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"published" : false, "oldId" : 322051, "snippet" : true } ], "journal" : { "otype" : "Journal", "mtid" : 10014463, "link" : "/api/journal/10014463", "label" : "HEMODIALYSIS INTERNATIONAL 1492-7535 1542-4758", "pIssn" : "1492-7535", "eIssn" : "1542-4758", "reviewType" : "REVIEWED", "noIF" : false, "sciIndexed" : true, "scopusIndexed" : true, "lang" : "FOREIGN", "hungarian" : false, "published" : true, "oldId" : 10014463, "snippet" : true }, "volume" : "15", "issue" : "4", "firstPage" : "501", "lastPage" : "508", "firstPageOrInternalIdForSort" : "501", "pageLength" : 8, "publishedYear" : 2011, "abstractText" : "The relationship between renal disease progression and genetic \npolymorphism of enzymes influencing endothelial function remains \nincompletely understood. We genotyped three cohorts of elderly \nHungarian patients: 245 patients with end-stage renal disease \n(ESRD) on chronic hemodialysis (HD), 88 patients with mild \nchronic kidney disease (CKD), and 200 healthy controls. The \nunderlying diagnoses of renal diseases were primary \nglomerulonephritis, interstitial nephritis, hypertension, \ndiabetic nephropathy, and hereditary diseases. We examined \ngenetic polymorphisms of eight candidate genes associated with \nendothelial function: endothelial constitutive nitric oxide \nsynthase (ecNOS) T-786C, endothelin-1 G5727T, \nmethylenetetrahydrofolate reductase (MTHFR) C677T, paraoxonase-1 \nQ192R and M55L, angiotensinogen M235T, angiotensin-converting \nenzyme (ACE) I/D and angiotensin II type 1 receptor A1166C gene. \nSix gene polymorphisms were detected by real-time polymerase \nchain reaction with melting-point analysis, and two via allele-\nspecific amplification and gel electrophoresis. Control group \npatients were in Hardy-Weinberg equilibrium for all tested \ngenotypes. In ESRD patients attributed to hypertension, the \nendothelin gene G5727T GG genotype occurred significantly less \nbut GT genotype more frequently (P<0.01 for both). In ESRD \npatients attributed to primary glomerulonephritis, more ACE DD \nand less ID genotypes were found (P<0.02 for both) than in the \ncontrols. The underlying diagnosis may modify the association of \ngenetic polymorphism and dialysis-dependent ESRD. © 2011 The \nAuthors; Hemodialysis International © 2011 International Society \nfor Hemodialysis.", "subjects" : [ { "otype" : "Classification", "mtid" : 12215, "link" : "/api/classification/12215", "label" : "Klinikai orvostan", "published" : true, "snippet" : true } ], "keywords" : [ { "otype" : "Keyword", "mtid" : 50, "link" : "/api/keyword/50", "label" : "Aged", "published" : true, "oldId" : 50, "snippet" : true }, { "otype" : "Keyword", "mtid" : 52, "link" : "/api/keyword/52", "label" : "Adult", "published" : true, "oldId" : 52, "snippet" : true }, { "otype" : "Keyword", "mtid" : 56, "link" : "/api/keyword/56", "label" : "Female", "published" : true, "oldId" : 56, "snippet" : true }, { "otype" : "Keyword", "mtid" : 61, "link" : "/api/keyword/61", "label" : "Middle Aged", "published" : true, "oldId" : 61, "snippet" : true }, { "otype" : "Keyword", "mtid" : 62, "link" : 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