Drosophila Rab39 Attenuates Lysosomal Degradation

Lakatos, Zsolt [Lakatos, Zsolt (Sejtbiológia), author]; Benkő, Péter [Benkő, Péter (élettan), author] Department of Physiology (SU / FM / I); Juhász, Gábor ✉ [Juhász, Gábor (Sejtbiológia), author] Institute of Genetics; Department of Anatomy, Cell and Developmental B... (ELTE / ELU FoS / Bio_I); Lőrincz, Péter ✉ [Lőrincz, Péter (Sejtbiológia), author] Department of Anatomy, Cell and Developmental B... (ELTE / ELU FoS / Bio_I)

English Article (Journal Article) Scientific
Published: INTERNATIONAL JOURNAL OF MOLECULAR SCIENCES 1661-6596 1422-0067 22 (19) Paper: 10635 , 12 p. 2021
  • SJR Scopus - Spectroscopy: D1
Identifiers
Fundings:
  • (ÚNKP-20-3)
  • (ÚNKP-20-3-II-ELTE-730)
  • (PPD-2018-222) Funder: MTA
  • (LP-2014/2) Funder: MTA
  • (FK 138851) Funder: NRDIO
  • European Regional Development Fund(GINOP-2.3.2-15-2016-00006) Funder: GINOP
  • (GINOP-2.3.2-15-2016-00032)
  • (KKP129797) Funder: NRDIO
  • (K119842) Funder: NRDIO
Lysosomal degradation, the common destination of autophagy and endocytosis, is one of the most important elements of eukaryotic metabolism. The small GTPases Rab39A and B are potential new effectors of this pathway, as their malfunction is implicated in severe human diseases like cancer and neurodegeneration. In this study, the lysosomal regulatory role of the single Drosophila Rab39 ortholog was characterized, providing valuable insight into the potential cell biological mechanisms mediated by these proteins. Using a de novo CRISPR-generated rab39 mutant, we found no failure in the early steps of endocytosis and autophagy. On the contrary, we found that Rab39 mutant nephrocytes internalize and degrade endocytic cargo at a higher rate compared to control cells. In addition, Rab39 mutant fat body cells contain small yet functional autolysosomes without lysosomal fusion defect. Our data identify Drosophila Rab39 as a negative regulator of lysosomal clearance during both endocytosis and autophagy.
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2025-04-25 16:24