@article{MTMT:34772212,
	title = {Subacute onset of dysphagia and diplopia},
	url = {https://m2.mtmt.hu/api/publication/34772212},
	author = {Oláh, László and Csiba, László},
	journal-iso = {EUR J NEUROL},
	journal = {EUROPEAN JOURNAL OF NEUROLOGY},
	volume = {30},
	unique-id = {34772212},
	issn = {1351-5101},
	year = {2023},
	eissn = {1468-1331},
	pages = {38-38}
}

@article{MTMT:34693306,
	title = {Hemorrhagic transformation and non-CNS complications after iv. thrombolysis: a brain and body autopsy study},
	url = {https://m2.mtmt.hu/api/publication/34693306},
	author = {Hudák, Lilla and Nagy, A. and Perjesi-Kiss, B. and Fulesdi, Z. and Busi, B. and Nagy, K. and Méhes, Gábor and Oláh, László and Csiba, László},
	journal-iso = {EUR J NEUROL},
	journal = {EUROPEAN JOURNAL OF NEUROLOGY},
	volume = {30},
	unique-id = {34693306},
	issn = {1351-5101},
	year = {2023},
	eissn = {1468-1331},
	pages = {455-455}
}

@article{MTMT:34493937,
	title = {Role of carotid duplex in the assessment of carotid artery restenosis after endarterectomy or stenting},
	url = {https://m2.mtmt.hu/api/publication/34493937},
	author = {Szegedi, István and Potvorszki, Fanni and Mészáros, Zsófia Réka and Daniel, Cecilia and Csiba, László and Oláh, László},
	doi = {10.3389/fneur.2023.1226220},
	journal-iso = {FRONT NEUR},
	journal = {FRONTIERS IN NEUROLOGY},
	volume = {14},
	unique-id = {34493937},
	issn = {1664-2295},
	year = {2023},
	eissn = {1664-2295},
	pages = {14},
	orcid-numbers = {Szegedi, István/0000-0003-4181-4361}
}

@article{MTMT:34443842,
	title = {Enalapril Is Superior to Lisinopril in Improving Endothelial Function without a Difference in Blood–Pressure–Lowering Effects in Newly Diagnosed Hypertensives},
	url = {https://m2.mtmt.hu/api/publication/34443842},
	author = {Nagy, Attila Csaba and Májer, Réka and Boczán, Judit and Sipka, Sándor and Szabó, Attila Ádám and Enyedi, Enikő Edit and Tatai, Ottó and Fagyas, Miklós and Papp, Zoltán and Csiba, László and Tóth, Attila},
	doi = {10.3390/biomedicines11123323},
	journal-iso = {BIOMEDICINES},
	journal = {BIOMEDICINES},
	volume = {11},
	unique-id = {34443842},
	abstract = {Angiotensin–converting enzyme (ACE) inhibitors are the primarily chosen drugs to treat various cardiovascular diseases, such as hypertension. Although the most recent guidelines do not differentiate among the various ACE inhibitory drugs, there are substantial pharmacological differences. Goal: Here, we tested if lipophilicity affects the efficacy of ACE inhibitory drugs when used as the first therapy in newly identified hypertensives in a prospective study. Methods: We tested the differences in the cardiovascular efficacy of the hydrophilic lisinopril (8.3 ± 3.0 mg/day) and the lipophilic enalapril (5.5 ± 2.3 mg/day) (n = 59 patients). The cardiovascular parameters were determined using sonography (flow-mediated dilation (FMD) in the brachial artery, intima-media thickness of the carotid artery), 24 h ambulatory blood pressure monitoring (peripheral arterial blood pressure), and arteriography (aortic blood pressure, augmentation index, and pulse wave velocity) before and after the initiation of ACE inhibitor therapy. Results: Both enalapril and lisinopril decreased blood pressure. However, lisinopril failed to improve arterial endothelial function (lack of effects on FMD) when compared to enalapril. Enalapril-mediated improved arterial endothelial function (FMD) positively correlated with its blood–pressure–lowering effect. In contrast, there was no correlation between the decrease in systolic blood pressure and FMD in the case of lisinopril treatment. Conclusion: The blood–pressure–lowering effects of ACE inhibitor drugs are independent of their lipophilicity. In contrast, the effects of ACE inhibition on arterial endothelial function are associated with lipophilicity: the hydrophilic lisinopril was unable to improve, while the lipophilic enalapril significantly improved endothelial function. Moreover, the effects on blood pressure and endothelial function did not correlate in lisinopril-treated patients, suggesting divergent mechanisms in the regulation of blood pressure and endothelial function upon ACE inhibitory treatment.},
	year = {2023},
	eissn = {2227-9059},
	orcid-numbers = {Nagy, Attila Csaba/0000-0002-0554-7350; Papp, Zoltán/0000-0002-4675-1542; Tóth, Attila/0000-0001-6503-3653}
}

