mtmt.hu/api/publication/34043587},
	author = {Gulyás, Dániel and Kamotsay, Katalin and Szabó, Dóra and Kocsis, Béla},
	doi = {10.3390/microorganisms11061602},
	journal-iso = {MICROORGANISMS},
	journal = {MICROORGANISMS},
	volume = {11},
	unique-id = {34043587},
	issn = {2076-2607},
	abstract = {Delafloxacin is a novel fluoroquinolone agent that is approved for clinical application. In this study, we analyzed the antibacterial efficacy of delafloxacin in a collection of 47 Escherichia coli strains. Antimicrobial susceptibility testing was performed by the broth microdilution method and minimum inhibitory concentration (MIC) values were determined for delafloxacin, ciprofloxacin, levofloxacin, moxifloxacin, ceftazidime, cefotaxime, and imipenem. Two multidrug-resistant E. coli strains, which exhibited delafloxacin and ciprofloxacin resistance as well as extended-spectrum beta-lactamase (ESBL) phenotype, were selected for whole-genome sequencing (WGS). In our study, delafloxacin and ciprofloxacin resistance rates were 47% (22/47) and 51% (24/47), respectively. In the strain collection, 46 E. coli were associated with ESBL production. The MIC50 value for delafloxacin was 0.125 mg/L, while all other fluoroquinolones had an MIC50 value of 0.25 mg/L in our collection. Delafloxacin susceptibility was detected in 20 ESBL positive and ciprofloxacin resistant E. coli strains; by contrast, E. coli strains that exhibited a ciprofloxacin MIC value above 1 mg/L were delafloxacin-resistant. WGS analysis on the two selected E. coli strains (920/1 and 951/2) demonstrated that delafloxacin resistance is mediated by multiple chromosomal mutations, namely, five mutations in E. coli 920/1 (gyrA S83L, D87N, parC S80I, E84V, and parE I529L) and four mutations in E. coli 951/2 (gyrA S83L, D87N, parC S80I, and E84V). Both strains carried an ESBL gene, blaCTX-M-1 in E. coli 920/1 and blaCTX-M-15 in E. coli 951/2. Based on multilocus sequence typing, both strains belong to the E. coli sequence type 43 (ST43). In this paper, we report a remarkable high rate (47%) of delafloxacin resistance among multidrug-resistant E. coli as well as the E. coli ST43 international high-risk clone in Hungary.},
	year = {2023},
	eissn = {2076-2607},
	orcid-numbers = {Gulyás, Dániel/0000-0002-6008-2269; Szabó, Dóra/0000-0002-8601-3923; Kocsis, Béla/0000-0002-7067-7055}
}

@{MTMT:34027246,
	title = {A mikrobióta sebészeti jelentősége},
	url = {https://m2.mtmt.hu/api/publication/34027246},
	author = {Szabó, Dóra and Dobó, Márta and Sándor, József},
	booktitle = {Sebészet tankönyv és tudománytörténet: traumatológiai, szülészeti és urológiai alapismeretekkel kiegészítve},
	unique-id = {34027246},
	year = {2023},
	pages = {62-67},
	orcid-numbers = {Szabó, Dóra/0000-0002-8601-3923}
}

@article{MTMT:34026885,
	title = {Specific nasopharyngeal Corynebacterium strains serve as gatekeepers against SARS-CoV-2 infection},
	url = {https://m2.mtmt.hu/api/publication/34026885},
	author = {Szabó, Dóra and Ostorházi, Eszter and Stercz, Balázs and Makra, Nóra and Pénzes, Kinga and Kristóf, Katalin and Antal, István and Réthelyi, János and Zsigmond, Réka Ildikó and Birtalan, Ede and Merkely, Béla Péter and Tamás, László},
	doi = {10.1007/s11357-023-00850-1},
	journal-iso = {GEROSCIENCE},
	journal = {GEROSCIENCE: OFFICIAL JOURNAL OF THE AMERICAN AGING ASSOCIATION (AGE)},
	volume = {45},
	unique-id = {34026885},
	issn = {2509-2715},
	abstract = {The SARS-CoV-2 virus is still causing a worldwide problem. The virus settles primarily on the nasal mucosa, and the infection and its course depend on individual susceptibility. Our aim was to investigate the nasopharynx composition’s role in the individual susceptibility. During the first phase of SARS-CoV-2 pandemic, nasopharyngeal microbiome samples of close contact unvaccinated patients were investigated by 16S rRNA analysis and by culturing. The whole genome of cultured Corynebacteria was sequenced. The relative expression of ACE2, TMPRSS2, and cathepsin L on Caco-2 cells and the strength of S1-ACE2 binding were determined in the presence of Corynebacteria . From 55 close contacts exposed to identical SARS-CoV-2 exposure, 26 patients became infected and 29 remained uninfected. The nasopharyngeal microbiome analysis showed significantly higher abundance of Corynebacteria in uninfected group. Corynebacterium accolens could be cultivated only from uninfected individuals and Corynebacterium propinquum from both infected and uninfected. Corynebacteria from uninfected patient significantly reduced the ACE2 and cathepsin L expression. C. accolens significantly reduced the TMPRSS2 expression compared to other Corynebacteria . Furthermore, Corynebacterium spp. weakened the binding of the S1-ACE2. Most C. accolens isolates harbored the TAG lipase LipS1 gene. Based on these results, the presence of Corynebacterium spp. in the nasopharyngeal microbiota, especially C. accolens strains, could reduce the individual susceptibility to SARS-CoV-2 infection by several mechanisms: by downregulation the ACE2, the TMPRSS2 receptors, and cathepsin L in the host; through the inhibition of S1-ACE2 binding; and lipase production. These results suggest the use of C. accolens strains as probiotics in the nasopharynx in the future.},
	year = {2023},
	eissn = {2509-2723},
	pages = {2927-2938},
	orcid-numbers = {Szabó, Dóra/0000-0002-8601-3923; Ostorházi, Eszter/0000-0002-9459-7316; Stercz, Balázs/0000-0002-9585-8397; Kristóf, Katalin/0000-0002-5189-4636; Antal, István/0000-0002-5434-201X; Réthelyi, János/0000-0002-3641-012X; Zsigmond, Réka Ildikó/0000-0002-7019-6761; Birtalan, Ede/0000-0002-5699-3545; Merkely, Béla Péter/0000-0001-6514-0723; Tamás, László/0000-0003-3723-9149}
}

@book{MTMT:34494073,
	title = {Az orvosi mikrobiológia alapja – e-tankönyv},
	url = {https://m2.mtmt.hu/api/publication/34494073},
	isbn = {9789633315705},
	author = {Dobay, Orsolya and Ghidán, Ágoston and Horváth, Andrea and Kádár, Béla and Kis, Zoltán and Kocsis, Béla and Nagy, Orsolya and Ostorházi, Eszter and Stercz, Balázs and Szabó, Dóra and Takács, Mária},
	editor = {Szabó, Dóra},
	publisher = {Semmelweis Kiadó és Multimédia Stúdió},
	unique-id = {34494073},
	year = {2022},
	orcid-numbers = {Szabó, Dóra/0000-0002-8601-3923; Dobay, Orsolya/0000-0001-7094-2288; Ghidán, Ágoston/0000-0002-2397-1809; Horváth, Andrea/0000-0003-4208-3417; Kádár, Béla/0000-0002-7126-5415; Kocsis, Béla/0000-0002-7067-7055; Ostorházi, Eszter/0000-0002-9459-7316; Stercz, Balázs/0000-0002-9585-8397; Szabó, Dóra/0000-0002-8601-3923}
}