@{MTMT:34823733,
	title = {Poszttraumás convulsiók},
	url = {https://m2.mtmt.hu/api/publication/34823733},
	author = {Janszky, József Vladimír},
	booktitle = {Súlyos baleseti agysérültek ellátása},
	unique-id = {34823733},
	year = {2024},
	pages = {181-182},
	orcid-numbers = {Janszky, József Vladimír/0000-0001-6100-832X}
}

@article{MTMT:34800559,
	title = {Prevalence of celebrity worship: Development and application of the short version of the Celebrity Attitude Scale (CAS-7) on a large-scale representative sample},
	url = {https://m2.mtmt.hu/api/publication/34800559},
	author = {Zsila, Ágnes and McCutcheon, Lynn E. and Horváth, Rita and Urbán, Róbert and Paksi, Borbála and Darnai, Gergely and Janszky, József Vladimír and Demetrovics, Zsolt},
	doi = {10.1556/2006.2024.00019},
	journal-iso = {J BEHAV ADDICT},
	journal = {JOURNAL OF BEHAVIORAL ADDICTIONS},
	unique-id = {34800559},
	issn = {2062-5871},
	year = {2024},
	eissn = {2063-5303},
	orcid-numbers = {Zsila, Ágnes/0000-0002-8291-5997; Horváth, Rita/0009-0005-2662-2136; Urbán, Róbert/0000-0002-2058-5937; Paksi, Borbála/0000-0002-3616-2867; Darnai, Gergely/0000-0002-7042-8059; Janszky, József Vladimír/0000-0001-6100-832X; Demetrovics, Zsolt/0000-0001-5604-7551}
}

@article{MTMT:34420912,
	title = {Altered functional brain networks in problematic smartphone and social media use: resting-state fMRI study},
	url = {https://m2.mtmt.hu/api/publication/34420912},
	author = {Áfra, Eszter and Janszky, József Vladimír and Perlaki, Gábor and Orsi, Gergely and Nagy, Szilvia Anett and Arató, Ákos and Szente, Anna Tímea and Alhour, Husamalddin Ali Mohammad and Kis-Jakab, Gréta and Darnai, Gergely},
	doi = {10.1007/s11682-023-00825-y},
	journal-iso = {BRAIN IMAGING BEHAV},
	journal = {BRAIN IMAGING AND BEHAVIOR},
	unique-id = {34420912},
	issn = {1931-7557},
	abstract = {Nowadays, the limitless availability to the World Wide Web can lead to general Internet misuse and dependence. Currently, smartphone and social media use belong to the most prevalent Internet-related behavioral addiction forms. However, the neurobiological background of these Internet-related behavioral addictions is not sufficiently explored. In this study, these addiction forms were assessed with self-reported questionnaires. Resting-state functional magnetic resonance imaging was acquired for all participants ( n  = 59, 29 males) to examine functional brain networks. The resting-state networks that were discovered using independent component analysis were analyzed to estimate within network differences. Significant negative associations with social media addiction and smartphone addiction were found in the language network, the lateral visual networks, the auditory network, the sensorimotor network, the executive network and the frontoparietal network. These results suggest that problematic smartphone and social media use are associated with sensory processing and higher cognitive functioning .},
	year = {2024},
	eissn = {1931-7565},
	orcid-numbers = {Janszky, József Vladimír/0000-0001-6100-832X; Nagy, Szilvia Anett/0000-0001-6483-9209; Alhour, Husamalddin Ali Mohammad/0000-0001-5841-1652}
}

@article{MTMT:34226087,
	title = {Structural neural correlates of mental fatigue and reward-induced improvement in performance},
	url = {https://m2.mtmt.hu/api/publication/34226087},
	author = {Matuz, András and Darnai, Gergely and Zsidó, András Norbert and Janszky, József Vladimír and Csathó, Árpád},
	doi = {10.1007/s42977-023-00187-y},
	journal-iso = {BIOL FUTURA},
	journal = {BIOLOGIA FUTURA},
	unique-id = {34226087},
	issn = {2676-8615},
	abstract = {Neuroimaging studies investigating the association between mental fatigue (henceforth fatigue) and brain physiology have identified many brain regions that may underly the cognitive changes induced by fatigue. These studies focused on the functional changes and functional connectivity of the brain relating to fatigue. The structural correlates of fatigue, however, have received little attention. To fill this gap, this study explored the associations of fatigue with cortical thickness of frontal and parietal regions. In addition, we aimed to explore the associations between reward-induced improvement in performance and neuroanatomical markers in fatigued individuals. Thirty-nine healthy volunteers performed the psychomotor vigilance task for 15 min (i.e., 3 time-on-task blocks of 5 min) out of scanner; followed by an additional rewarded block of the task lasting 5 min. Baseline high-resolution T1-weigthed MR images were obtained. Reaction time increased with time-on-task but got faster again in the rewarded block. Participants’ subjective fatigue increased during task performance. In addition, we found that higher increase in subjective mental fatigue was associated with the cortical thickness of the following areas: bilateral precuneus, right precentral gyrus; right pars triangularis and left superior frontal gyrus. Our results suggest that individual differences in subjective mental fatigue may be explained by differences in the degree of cortical thickness of areas that are associated with motor processes, executive functions, intrinsic alertness and are parts of the default mode network.},
	year = {2024},
	eissn = {2676-8607},
	orcid-numbers = {Zsidó, András Norbert/0000-0003-0506-6861; Janszky, József Vladimír/0000-0001-6100-832X}
}