@article{MTMT:34238940,
	title = {Hemorrhagic Transformation of Ischemic Strokes},
	url = {https://m2.mtmt.hu/api/publication/34238940},
	author = {Kovács, Kitti Bernadett and Bencs, Viktor and Hudák, Lilla and Oláh, László and Csiba, László},
	doi = {10.3390/ijms241814067},
	journal-iso = {INT J MOL SCI},
	journal = {INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES},
	volume = {24},
	unique-id = {34238940},
	issn = {1661-6596},
	abstract = {Ischemic stroke, resulting from insufficient blood supply to the brain, is among the leading causes of death and disability worldwide. A potentially severe complication of the disease itself or its treatment aiming to restore optimal blood flow is hemorrhagic transformation (HT) increasing morbidity and mortality. Detailed summaries can be found in the literature on the pathophysiological background of hemorrhagic transformation, the potential clinical risk factors increasing its chance, and the different biomarkers expected to help in its prediction and clinical outcome. Clinicopathological studies also contribute to the improvement in our knowledge of hemorrhagic transformation. We summarized the clinical risk factors of the hemorrhagic transformation of ischemic strokes in terms of risk reduction and collected the most promising biomarkers in the field. Also, auxiliary treatment options in reperfusion therapies have been reviewed and collected. We highlighted that the optimal timing of revascularization treatment for carefully selected patients and the individualized management of underlying diseases and comorbidities are pivotal. Another important conclusion is that a more intense clinical follow-up including serial cranial CTs for selected patients can be recommended, as clinicopathological investigations have shown HT to be much more common than clinically suspected.},
	keywords = {PATHOPHYSIOLOGY; Biomarkers; ischemic stroke; HEMORRHAGIC TRANSFORMATION; antithrombotic treatment; Clinical risk factors; clinicopathological studies},
	year = {2023},
	eissn = {1422-0067},
	orcid-numbers = {Kovács, Kitti Bernadett/0000-0003-1781-2390}
}

@article{MTMT:34178370,
	title = {A European Academy of Neurology konferenciája Budapesten},
	url = {https://m2.mtmt.hu/api/publication/34178370},
	author = {Csiba, László},
	journal-iso = {IDEGGYOGY SZEMLE},
	journal = {IDEGGYOGYASZATI SZEMLE / CLINICAL NEUROSCIENCE},
	volume = {76},
	unique-id = {34178370},
	issn = {0019-1442},
	year = {2023},
	eissn = {2498-6208},
	pages = {318}
}

@article{MTMT:34071483,
	title = {In memoriam Molnár László: szoboravatás a Debreceni Egyetemen = In memoriam László Molnár: statue unveiling at the University of Debrecen},
	url = {https://m2.mtmt.hu/api/publication/34071483},
	author = {Csiba, László},
	doi = {10.18071/isz.76.0230},
	journal-iso = {IDEGGYOGY SZEMLE},
	journal = {IDEGGYOGYASZATI SZEMLE / CLINICAL NEUROSCIENCE},
	volume = {76},
	unique-id = {34071483},
	issn = {0019-1442},
	abstract = {Molnár László professzor 1923-ban született. Egyetemi tanulmányait Szegeden végezte, klinikai munkáját Pécsett folytatta. Ideggyógyász, pszichiáter és idegsebész szakképesítést szerzett. Előbb Németországban tanulmányozta az agyi vérkeringés szabályozását állatkísérletekben, majd Párizsban dolgozott ösztöndíjasként, Seylaz professzorral. A Sorbonne-on doktorált. Kandidátusi disszertációját 1966-ban, az MTA doktora címet 1977-ben szerezte meg. 1969 és 1992 között a Debreceni Egyetem Neurológiai Klinikáját vezette, ahol a fokális ischaemia következményeit tanulmányozta állatkísérletekben. Debrecenben megalapította – Európában másodikként – a stroke-betegek ellátására specializálódott Cerebrovascularis Osztályt. Munkatársai közül tizenegyen lettek osztályvezető főorvosok, négyen egyetemi tanárok, hatan szereztek PhD- és hárman MTA doktori címet. 1999-ben halt meg.},
	year = {2023},
	eissn = {2498-6208},
	pages = {230-232}
}

