https://m2.mtmt.hu/ hun 2026-10-23 03:07 JournalArticle 1005053 VALIDATED true 0 2018-09-13T03:24:01.000+0000 false 1005053 2026-10-13T04:22:42.419+0000 2025-07-01T22:37:18.407+0000 2006-02-13T23:00:00.000+0000 true 10000069 Burián Katalin /api/author/10000069 Author true 10000069 2025-07-01T22:37:18.606+0000 2025-07-01T22:37:18.606+0000 2023-11-08T13:21:36.444+0000 true 10065569 Pécsi Éva /api/admin/10065569 Admin true Admin 10067876 /api/admin/10067876 Ladányi Gusztáv true true Admin 10080105 /api/admin/10080105 Kériné Tornyi Katalin true true 2024-10-17T16:18:31.199+0000 true false true 24 24 /api/publicationtype/24 PublicationType 1 true 24 JournalArticle true 10000059 Article true 24 24 /api/publicationtype/24 PublicationType 1 true 24 JournalArticle /api/subtype/10000059 Szakcikk SubType 101 true 10000059 true 1 /api/category/1 Category true 1 Rugonfalvi, Kiss S Association of Chlamydia pneumoniae with coronary artery disease and its progression is dependent on the modifying effect of mannose-binding lectin true true /api/journal/1023 REVIEWED true false 1023 false 1023 true 0009-7322 1524-4539 Journal FOREIGN 106 9 1071 1076 1071 6 2002 BACKGROUND: The possible association between coronary artery disease (CAD) and Chlamydia pneumoniae (C pneumoniae) infection is controversial. On the basis of the recent suggestion that mannose-binding lectin (MBL) variant alleles are related to an increased risk of severe atherosclerosis, and on the in vitro interaction of MBL with C pneumoniae, we asked whether MBL might contribute to CAD in conjunction with C pneumoniae. METHODS AND RESULTS: Antibodies to C pneumoniae were measured by immunofluorescence and MBL alleles were determined by polymerase chain reaction technique in samples from 210 patients with CAD and 257 healthy subjects from Hungary collected between 1995 and 1996. A higher percentage of patients with CAD were anti-C pneumoniae positive as compared with the control group (P=0.058). However, at logistic regression analysis adjusted to age, sex, and serum lipid levels, this difference was confined only to subjects carrying MBL variant alleles (P=0.035, odds ratio 2.63, [95% CI: 1.07 to 6.45]). In contrast, no significant difference was seen in those homozygous for the normal MBL allele (P=0.412). During a 65+/-5.8-month follow-up period, major outcomes (new myocardial infarction, and/or bypass operation or cardiovascular death) occurred in 11 C pneumoniae positive and 3 C pneumoniae negative patients. In the C pneumoniae positive group, the odds ratio of development of outcomes was 3.27 (95% CI: 1.10 to 9.71, P=0.033) in the carriers of the MBL variant alleles compared with the homozygous carriers of the normal MBL allele. CONCLUSIONS: These results indicate that infection with C pneumoniae leads mainly to the development and progression of severe CAD in patients with variation in the MBL gene. 2006 true true true false true false GREEN GREEN 2026-10-13 true https://real.mtak.hu/3060 true 10018695 Bernátskyné Babus Csilla /api/admin/10018695 Admin true 10018695 90 0 88 75 13 88 80 78 85 88 88 75 75 77 13 0 90 77 88 75 0 1 1 Folyóiratcikk Journal Article 24 83 Könyvrészlet Chapter in Book 25 4 Könyv Book 23 1 Egyéb konferenciaközlemény Conference paper 31 0 Egyéb konferenciakötet Conference proceedings 32 0 Oltalmi formák Protection forms 26 0 Disszertáció Thesis 28 2 Egyéb Miscellaneous 29 0 Alkotás Achievement 22 0 Kutatási adat Research data 33 0 Folyóiratcikk Journal Article 24 0 0 0 Könyvrészlet Chapter in Book 25 0 0 0 Könyv Book 23 0 0 0 Egyéb konferenciaközlemény Conference paper 31 0 0 0 Egyéb konferenciakötet Conference proceedings 32 0 0 0 Oltalmi formák Protection forms 26 0 0 0 Disszertáció Thesis 28 0 0 0 Egyéb Miscellaneous 29 0 0 0 Alkotás Achievement 22 0 0 0 