@article{MTMT:34113102,
	title = {Gray Matter Changes Following Mild COVID-19 : An MR Morphometric Study in Healthy Young People},
	url = {https://m2.mtmt.hu/api/publication/34113102},
	author = {Perlaki, Gábor and Darnai, Gergely and Arató, Ákos and Alhour, Husamalddin Ali Mohammad and Szente, Anna Tímea and Áfra, Eszter and Nagy, Szilvia Anett and Horváth, Réka and Kovács, Norbert and Dóczi, Tamás Péter and Orsi, Gergely and Janszky, József Vladimír},
	doi = {10.1002/jmri.28970},
	journal-iso = {JMRI - J MAGN RESON IM},
	journal = {JOURNAL OF MAGNETIC RESONANCE IMAGING},
	unique-id = {34113102},
	issn = {1053-1807},
	abstract = {Although COVID-19 is primarily an acute respiratory infection, 5%-40% of patients develop late and prolonged symptoms with frequent neurological complaints, known as long COVID syndrome. The presentation of the disease suggests that COVID infection may cause functional and/or morphological central nervous system alterations, but studies published in the literature report contradictory findings.To investigate the chronic effects of COVID-19 on cerebral grey matter in a group of young patients without comorbidities, with mild course of COVID infection and no medical complaints at the time of examination.Prospective.Thirty-eight young (age = 26.6 ± 5.0 years; male/female = 14/24), adult participants who recovered from mild COVID infection without a history of clinical long COVID and 37 healthy control subjects (age = 25.9 ± 2.8 years; male/female = 14/23).Three Tesla, 3D T1-weighted magnetization-prepared rapid gradient-echo, 2D T2-weighted turbo spin-echo.MRI-based morphometry and volumetry along with neuropsychological testing and self-assessed questionnaire.Fisher's exact test, Mann-Whitney U-test, and multiple linear regression analyses were used to assess differences between COVID and healthy control groups. P < 0.05 was used as cutoff for significance.In the COVID group, significantly lower bilateral mean cortical thickness (left/right-hemisphere: 2.51 ± 0.06 mm vs. 2.56 ± 0.07 mm, η2 p  = 0.102/2.50 ± 0.06 mm vs. 2.54 ± 0.07 mm, η2 p  = 0.101), lower subcortical gray matter (57881 ± 3998 mm3 vs. 60470 ± 5211 mm3 , η2 p  = 0.100) and lower right olfactory bulb volume (52.28 ± 13.55 mm3 vs. 60.98 ± 15.8 mm3 , η2 p  = 0.078) were found. In patients with moderate to severe anosmia, cortical thickness was significantly lower bilaterally, as compared to patients without olfactory function loss (left/right-hemisphere: 2.50 ± 0.06 mm vs. 2.56 ± 0.05 mm, η2  = 0.173/2.49 ± 0.06 mm vs. 2.55 ± 0.05 mm, η2  = 0.189). Using further exploratory analysis, significantly reduced cortical thickness was detected locally in the right lateral orbitofrontal cortex in the COVID group (2.53 ± 0.10 mm vs. 2.60 ± 0.09 mm, η2 p  = 0.112).Even without any subjective or objective neurological complaints at the time of the MR scan, subjects in the COVID group showed gray matter alterations in cortical thickness and subcortical gray matter volume.2 TECHNICAL EFFICACY: Stage 3.},
	keywords = {Brain; morphometry; cortical thickness; SARS-CoV-2},
	year = {2024},
	eissn = {1522-2586},
	orcid-numbers = {Alhour, Husamalddin Ali Mohammad/0000-0001-5841-1652; Nagy, Szilvia Anett/0000-0001-6483-9209; Kovács, Norbert/0000-0002-7332-9240; Janszky, József Vladimír/0000-0001-6100-832X}
}

@misc{MTMT:34123923,
	title = {Amikor a haszon (még) ellensúlyozza a kockázatot: Egy LECIG kezelésben részesülő Parkinson-kóros beteg esete},
	url = {https://m2.mtmt.hu/api/publication/34123923},
	author = {Pintér, Dávid and Harmat, Márk and Rohonczi, Mirtill and Aschermann, Zsuzsanna and Janszky, József Vladimír and Kovács, Norbert},
	unique-id = {34123923},
	year = {2023},
	orcid-numbers = {Janszky, József Vladimír/0000-0001-6100-832X; Kovács, Norbert/0000-0002-7332-9240}
}

