@{MTMT:34441267,
	title = {The brain-mind-computer trichotomy: Hermeneutic approach},
	url = {https://m2.mtmt.hu/api/publication/34441267},
	author = {Érdi, Péter},
	booktitle = {Artificial Intelligence in the Age of Neural Networks and Brain Computing, Second Edition},
	doi = {10.1016/B978-0-323-96104-2.00019-1},
	unique-id = {34441267},
	year = {2023},
	pages = {77-89}
}

@book{MTMT:34020694,
	title = {Repair. When and How to Improve Broken Objects, Ourselves, and Our Society},
	url = {https://m2.mtmt.hu/api/publication/34020694},
	isbn = {9783030989071},
	author = {Érdi, Péter and Szvetelszky, Zsuzsanna},
	doi = {10.1007/978-3-030-98908-8},
	publisher = {Springer International Publishing},
	unique-id = {34020694},
	year = {2022}
}

@{MTMT:32543791,
	title = {The Cognitive Science of the Ranking Game},
	url = {https://m2.mtmt.hu/api/publication/32543791},
	author = {Érdi, Péter},
	booktitle = {A Life in Cognition},
	doi = {10.1007/978-3-030-66175-5_19},
	volume = {11},
	unique-id = {32543791},
	year = {2022},
	pages = {267-276}
}

@article{MTMT:30916265,
	title = {The cognitive science of the ranking game},
	url = {https://m2.mtmt.hu/api/publication/30916265},
	author = {Érdi, Péter},
	journal-iso = {CEUR WORKSHOP PROC},
	journal = {CEUR WORKSHOP PROCEEDINGS},
	volume = {2483},
	unique-id = {30916265},
	year = {2019},
	eissn = {1613-0073},
	pages = {95-103}
}

@{MTMT:30732584,
	title = {The Brain-Mind-Computer Trichotomy: Hermeneutic Approach},
	url = {https://m2.mtmt.hu/api/publication/30732584},
	author = {Érdi, Péter},
	booktitle = {ARTIFICIAL INTELLIGENCE IN THE AGE OF NEURAL NETWORKS AND BRAIN COMPUTING},
	doi = {10.1016/B978-0-12-815480-9.00004-9},
	unique-id = {30732584},
	year = {2019},
	pages = {79-92}
}

@inproceedings{MTMT:3355584,
	title = {From ranking and clustering of evolving networks to patent citation analysis},
	url = {https://m2.mtmt.hu/api/publication/3355584},
	author = {Beltz, H and Fulop, A and Wadhwa, RR and Érdi, Péter},
	booktitle = {International Joint Conference on Neural Networks, IJCNN 2017},
	doi = {10.1109/IJCNN.2017.7966015},
	unique-id = {3355584},
	year = {2017},
	pages = {1388-1394}
}

@inproceedings{MTMT:3270478,
	title = {The brain-mind computer trichotomy: Hermeneutic approach},
	url = {https://m2.mtmt.hu/api/publication/3270478},
	author = {Érdi, Péter},
	booktitle = {4th International Workshop on Artificial Intelligence and Cognition, AIC 2016},
	unique-id = {3270478},
	year = {2017},
	pages = {106-116}
}

@{MTMT:3248732,
	title = {Connecting Epilepsy and Alzheimer’s Disease: Modeling of Normal and Pathological Rhythmicity and Synaptic Plasticity Related to Amyloidββ (Aββ) Effects},
	url = {https://m2.mtmt.hu/api/publication/3248732},
	author = {Érdi, Péter and Matsuzawa, T and John, T and Kiss, Tamás and Zalányi, László András},
	booktitle = {Computational Neurology and Psychiatry},
	doi = {10.1007/978-3-319-49959-8_5},
	unique-id = {3248732},
	abstract = {This chapter is motivated by the hidden links between neurodegeneration due to Alzheimer’s disease and temporal lobe epileptic activity. The following argument is based on the multiple effects of $$\beta $$ -amyloid peptides (A $$\beta $$ ) forming these links potentially at molecular, cellular, synaptic, network, as well as system levels. To explore these links a computational framework was discussed, and two parts of the framework, i.e. pathological rhythm generation and altered bidirectional synaptic plasticity have been constructed and analyzed. By using a skeleton network model of the hippocampal rhythm generation it was demonstrated how A $$\beta $$ affects the ability of neurons in hippocampal networks to fire in unison at theta frequency resulting in reduced power of the theta rhythm. The dual qualitative effects of elevated A $$\beta $$ at the synaptic level, i.e. LTD facilitation and LTP impairment is studied by a modified calcium control hypothesis. The modification implemented the $$\beta $$ -amyloid effects on the bidirectional synaptic plasticity and explained well the experimental findings of decreased LTP and increased LTD. The analysis of a kinetic model taking into account the phosphorylation and dephosphorylation pathways associated with potentiation and depression of the AMPA receptor activity supported the biological plausibility of the modification. Such kinds of models offer an integrative perspective to organize scattered data obtained by methods of anatomy, electrophysiology, brain imaging, neurochemistry, behavioral studies, etc. into a coherent picture.},
	year = {2017},
	pages = {93-120}
}

@book{MTMT:3248731,
	title = {Computational Neurology and Psychiatry},
	url = {https://m2.mtmt.hu/api/publication/3248731},
	isbn = {9783319499598},
	editor = {Érdi, Péter and Sen, Bhattacharya B and Cochran, AL},
	publisher = {Springer Netherlands},
	unique-id = {3248731},
	year = {2017}
}

@article{MTMT:3243817,
	title = {Multi-scale modeling of altered synaptic plasticity related to Amyloid effects},
	url = {https://m2.mtmt.hu/api/publication/3243817},
	author = {Matsuzawa, T and Zalányi, László András and Kiss, Tamás and Érdi, Péter},
	doi = {10.1016/j.neunet.2017.06.002},
	journal-iso = {NEURAL NETWORKS},
	journal = {NEURAL NETWORKS},
	volume = {93},
	unique-id = {3243817},
	issn = {0893-6080},
	year = {2017},
	eissn = {1879-2782},
	pages = {230-239}
}