Kutatási adat Research data 33 0 0 0 2002 0 1 1 1 1 2003 0 6 6 6 6 2004 0 14 9 14 9 2005 0 11 10 11 10 2006 0 5 4 5 4 2007 0 7 7 7 7 2008 0 5 5 5 5 2009 0 9 8 9 8 2010 0 6 4 6 4 2011 0 3 3 3 3 2012 0 3 2 3 2 2013 0 2 2 2 2 2014 0 3 3 3 3 2015 0 1 1 1 1 2016 0 4 3 4 3 2017 0 2 1 2 1 2018 0 1 1 1 1 2019 0 1 1 1 1 2020 0 2 2 2 2 2021 0 2 2 2 2 2022 0 1 1 1 1 2023 0 1 1 1 1 D1 false 10000069 false false false 2015-03-08T18:43:29.000+0000 2012-09-13T18:32:11.000+0000 Akadémikus bibliográfia; III. Sz. Belgyógyászati Klinika (SE / AOK / K); MTA-SE Anyagcsere és Atherosclerosis Kutatócsop... 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mtid="10001711"><a href="/gui2/?type=authors&mode=browse&sel=10001711" target="_blank">Endresz V (<span class="authorship-author-name">Endrész Valéria</span> <span class="authorAux-mtmt"> Mikrobiológia</span>) </a> </span> <span class="author-affil"><span title="Szegedi Tudományegyetem">SZTE</span>/<span title="Általános Orvostudományi Kar">ÁOK</span>/Orvosi Mikrobiológiai és Immunbiológiai Intézet</span> ;&nbsp;&nbsp;&nbsp; <span class="author-name" >Madsen H O </span> ;&nbsp;&nbsp;&nbsp; <span class="author-name" mtid="10000069"><a href="/gui2/?type=authors&mode=browse&sel=10000069" target="_blank">Burian K (<span class="authorship-author-name">Burián Katalin</span> <span class="authorAux-mtmt"> orvosi mikrobiológia</span>) </a> </span> <span class="author-affil"><span title="Szegedi Tudományegyetem">SZTE</span>/<span title="Általános Orvostudományi Kar">ÁOK</span>/Orvosi Mikrobiológiai és Immunbiológiai Intézet</span> ;&nbsp;&nbsp;&nbsp; <span class="author-name" >Duba J </span> ;&nbsp;&nbsp;&nbsp; <span class="author-name" mtid="10000530"><a href="/gui2/?type=authors&mode=browse&sel=10000530" target="_blank">Prohaszka Z (<span class="authorship-author-name">Prohászka Zoltán</span> <span class="authorAux-mtmt"> Immunológia</span>) </a> </span> <span class="author-affil"><span title="MTA Támogatott Kutatócsoportok">MTA TKI</span>/MTA-SE Anyagcsere és Atherosclerosis Kutatócsoport (2002-ig: MTA-SE Anyagcsere Genetikai és Immunológiai Kutatócsoport, 2007-től beolvadt az SE03-ba)</span> ;&nbsp;&nbsp;&nbsp; <span class="author-name" mtid="10000449"><a href="/gui2/?type=authors&mode=browse&sel=10000449" target="_blank">Karadi I (<span class="authorship-author-name">Karádi István</span> <span class="authorAux-mtmt"> Endokrinológia</span>) </a> </span> <span class="author-affil"><span title="Semmelweis Egyetem">SE</span>/<span title="Általános Orvostudományi Kar">AOK</span>/<span title="Klinikum">K</span>/III. Sz. Belgyógyászati Klinika</span> ;&nbsp;&nbsp;&nbsp; <span class="author-name" mtid="10001049"><a href="/gui2/?type=authors&mode=browse&sel=10001049" target="_blank">Romics L (<span class="authorship-author-name">Romics László</span> <span class="authorAux-mtmt"> Belgyógyászat</span>) </a> </span> <span class="author-affil"><span title="MTA Támogatott Kutatócsoportok">MTA TKI</span>/MTA-SE Anyagcsere és Atherosclerosis Kutatócsoport (2002-ig: MTA-SE Anyagcsere Genetikai és Immunológiai Kutatócsoport, 2007-től beolvadt az SE03-ba)</span> ;&nbsp;&nbsp;&nbsp; <span class="author-name" mtid="10010166"><a href="/gui2/?type=authors&mode=browse&sel=10010166" target="_blank">Gonczol E (<span class="authorship-author-name">Gönczöl Éva</span> <span class="authorAux-mtmt"> Mikrobiológia, virológia, immunológia</span>) </a> </span> ;&nbsp;&nbsp;&nbsp; <span class="author-name" mtid="10007973"><a href="/gui2/?type=authors&mode=browse&sel=10007973" target="_blank">Fust G (<span class="authorship-author-name">Füst György</span> <span class="authorAux-mtmt"> Immunológia</span>) </a> </span> <span class="author-affil"><span title="MTA Támogatott Kutatócsoportok">MTA TKI</span>/MTA-SE Anyagcsere és Atherosclerosis