@article{MTMT:33729016,
	title = {Alkalmas-e a carotisultrahang a carotisintervenciót követő neurológiai kockázat előrejelzésére?},
	url = {https://m2.mtmt.hu/api/publication/33729016},
	author = {Yi-chun, Chen and Oláh, László and Csiba, László},
	doi = {10.18071/isz.76.0095},
	journal-iso = {IDEGGYOGY SZEMLE},
	journal = {IDEGGYOGYASZATI SZEMLE / CLINICAL NEUROSCIENCE},
	volume = {76},
	unique-id = {33729016},
	issn = {0019-1442},
	abstract = {Háttér és cél – Mind a carotisstent  (CAS), mind a carotisendarterectomia (CEA) alkalmazható a carotisszűkület által okozott ischaemiás stroke megelőzésére, de mindkét beavatkozás komplikációkkal járhat. Célunk annak megállapítása, melyik carotis­-ultrahang-módszer a legalkalmasabb a beavatkozáshoz kapcsolódó szövődmények előrejelzésére (embolisatio, új neurológiai tünetek).   Módszerek – A PubMed, az EMBASE és a Cochrane adatbázisokban 2000-től 2022-ig megjelent közlemények elemzése.  Eredmények – A plakk grayscale-median(GSM-) értéke a legérzékenyebb módszer a periproceduralis szövődmények jóslására. A viszonylag kis létszámú vizsgálatok szerint a ≤ 20 GSM-értékek már jelentős  periproceduralis szövődményveszélyre utalnak. Megbeszélés – A diffúziósúlyozott MR a legérzékenyebb módszer annak eldöntésére, hogy a CEA vagy a stent okozott-e ischaemiás szövődményt. Következtetés – Nagy létszámú, multicentrikus vizsgálatnak kell eldöntenie, melyik GSM-érték az optimális a periproceduralis komplikációk előrejelzésére.},
	keywords = {grayscale median; ischaemiás stroke; carotisultrahang; arteria carotis stent; carotisendarterectomia},
	year = {2023},
	eissn = {2498-6208},
	pages = {95-101},
	orcid-numbers = {Yi-chun, Chen/0000-0001-7503-1264; Csiba, László/0000-0003-3273-8555}
}

@article{MTMT:33710453,
	title = {Stroke},
	url = {https://m2.mtmt.hu/api/publication/33710453},
	author = {Bereczki, Dániel and Csiba, László},
	journal-iso = {PSYCHIATRIC & NEUROLOGY TIMES (MAGYAR KIADÁS)},
	journal = {PSYCHIATRIC & NEUROLOGY TIMES (MAGYAR KIADÁS)},
	volume = {1},
	unique-id = {33710453},
	issn = {2559-9437},
	year = {2023},
	pages = {43-46},
	orcid-numbers = {Bereczki, Dániel/0000-0002-8374-0500}
}

@article{MTMT:33699975,
	title = {Elevated Blood Alcohol Concentration Is Associated with Improved Clinical Outcomes of Intravenous Thrombolysis Treatment in Acute Ischemic Stroke Patients—A Retrospective Study},
	url = {https://m2.mtmt.hu/api/publication/33699975},
	author = {Árokszállási, Tamás and Balogh, Eszter and Orbán-Kálmándi, Rita Angéla and Pásztor, Máté and Árokszállási, Anita and Nagy, Edit Boglárka and Belán, Ivett and May, Zsolt and Csépány, Tünde and Csiba, László and Bagoly, Zsuzsa and Oláh, László},
	doi = {10.3390/jcm12062238},
	journal-iso = {J CLIN MED},
	journal = {JOURNAL OF CLINICAL MEDICINE},
	volume = {12},
	unique-id = {33699975},
	abstract = {Background: Intravenous thrombolysis (IVT) improves acute ischemic stroke (AIS) outcomes, but with limited success. In addition, ethanol potentiates the effect of r-tPA in ischemia models. Methods: The effect of acute alcohol consumption on IVT outcomes was investigated in a retrospective cohort study. AIS patients with detectable blood alcohol concentration (BAC) during IVT were included (alcohol group; n = 60). For each case, 3 control subjects who underwent IVT but denied alcohol consumption were matched in terms of age, sex, affected brain area, and stroke severity. Outcomes were determined using the NIHSS at 7 days and the modified Rankin scale (mRS) at 90 days. Results: Patients were younger and had a less severe stroke than in a standard stroke study. Favorable long-term outcomes (mRS 0–2) occurred significantly more frequently in the alcohol group compared to controls (90% vs. 63%, p < 0.001). However, the rates of hemorrhagic transformation were similar. Multiple logistic regression models identified elevated BAC as a significant protective factor against unfavorable short-term (OR: 0.091, 95% CI: 0.036–0.227, p < 0.001) and long-term outcomes (OR: 0.182, 95% CI: 0.062–0.535, p = 0.002). In patients with BAC > 0.2%, significantly lower NIHSS was observed at 3 and 7 days after IVT vs. in those with 0.01–0.2% ethanol levels. Conclusion: Elevated BAC is associated with improved outcomes in IVT-treated AIS without affecting safety.},
	year = {2023},
	eissn = {2077-0383},
	orcid-numbers = {Balogh, Eszter/0000-0001-5343-9867; Orbán-Kálmándi, Rita Angéla/0000-0002-2155-8279; Csépány, Tünde/0000-0002-8305-3209; Bagoly, Zsuzsa/0000-0001-5314-5607}
}