@{MTMT:34774903,
	title = {Additive manufacturing in limb prosthetics and orthotics: the past, present and future of 3D printing orthopedic assistive devices},
	url = {https://m2.mtmt.hu/api/publication/34774903},
	author = {Maróti, Péter and Schlégl, Ádám Tibor and Nagy, Bálint and Tóth, Luca and Bogár, Péter Zoltán and Józsa, Gergő and Rendeki, Szilárd and Mallakpour, Shadpour and Hussain, Chaudhery Mustansar},
	booktitle = {Medical Additive Manufacturing},
	doi = {10.1016/B978-0-323-95383-2.00028-7},
	unique-id = {34774903},
	year = {2024},
	pages = {179-207},
	orcid-numbers = {Maróti, Péter/0000-0001-7538-0675}
}

@article{MTMT:34568744,
	title = {Examining the Efficacy of Lower Extremity Exoskeletons in the Rehabilitation Process of Spinal Cord Injury Patients. Case Studies},
	url = {https://m2.mtmt.hu/api/publication/34568744},
	author = {Shenker, Benjámin and Fehér, Melinda and Erdősi, Petra and Papp, Leila and Vadai, Kitti and Hrivnák, Gergely and Tóth, Luca and Maróti, Péter and Horváth, Csaba and Cserháti, Péter},
	doi = {10.12700/APH.20.8.2023.8.7},
	journal-iso = {ACTA POLYTECH HUNG},
	journal = {ACTA POLYTECHNICA HUNGARICA},
	volume = {20},
	unique-id = {34568744},
	issn = {1785-8860},
	year = {2023},
	eissn = {1785-8860},
	pages = {111-131},
	orcid-numbers = {Shenker, Benjámin/0000-0003-4087-1751; Erdősi, Petra/0009-0001-4451-3188; Maróti, Péter/0000-0001-7538-0675; Horváth, Csaba/0000-0002-0490-7932}
}

@article{MTMT:33647818,
	title = {Successful International EMBS Conference on Biomedical Engineering and Innovation Held in Hungary},
	url = {https://m2.mtmt.hu/api/publication/33647818},
	author = {Akay, Y. and Tóth, Luca},
	doi = {10.1109/MPULS.2022.3227856},
	journal-iso = {IEEE PULSE},
	journal = {IEEE PULSE},
	volume = {13},
	unique-id = {33647818},
	issn = {2154-2287},
	year = {2022},
	eissn = {2154-2317},
	pages = {33-36}
}

@article{MTMT:33121205,
	title = {Gerincvelősérültek alsó végtagi exoszkeletonnal végzett rehabilitációja a COVID-járvány alatt},
	url = {https://m2.mtmt.hu/api/publication/33121205},
	author = {Shenker, Benjámin and Tóth, Luca and Maróti, Péter and Fehér, Melinda and Cserháti, Péter},
	journal-iso = {REHABILITÁCIÓ},
	journal = {REHABILITÁCIÓ: A MAGYAR REHABILITÁCIÓS TÁRSASÁG FOLYÓIRATA},
	volume = {32},
	unique-id = {33121205},
	issn = {0866-479X},
	year = {2022},
	pages = {79-79},
	orcid-numbers = {Shenker, Benjámin/0000-0003-4087-1751; Maróti, Péter/0000-0001-7538-0675}
}

