https://m2.mtmt.hu/ hun 2026-09-22 10:12 JournalArticle 31159945 VALIDATED true 0 false 2026-09-11T12:19:59.987+0000 2020-04-24T12:00:36.484+0000 2020-02-05T14:59:22.753+0000 true 10022707 Najbauerné Bacsics Zsuzsanna /api/admin/10022707 Admin true 10022707 2021-08-03T09:34:15.907+0000 2021-08-03T09:34:15.907+0000 2020-04-24T11:58:39.518+0000 true 10026589 Tichy Rita /api/admin/10026589 Admin true 10026589 2020-04-24T12:00:36.509+0000 true 10026589 Tichy Rita /api/admin/10026589 Admin true 10026589 true false true 24 24 /api/publicationtype/24 PublicationType 1 true 24 JournalArticle true 10000359 Study Group true 24 24 /api/publicationtype/24 PublicationType 1 true 24 JournalArticle /api/subtype/10000359 Sokszerzős vagy csoportos szerzőségű szakcikk SubType 104 true 10000359 true 1 /api/category/1 Category true 1 Barthel, Floris P. Longitudinal molecular trajectories of diffuse glioma in adults true true /api/journal/3308 REVIEWED true false 3308 false 3308 true 0028-0836 1476-4687 Journal FOREIGN 576 7785 112 120 112 28 2019 The evolutionary processes that drive universal therapeutic resistance in adult patients with diffuse glioma remain unclear(12). Here we analysed temporally separated DNA-sequencing data and matched clinical annotation from 222 adult patients with glioma. By analysing mutations and copy numbers across the three major subtypes of diffuse glioma, we found that driver genes detected at the initial stage of disease were retained at recurrence, where as there was little evidence of recurrence-specific gene alterations. Treatment with alkylating agents resulted in a hypermutator phenotype at different rates across the glioma subtypes, and hypermutation was not associated with differences in overall survival. Acquired aneuploidy was frequently detected in recurrent gliomas and was characterized by IDH mutation but without co-deletion of chromosome arms 1p/19q, and further converged with acquired alterations in the cell cycle and poor outcomes. The clonal architecture of each tumour remained similar overtime, but the presence of subclonal selection was associated with decreased survival. Finally, there were no differences in the levels of immunoediting between initial and recurrentgliomas. Collectively, our results suggest that the strongest selective pressures occur during earlyglioma development and that current therapies shape this evolution in a largely stochastic manner. true true 2020 true true true false true false NONE 2026-09-11 false 433 0 429 320 4 428 424 427 377 433 429 324 320 324 109 0 433 324 429 320 0 0 0 Folyóiratcikk Journal Article 24 429 Könyvrészlet Chapter in Book 25 0 Könyv Book 23 0 Egyéb konferenciaközlemény Conference paper 31 0 Egyéb konferenciakötet Conference proceedings 32 0 Oltalmi formák Protection forms 26 0 Disszertáció Thesis 28 4 Egyéb Miscellaneous 29 0 Alkotás Achievement 22 0 Kutatási adat Research data 33 0 Folyóiratcikk Journal Article 24 0 0 0 Könyvrészlet Chapter in Book 25 0 0 0 Könyv Book 23 0 0 0 Egyéb konferenciaközlemény Conference paper 31 0 0 0 Egyéb konferenciakötet Conference proceedings 32 0 0 0 Oltalmi formák Protection forms 26 0 0 0 Disszertáció Thesis 28 0 0 0 Egyéb Miscellaneous 29 0 0 0 Alkotás Achievement 22 0 0 0 Kutatási adat Research data 33 0 0 0 2020 0 32 23 32 23 2021 0 77 55 77 55 2022 0 66 52 66 52 2023 0 89 69 89 69 2024 0 81 57 81 57 2025 0 56 40 56 40 2026 0 32 28 32 28 D1 A false 10018054 false false false 2020-02-04T15:04:25.040+0000 I. 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Sz. Patológiai és Kísérleti Rákkutató Intézet</span> ;&nbsp;&nbsp;&nbsp; et al. (88) </div> <div class="autype autype-11"> Kollaborációs közreműködő: <span class="author-name" >Barthel FP </span> ;&nbsp;&nbsp;&nbsp; et al. 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