@article{MTMT:34106202,
	title = {Comment on “Summing MDS-UPDRS Parts 1 + 2 (Nonmotor and Motor Experience of Daily Living): The Patient's Voice”},
	url = {https://m2.mtmt.hu/api/publication/34106202},
	author = {Kovács, Norbert and Aschermann, Zsuzsanna and Harmat, Márk and Rohonczi, Mirtill and Janszky, József Vladimír and Pintér, Dávid},
	doi = {10.1002/mds.29512},
	journal-iso = {MOVEMENT DISORD},
	journal = {MOVEMENT DISORDERS},
	volume = {38},
	unique-id = {34106202},
	issn = {0885-3185},
	year = {2023},
	eissn = {1531-8257},
	pages = {1563-1564},
	orcid-numbers = {Kovács, Norbert/0000-0002-7332-9240; Janszky, József Vladimír/0000-0001-6100-832X}
}

@misc{MTMT:34078356,
	title = {Applicability of imaging-guided programming during initiating deep brain stimulation for Parkinson’s disease},
	url = {https://m2.mtmt.hu/api/publication/34078356},
	author = {Izsó, Lujza and Aschermann, Zsuzsanna and Járdaházi, Evelyn and Harmat, Márk and Janszky, József Vladimír and Kovács, Norbert and Pintér, Dávid},
	unique-id = {34078356},
	year = {2023},
	orcid-numbers = {Janszky, József Vladimír/0000-0001-6100-832X; Kovács, Norbert/0000-0002-7332-9240}
}

@article{MTMT:34050785,
	title = {White matter hyperintensities associated with impulse control disorders in Parkinson's Disease},
	url = {https://m2.mtmt.hu/api/publication/34050785},
	author = {Hernádi, Gabriella and Perlaki, Gábor and Kovács, Márton and Pintér, Dávid and Orsi, Gergely and Janszky, József Vladimír and Kovács, Norbert},
	doi = {10.1038/s41598-023-37054-8},
	journal-iso = {SCI REP},
	journal = {SCIENTIFIC REPORTS},
	volume = {13},
	unique-id = {34050785},
	issn = {2045-2322},
	abstract = {Impulse control disorders (ICDs) in Parkinson's disease (PD) are increasingly recognized as clinically significant non-motor features that potentially impair the quality of life. White matter hyperintensities (WMHs), detected by magnetic resonance imaging, are frequently observed in PD and can be associated with both motor- and certain non-motor symptoms. Given the limited number of non-motor features studied in this context, our aim was to reveal the potential association between the severity of WMHs and ICDs in PD. Fluid-attenuated inversion recovery magnetic resonance images were retrospectively evaluated in 70 patients with PD (48 males; 59.3 ± 10.1 years). The severity of WMHs was assessed by Fazekas scores and by the volume and number of supratentorial WMHs. ICDs were evaluated using the modified Minnesota Impulsive Disorders Interview. Significant interaction between age and the severity of WMHs was present for ICDs. In our younger patients (< 60.5 years), severity of WMHs was positively associated with ICDs (p = 0.004, p = 0.021, p < 0.001 and p < 0.001, respectively for periventricular white matter and total Fazekas scores and the volume and number of WMHs). Our study supports the hypothesis that WMHs of presumed vascular origin may contribute to ICDs in PD. Future prospective studies are needed to assess the prognostic relevance of this finding.},
	year = {2023},
	eissn = {2045-2322},
	orcid-numbers = {Janszky, József Vladimír/0000-0001-6100-832X; Kovács, Norbert/0000-0002-7332-9240}
}

@article{MTMT:34012838,
	title = {Urinary titin in myotonic dystrophy type 1},
	url = {https://m2.mtmt.hu/api/publication/34012838},
	author = {Varga, Dávid and Ruszin-Perecz, Brigitta and Fülöp, Krisztina and Sipos, Andrea and Janszky, József Vladimír and Hajdú, Nándor and Pál, Endre},
	doi = {10.1002/mus.27917},
	journal-iso = {MUSCLE NERVE},
	journal = {MUSCLE & NERVE},
	volume = {68},
	unique-id = {34012838},
	issn = {0148-639X},
	abstract = {Urinary titin, an easy-to-obtain marker, has been investigated in muscular dystrophies, but not in myotonic dystrophy type 1 (DM1). We investigated the role of titin as a biomarker of muscle injury in DM1.We compared the urinary titin N-fragment/creatinine ratio in 29 patients with DM1 vs. 30 healthy controls. We also recorded clinical data such as muscle strength, serum creatine kinase, DM1-related outcome measures, and the 20-item DM1-activ questionnaire. The severity of the disease was graded using the Muscular Impairment Rating Scale (MIRS).The titin/creatinine ratio was significantly higher in the urine samples of DM1 patients than of healthy controls (median ± mean absolute deviation [MAD]: 39.313 ± 26.546 vs. 6.768 ± 5.245 pmol/mg creatinine; P < .001), and was related to muscle impairment graded by MIRS (τ = 0.503, P = .038).Urinary titin may be a biomarker for DM1. Long-term follow-up of DM1 patients is needed to investigate the potential role of titin as a biomarker for disease activity and progression.},
	keywords = {myotonic dystrophy type 1; muscle impairment rating scale; urinary titin},
	year = {2023},
	eissn = {1097-4598},
	pages = {215-218},
	orcid-numbers = {Janszky, József Vladimír/0000-0001-6100-832X; Hajdú, Nándor/0000-0003-4012-4932}
}