Kutatócsoport (2002-ig: MTA-SE Anyagcsere Genetikai és Immunológiai Kutatócsoport, 2007-től beolvadt az SE03-ba)</span> ;&nbsp;&nbsp;&nbsp; <span class="author-name" >Garred P ✉ </span> </div> </div> <div class="title"><a href="/gui2/?mode=browse&params=publication;1005053" target="_blank">Association of Chlamydia pneumoniae with coronary artery disease and its progression is dependent on the modifying effect of mannose-binding lectin</a></div> <div> <span class="journal-title">CIRCULATION</span> <span class="journal-issn">(<a target="_blank" href="https://portal.issn.org/resource/ISSN/0009-7322">0009-7322</a> <a target="_blank" href="https://portal.issn.org/resource/ISSN/1524-4539">1524-4539</a>)</span>: <span class="journal-volume">106</span> <span class="journal-issue">9</span> <span class="page"> pp 1071-1076 </span> <span class="year">(2002)</span> </div> <div class="pub-footer"> <span class="language" xmlns="http://www.w3.org/1999/html">Nyelv: Angol | </span> <span class="identifiers"> <span class="id identifier oa_none" title="none"> <a style="color:blue" title="10.1161/01.CIR.0000027137.96791.6A" target="_blank" href="https://doi.org/10.1161/01.CIR.0000027137.96791.6A"> DOI </a> </span> <span class="id identifier oa_GREEN" title=" Green "> <a style="color:blue" title="3060" target="_blank" href="https://real.mtak.hu/3060"> REAL </a> </span> <span class="id identifier oa_none" title="none"> <a style="color:blue" title="000177794800018" target="_blank" href="https://www.webofscience.com/wos/woscc/full-record/000177794800018"> WoS </a> </span> <span class="id identifier oa_none" title="none"> <a style="color:blue" title="0037183649" target="_blank" href="http://www.scopus.com/record/display.url?origin=inward&eid=2-s2.0-0037183649"> Scopus </a> </span> <span class="id identifier oa_none" title="none"> <a style="color:blue" title="12196331" target="_blank" href="http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=12196331&dopt=Abstract"> PubMed </a> </span> <span class="id identifier oa_CLOSED" title=" Zárt "> <a style="color:blue" title="28616" target="_blank" href="http://publicatio.bibl.u-szeged.hu/28616"> SZTE Publicatio </a> </span> </span> <OnlyViewableByAuthor><div class="ratings"> <div class="journal-subject">Folyóirat szakterülete: Scopus - Cardiology and Cardiovascular Medicine&nbsp;&nbsp;&nbsp;SJR indikátor:&nbsp;D1</div> <div class="journal-subject">Folyóirat szakterülete: Scopus - Physiology (medical)&nbsp;&nbsp;&nbsp;SJR indikátor:&nbsp;D1</div> </div></OnlyViewableByAuthor> <div class="publication-citation" style="margin-left: 0.5cm;"> <span title="Nyilvános idézőközlemények összesen, említések nélkül" class="citingPub-count">Nyilvános idéző összesen: 90</span> | Független: 77 | Függő: 13 | Nem jelölt: 0 | WoS jelölt: 78 | Scopus jelölt:&nbsp;85 | WoS/Scopus jelölt:&nbsp;88 | DOI jelölt:&nbsp;80 </div> <div class="publication-citation"> <a target="_blank" href="/api/publication?cond=citations.related;eq;1005053&sort=publishedYear,desc&sort=title"> Idézett közlemények száma: 1 </a> </div> <div class="mtid"><span class="long-pub-mtid">Közlemény: 1005053</span> | <span class="status-data status-VALIDATED"> Egyeztetett </span> <span class="oldId">Régi azonosító: 1005053</span> | Forrás Idéző | <span class="type-subtype">Folyóiratcikk ( Szakcikk ) </span> | <span class="pub-category">Tudományos</span> | <span class="publication-sourceOfData">MEDLINE</span> </div> <div class="special-affiliations"> Speciális affiliáció: Akadémikus bibliográfia </div> <div class="lastModified">Utolsó módosítás: 2025.07.02. 00:37 Ladányi Gusztáv (MTMT API user, admin) </div> <div class="lockedBy">Központi kezelésű 2024.10.17. 18:18 Ladányi Gusztáv (MTMT API user, admin) Kériné Tornyi Katalin (MTMT ellenőrzés admin5) </div> </div></div>