@article{MTMT:33032134,
	title = {Age-related decline in circulating IGF-1 associates with impaired neurovascular coupling responses in older adults.},
	url = {https://m2.mtmt.hu/api/publication/33032134},
	author = {Tóth, Luca and Czigler, András and Hegedüs, Emőke and Komáromy, Hedvig and Amrein, Krisztina and Czeiter, Endre and Yabluchanskiy, Andriy and Koller, Ákos and Orsi, Gergely and Perlaki, Gábor and Schwarcz, Attila and Büki, András and Ungvári, Zoltán István and Tóth, Péter József},
	doi = {10.1007/s11357-022-00623-2},
	journal-iso = {GEROSCIENCE},
	journal = {GEROSCIENCE: OFFICIAL JOURNAL OF THE AMERICAN AGING ASSOCIATION (AGE)},
	volume = {44},
	unique-id = {33032134},
	issn = {2509-2715},
	abstract = {Impairment of moment-to-moment adjustment of cerebral blood flow (CBF) to the increased oxygen and energy requirements of active brain regions via neurovascular coupling (NVC) contributes to the genesis of age-related cognitive impairment. Aging is associated with marked deficiency in the vasoprotective hormone insulin-like growth factor-1 (IGF-1). Preclinical studies on animal models of aging suggest that circulating IGF-1 deficiency is causally linked to impairment of NVC responses. The present study was designed to test the hypotheses that decreases in circulating IGF-1 levels in older adults also predict the magnitude of age-related decline of NVC responses. In a single-center cross-sectional study, we enrolled healthy young (n = 31, 11 female, 20 male, mean age: 28.4 + / - 4.2 years) and aged volunteers (n = 32, 18 female, 14 male, mean age: 67.9 + / - 4.1 years). Serum IGF-1 level, basal CBF (phase contrast magnetic resonance imaging (MRI)), and NVC responses during the trail making task (with transcranial Doppler sonography) were assessed. We found that circulating IGF-1 levels were significantly decreased with age and associated with decreased basal CBF. Age-related decline in IGF-1 levels predicted the magnitude of age-related decline in NVC responses. In conclusion, our study provides additional evidence in support of the concept that age-related circulating IGF-1 deficiency contributes to neurovascular aging, impairing CBF and functional hyperemia in older adults.},
	keywords = {Aging; cognitive decline; VCI; VCID; Vascular cognitive impairment; neurovascular uncoupling},
	year = {2022},
	eissn = {2509-2723},
	pages = {2771-2783},
	orcid-numbers = {Czeiter, Endre/0000-0002-9578-6944; Koller, Ákos/0000-0003-3256-8701; Ungvári, Zoltán István/0000-0002-6035-6039}
}

@inproceedings{MTMT:33027020,
	title = {Initial Results of Lower Limb Exoskeleton Therapy with Human Gait Analysis for a Paraplegic Patient},
	url = {https://m2.mtmt.hu/api/publication/33027020},
	author = {Tóth, Luca and Schiffer, Ádám and Pinczker, Veronika and Müller, Péter János and Büki, András and Maróti, Péter},
	booktitle = {Innovations and Developments of Technologies in Medicine, Biology and Healthcare},
	doi = {10.1007/978-3-030-88976-0_20},
	unique-id = {33027020},
	year = {2022},
	pages = {151-157},
	orcid-numbers = {Schiffer, Ádám/0000-0002-0769-2375; Müller, Péter János/0000-0002-2283-1791; Maróti, Péter/0000-0001-7538-0675}
}

@article{MTMT:32320749,
	title = {Cerebral Microbleeds May Be Less Detectable by Susceptibility Weighted Imaging MRI From 24 to 72 Hours After Traumatic Brain Injury},
	url = {https://m2.mtmt.hu/api/publication/32320749},
	author = {Környei, Bálint Soma and Szabó, Viktor and Perlaki, Gábor and Balogh, Bendegúz and Szabó Steigerwald, Dorottya K. and Nagy, Szilvia Anett and Tóth, Luca and Büki, András and Dóczi, Tamás Péter and Bogner, Péter and Schwarcz, Attila and Tóth, Arnold},
	doi = {10.3389/fnins.2021.711074},
	journal-iso = {FRONT NEUROSCI-SWITZ},
	journal = {FRONTIERS IN NEUROSCIENCE},
	volume = {15},
	unique-id = {32320749},
	issn = {1662-4548},
	abstract = {Purpose: A former rodent study showed that cerebral traumatic microbleeds (TMBs) may temporarily become invisible shortly after injury when detected by susceptibility weighted imaging (SWI). The present study aims to validate this phenomenon in human SWI. Methods: In this retrospective study, 46 traumatic brain injury (TBI) patients in various forms of severity were included and willingly complied with our strict selection criteria. Clinical parameters potentially affecting TMB count, Rotterdam and Marshall CT score, Mayo Clinic Classification, contusion number, and total volume were registered. The precise time between trauma and MRI [5 h 19 min to 141 h 54 min, including SWI and fluid-attenuated inversion recovery (FLAIR)] was individually recorded; TMB and FLAIR lesion counts were assessed. Four groups were created based on elapsed time between the trauma and MRI: 0-24, 24-48, 48-72, and >72 h. Kruskal-Wallis, ANOVA, Chi-square, and Fisher's exact tests were used to reveal differences among the groups within clinical and imaging parameters; statistical power was calculated retrospectively for each comparison. Results: The Kruskal-Wallis ANOVA with Conover post hoc analysis showed significant (p = 0.01; 1-β > 0.9) median TMB number differences in the subacute period: 0-24 h = 4.00 (n = 11); 24-48 h = 1 (n = 14); 48-72 h = 1 (n = 11); and 72 h ≤ 7.5 (n = 10). Neither clinical parameters nor FLAIR lesions depicted significant differences among the groups. Conclusion: Our results demonstrate that TMBs on SWI MRI may temporarily become less detectable at 24-72 h following TBI.},
	year = {2021},
	eissn = {1662-453X},
	orcid-numbers = {Nagy, Szilvia Anett/0000-0001-6483-9209}
}

@{MTMT:32256992,
	title = {First impressions with lower extremity exoskeleton during rehabilitation of spinal cord injured patients},
	url = {https://m2.mtmt.hu/api/publication/32256992},
	author = {Shenker, Benjámin and Tóth, Luca and Maróti, Péter and Hrivnák, Gergely and Cserháti, Péter},
	booktitle = {Congress of European Forum for Research in Rehabilitation},
	unique-id = {32256992},
	year = {2021},
	pages = {58-58},
	orcid-numbers = {Shenker, Benjámin/0000-0003-4087-1751; Maróti, Péter/0000-0001-7538-0675}
}

@article{MTMT:32226057,
	title = {Alsó végtagi exoskeleton segítségével történő rehabilitációs tevékenység kezdeti tapasztalatai},
	url = {https://m2.mtmt.hu/api/publication/32226057},
	author = {Shenker, Benjámin and Tóth, Luca and Maróti, Péter and Büki, András and Fehér, Melinda and Klauber, András and Révay, Edit and Erdősi, Petra and Farkasinszky, Diána Edina and Nagy, Nikolett and Varga, Edina and Szabó-Szemenyei, Eszter and Vadai, Kitti and Papp, Leila and Hrivnák, Gergely and Cserháti, Péter},
	journal-iso = {IDŐSGYÓGYÁSZAT},
	journal = {IDŐSGYÓGYÁSZAT},
	volume = {6},
	unique-id = {32226057},
	issn = {2498-8057},
	year = {2021},
	pages = {15-20},
	orcid-numbers = {Shenker, Benjámin/0000-0003-4087-1751; Maróti, Péter/0000-0001-7538-0675; Erdősi, Petra/0009-0001-4451-3188; Szabó-Szemenyei, Eszter/0000-0002-4164-1678}
}

@article{MTMT:32172268,
	title = {The effect of mild traumatic brain injury on cerebral microbleeds in aging},
	url = {https://m2.mtmt.hu/api/publication/32172268},
	author = {Tóth, Luca and Czigler, András and Horváth, Péter and Szarka, Nikolett and Környei, Bálint Soma and Tóth, Arnold and Schwarcz, Attila and Ungvári, Zoltán István and Büki, András and Tóth, Péter József},
	doi = {10.3389/fnagi.2021.717391},
	journal-iso = {FRONT AGING NEUROSCI},
	journal = {FRONTIERS IN AGING NEUROSCIENCE},
	volume = {13},
	unique-id = {32172268},
	issn = {1663-4365},
	abstract = {A traumatic brain injury (TBI) induces the formation of cerebral microbleeds (CMBs), which are associated with cognitive impairments, psychiatric disorders, and gait dysfunctions in patients. Elderly people frequently suffer TBIs, especially mild brain trauma (mTBI). Interestingly, aging is also an independent risk factor for the development of CMBs. However, how TBI and aging may interact to promote the development of CMBs is not well established. In order to test the hypothesis that an mTBI exacerbates the development of CMBs in the elderly, we compared the number and cerebral distribution of CMBs and assessed them by analysing susceptibility weighted (SW) MRI in young (25 ± 10 years old, n = 18) and elder (72 ± 7 years old, n = 17) patients after an mTBI and in age-matched healthy subjects (young: 25 ± 6 years old, n = 20; aged: 68 ± 5 years old, n = 23). We found significantly more CMBs in elder patients after an mTBI compared with young patients; however, we did not observe a significant difference in the number of cerebral microhemorrhages between aged and aged patients with mTBI. The majority of CMBs were found supratentorially (lobar and basal ganglion). The lobar distribution of supratentorial CMBs showed that aging enhances the formation of parietal and occipital CMBs after mTBIs. This suggests that aging and mTBIs do not synergize in the induction of the development of CMBs, and that the different distribution of mTBI-induced CMBs in aged patients may lead to specific age-related clinical characteristics of mTBIs.},
	year = {2021},
	eissn = {1663-4365},
	orcid-numbers = {Ungvári, Zoltán István/0000-0002-6035-6039}
}